W. Goebel

Complementation of Listeria seeligeri with the plcA-prfA genes from L. monocytogenes activates transcription of seeligerolysin and leads to bacterial escape from the phagosome of infected mammalian cells.

I. Karunasagar, R. Lampidis, J. Kreft, 1997, FEMS microbiology letters.

Transcriptional activation of the Listeria monocytogenes hemolysin gene in Bacillus subtilis

P. Youngman, D. Portnoy, N. Freitag, 1992, Journal of bacteriology.

A naturally occurring mutation K220T in the pleiotropic activator PrfA of Listeria monocytogenes results in a loss of virulence due to decreasing DNA-binding affinity.

W. Goebel, A. Fedorov, S. Almo, 2007, Microbiology.

Functional Similarities between the Listeria monocytogenes Virulence Regulator PrfA and Cyclic AMP Receptor Protein: the PrfA* (Gly145Ser) Mutation Increases Binding Affinity for Target DNA

J. Vázquez-Boland, Y. Vega, M. Ripio, 1998, Journal of bacteriology.

Positive Selection of Mutations Leading to Loss or Reduction of Transcriptional Activity of PrfA, the Central Regulator ofListeria monocytogenes Virulence

W. Goebel, J. Vázquez-Boland, A. Bubert, 2001, Journal of bacteriology.