J. D. Bos

Human epidermal Langerhans cells undergo profound morphologic and phenotypical changes during in vitro culture.

P. Peters, J. Bos, M. Kapsenberg, 1990, The Journal of investigative dermatology.

The selective antigen-presenting cell capacity of activated B lymphocytes in HLA-II-restricted responses of CD4+ T lymphocytes.

J. Bos, M. Teunissen, T. C. T. M. van der Pouw Kraan, 1989, Immunology.

UVB radiation preferentially induces recruitment of memory CD4+ T cells in normal human skin: long-term effect after a single exposure.

M. D. de Rie, P. Das, J. Bos, 1998, The Journal of investigative dermatology.

Interleukin-17 and interferon-gamma synergize in the enhancement of proinflammatory cytokine production by human keratinocytes.

R. de Waal Malefyt, J. Bos, E. Wierenga, 1998, The Journal of investigative dermatology.

In contrast to their murine counterparts, normal human keratinocytes and human epidermoid cell lines A431 and HaCaT fail to express IL‐10 mRNA and protein

R. de Waal Malefyt, E. Schmitt, J. Bos, 1997, Clinical and experimental immunology.

Reduced antigen‐presenting function of human Epstein‐Barr virus (EBV)‐B cells and monocytes after UVB radiation is accompanied by decreased expression of B7, intercellular adhesion molecule‐1 (ICAM‐1) and LFA‐3

J. Bos, M. Teunissen, I. Kremer, 1995, Clinical and experimental immunology.

Intercellular adhesion molecule-3 (CD50) on human epidermal Langerhans cells participates in T-cell activation.

J. Bos, M. Teunissen, C. W. Koomen, 1995, The Journal of investigative dermatology.

Clinical improvement in chronic plaque‐type psoriasis lesions after narrow‐band UVB therapy is accompanied by a decrease in the expression of IFN‐γ inducers – IL‐12, IL‐18 and IL‐23

J. Bos, G. Piskin, R. Sylva-Steenland, 2004, Experimental dermatology.