Tomoko Hasunuma

发表

Monoclonal expansion of synoviocytes in rheumatoid arthritis.

T. Sumida, K. Nishioka, T. Hasunuma, 1998, Arthritis and rheumatism.

High frequencies of identical T cell clonotypes in synovial tissues of rheumatoid arthritis patients suggest the occurrence of common antigen-driven immune responses.

K. Nishioka, S. Yoshino, T. Kato, 1996, Arthritis and rheumatism.

Selective activation of the JNK/AP-1 pathway in Fas-mediated apoptosis of rheumatoid arthritis synoviocytes.

T. Sumida, K. Nishioka, T. Hasunuma, 1997, Arthritis and rheumatism.

Therapeutic effect of the anti-Fas antibody on arthritis in HTLV-1 tax transgenic mice.

T. Sumida, S. Yonehara, Y. Iwakura, 1996, The Journal of clinical investigation.

Molecular mechanism of immune response, synovial proliferation and apoptosis in rheumatoid arthritis

K. Nishioka, T. Kato, T. Hasunuma, 2004, Springer Seminars in Immunopathology.

Accumulation of soluble Fas in inflamed joints of patients with rheumatoid arthritis.

T. Sumida, K. Okumura, H. Yagita, 1997, Arthritis and rheumatism.

Overgrowth of human synovial cells driven by the human T cell leukemia virus type I tax gene.

T. Nakajima, I. Maruyama, K. Nishioka, 1993, The Journal of clinical investigation.

Novel gene therapy for rheumatoid arthritis by FADD gene transfer: induction of apoptosis of rheumatoid synoviocytes but not chondrocytes

H. Hamada, K. Nishioka, T. Kato, 2000, Gene Therapy.

Fas-associated death domain protein is a Fas-mediated apoptosis modulator in synoviocytes.

T. Sumida, K. Nishioka, T. Kato, 2000, Rheumatology.

Fas-mediated stimulation induces IL-8 secretion by rheumatoid arthritis synoviocytes independently of CPP32-mediated apoptosis.

K. Okumura, H. Yagita, K. Nishioka, 1996, Biochemical and biophysical research communications.