Tracy J. Lovatt

The rate of increase in the numbers of primary sporadic basal cell carcinomas during follow up is associated with age at first presentation.

Sudarshan Ramachandran, J. Lear, R. Strange, 2002, Carcinogenesis.

Susceptibility and modifier genes in cutaneous basal cell carcinomas and their associations with clinical phenotype.

J. Lear, R. Strange, A. Fryer, 2001, Journal of photochemistry and photobiology. B, Biology.

Associations between ultraviolet radiation, basal cell carcinoma site and histology, host characteristics, and rate of development of further tumors.

Andrew G Smith, V. Samarasinghe, J. Lear, 2005, Journal of the American Academy of Dermatology.

PTCH polymorphism is associated with the rate of increase in basal cell carcinoma numbers during follow‐up: Preliminary data on the influence of an exon 12‐exon 23 haplotype

Andrew G Smith, J. Lear, R. Strange, 2004, Environmental and molecular mutagenesis.

Polymorphism in the nuclear excision repair gene ERCC2/XPD: association between an exon 6–exon 10 haplotype and susceptibility to cutaneous basal cell carcinoma

Andrew G Smith, J. Lear, R. Strange, 2005, Human mutation.

Associations between UVR exposure and basal cell carcinoma site and histology.

Andrew G Smith, J. Lear, C. Griffiths, 2004, Cancer letters.

Combined effects of gender, skin type and polymorphic genes on clinical phenotype: use of rate of increase in numbers of basal cell carcinomas as a model system.

Andrew G Smith, J. Lear, R. Strange, 2003, Cancer letters.

Polymorphisms in glutathione S-transferases and non-melanoma skin cancer risk in Australian renal transplant recipients.

D. Nicol, P. Harden, C. Hawley, 2004, Carcinogenesis.