Michael Karin

发表

M-phase-specific phosphorylation of the POU transcription factor GHF-1 by a cell cycle-regulated protein kinase inhibits DNA binding

M. Karin, C. Caelles, H. Hennemann, 1995, Molecular and cellular biology.

p105 and p98 precursor proteins play an active role in NF-kappa B-mediated signal transduction.

M. Karin, F. Mercurio, C. Rosette, 1993, Genes & development.

Phosphorylation of I kappa B alpha precedes but is not sufficient for its dissociation from NF-kappa B

M. Karin, F. Mercurio, J. DiDonato, 1995, Molecular and cellular biology.

Interleukin-1 stimulates and all-trans-retinoic acid inhibits collagenase gene expression through its 5' activator protein-1-binding site.

M. Sporn, A. Roberts, M. Karin, 1990, Molecular endocrinology.

Negative transcriptional regulation by nuclear receptors.

M. Karin, F. Saatcioglu, F. Claret, 1994, Seminars in cancer biology.

BCL3 encodes a nuclear protein which can alter the subcellular location of NF-kappa B proteins

M. Karin, Q. Zhang, T. McKeithan, 1994, Molecular and cellular biology.

Molecular cloning and characterization of human JNKK2, a novel Jun NH2-terminal kinase-specific kinase

M. Karin, Z. Wu, E. Jacinto, 1997, Molecular and cellular biology.

Oncogenic Ras activates c-Jun via a separate pathway from the activation of extracellular signal-regulated kinases.

D. Brenner, M. Karin, C. Der, 1994, Proceedings of the National Academy of Sciences of the United States of America.

Potentiation of estrogen receptor activation function 1 (AF-1) by Src/JNK through a serine 118-independent pathway.

M. Karin, P. Webb, P. Kushner, 2001, Molecular endocrinology.

Cytoskeletal control of gene expression: depolymerization of microtubules activates NF-kappa B

M. Karin, C. Rosette, Michael Karin, 1995, The Journal of cell biology.

Mutations in the conserved C-terminal sequence in thyroid hormone receptor dissociate hormone-dependent activation from interference with AP-1 activity

E. Zandi, M. Karin, J. Baxter, 1997, Molecular and cellular biology.

Hypoestoxide, a novel anti-inflammatory natural diterpene, inhibits the activity of IkappaB kinase.

P. Kapahi, M. Karin, H. Cottam, 2001, Cellular immunology.

IkappaB kinase alpha is essential for development of the mammalian cornea and conjunctiva.

M. Karin, Y. Hu, K. Yoshida, 2000, Investigative ophthalmology & visual science.