Deniz A. Ucar

发表

Evaluation of deacetylase inhibition in metaplastic breast carcinoma using multiple derivations of preclinical models of a new patient-derived tumor

H. Wathieu, S. Elliott, B. Collins-Burow, 2019, bioRxiv.

Sulindac sulfide as a non-immune suppressive γ-secretase modulator to target triple-negative breast cancer

B. Collins-Burow, D. Wyczechowska, C. Hicks, 2023, Frontiers in immunology.

Targeting the Cbl-b-Notch1 axis as a novel immunotherapeutic strategy to boost CD8+ T-cell responses

A. Boulares, B. Osborne, L. Miele, 2022, Frontiers in Immunology.

Notch Signaling Regulates Mitochondrial Metabolism and NF-κB Activity in Triple-Negative Breast Cancer Cells via IKKα-Dependent Non-canonical Pathways

J. Crabtree, T. Golde, L. Minter, 2018, Front. Oncol..

Focal adhesion kinase signaling and function in pancreatic cancer.

S. Hochwald, L. Dang, D. Ucar, 2011, Frontiers in bioscience.

The enzymatic activity of human aldehyde dehydrogenases 1A2 and 2 (ALDH1A2 and ALDH2) is detected by Aldefluor, inhibited by diethylaminobenzaldehyde and has significant effects on cell proliferation and drug resistance.

Lung-Ji Chang, Shuhong Han, J. Amory, 2012, Chemico-Biological Interactions.

Molecular Mechanisms of Epithelial to Mesenchymal Transition Regulated by ERK5 Signaling

Jane E. Cavanaugh, M. Burow, L. Miele, 2021, Biomolecules.

Treatment of hypertriglyceridemia-induced acute pancreatitis with insulin

E. Oymacı, S. Yakan, M. Yıldırım, 2015, Przeglad gastroenterologiczny.

Notch Signaling in Myeloid Cells as a Regulator of Tumor Immune Responses

T. Golde, L. Minter, B. Osborne, 2018, Front. Immunol..

Aldehyde dehydrogenase activity as a functional marker for lung cancer.

E. Scott, R. Zori, C. Cogle, 2009, Chemico-biological interactions.

Elevated expression of IRS2 in the progression from neurofibroma to malignant peripheral nerve sheath tumor.

W. Cance, S. Hochwald, S. Grobmyer, 2012, Anticancer research.

Bone Marrow Contributes to Epithelial Cancers in Mice and Humans as Developmental Mimicry

E. Scott, C. Cogle, N. Theise, 2007, Stem cells.

A FAK scaffold inhibitor disrupts FAK and VEGFR-3 signaling and blocks melanoma growth by targeting both tumor and endothelial cells

W. Bshara, M. Seshadri, U. Sunar, 2014, Cell cycle.

ERK5 Is Required for Tumor Growth and Maintenance Through Regulation of the Extracellular Matrix in Triple Negative Breast Cancer

K. Nephew, L. Del Valle, Jane E. Cavanaugh, 2020, Frontiers in Oncology.

Targeting of the protein interaction site between FAK and IGF-1R.

A. Magis, V. Golubovskaya, W. Cance, 2009, Biochemical and biophysical research communications.

Evaluation of deacetylase inhibition in metaplastic breast carcinoma using multiple derivations of preclinical models of a new patient-derived tumor

H. Wathieu, S. Elliott, B. Collins-Burow, 2019, bioRxiv.

Correction: Evaluation of deacetylase inhibition in metaplastic breast carcinoma using multiple derivations of preclinical models of a new patient-derived tumor

H. Wathieu, S. Elliott, B. Collins-Burow, 2021, PLoS ONE.

Dual inhibition of MEK1/2 and MEK5 suppresses the EMT/migration axis in triple‐negative breast cancer through FRA‐1 regulation

K. Nephew, D. Rusch, D. Chrisey, 2021, Journal of cellular biochemistry.

Diverse and converging roles of ERK1/2 and ERK5 pathways on mesenchymal to epithelial transition in breast cancer

Jane E. Cavanaugh, M. Burow, L. Miele, 2021, Translational oncology.

Targeting Cblb-Notch1 axis as a novel immunotherapeutic strategy to boost CD8+ T-cells responses

A. Boulares, B. Osborne, L. Miele, 2022, bioRxiv.