Roland Martin

发表

T cell response to 2′,3′-cyclic nucleotide 3′-phosphodiesterase (CNPase) in multiple sclerosis patients

H. McFarland, R. Martin, P. Muraro, 2002, Journal of Neuroimmunology.

Immunodominance of a low-affinity major histocompatibility complex-binding myelin basic protein epitope (residues 111-129) in HLA-DR4 (B1*0401) subjects is associated with a restricted T cell receptor repertoire.

D E Banks, J. Nagle, H. McFarland, 1997, The Journal of clinical investigation.

Relationships among TCR ligand potency, thresholds for effector function elicitation, and the quality of early signaling events in human T cells.

R. Germain, B. Hemmer, R. Martin, 1998, Journal of immunology.

A novel population of CD4+CD56+ myelin-reactive T cells lyses target cells expressing CD56/neural cell adhesion molecule.

H. McFarland, R. Martin, M. Vergelli, 1996, Journal of immunology.

Probing degeneracy in T-cell recognition using peptide combinatorial libraries.

R. Houghten, C. Pinilla, B. Hemmer, 1998, Immunology today.

Immunogenicity. I. Use of peptide libraries to identify epitopes that activate clonotypic CD4+ T cells and induce T cell responses to native peptide ligands.

R A Houghten, R. Houghten, C. Pinilla, 1999, Journal of immunology.

Modifications of peptide ligands enhancing T cell responsiveness imply large numbers of stimulatory ligands for autoreactive T cells.

J. Coligan, B. Hemmer, H. McFarland, 1997, Journal of immunology.

Human autoreactive CD4+ T cell clones use perforin- or Fas/Fas ligand-mediated pathways for target cell lysis.

B. Hemmer, H. McFarland, R. Martin, 1997, Journal of immunology.

Predictable TCR antigen recognition based on peptide scans leads to the identification of agonist ligands with no sequence homology.

R. Houghten, C. Pinilla, B. Hemmer, 1998, Journal of immunology.