Hans-Ulrich Bernard

发表

Transcription of the transforming genes of the oncogenic human papillomavirus-16 is stimulated by tumor promotors through AP1 binding sites.

H. Bernard, G. Klock, T. Chong, 1990, Nucleic acids research.

Progesterone and glucocorticoid response elements occur in the long control regions of several human papillomaviruses involved in anogenital neoplasia

H. Bernard, G. Klock, W. K. Chan, 1989, Journal of virology.

Transcriptional activation of human papillomavirus 16 by nuclear factor I, AP1, steroid receptors and a possibly novel transcription factor, PVF: a model for the composition of genital papillomavirus enhancers.

H. Bernard, T. Chong, W. K. Chan, 1990, Nucleic acids research.

Numerous nuclear proteins bind the long control region of human papillomavirus type 16: a subset of 6 of 23 DNase I-protected segments coincides with the location of the cell-type-specific enhancer

B. Gloss, H. Bernard, T. Chong, 1989, Journal of virology.

The enhancer of human papillomavirus type 16: binding sites for the ubiquitous transcription factors oct-1, NFA, TEF-2, NF1, and AP-1 participate in epithelial cell-specific transcription

B. Gloss, H. Bernard, M. Isa, 1991, Journal of virology.

Transcription Factor Binding Sites in the Long Control Region of Genital HPVs

H. Bernard, Shih-Yen Chan, M. O'Connor, 1995 .

Nuclear factor I and epithelial cell-specific transcription of human papillomavirus type 16

H. Bernard, Y. Liu, D. Apt, 1993, Journal of virology.

During negative regulation of the human papillomavirus-16 E6 promoter, the viral E2 protein can displace Sp1 from a proximal promoter element.

B. Gloss, H. Bernard, S. H. Tan, 1992, Nucleic acids research.