Mark A. Israel

Cell cycle synchrony unmasks the influence of p53 function on radiosensitivity of human glioblastoma cells.

W. Dewey, M. Israel, M. Haas, 1996, Cancer research.

The intrinsic radioresistance of glioblastoma-derived cell lines is associated with a failure of p53 to induce p21(BAX) expression.

M. Israel, H. Shu, C. Julin, 1998, Proceedings of the National Academy of Sciences of the United States of America.

EGFR overexpression and radiation response in glioblastoma multiforme.

M. Berger, K. Aldape, M. Prados, 2001, International journal of radiation oncology, biology, physics.

p53-dependent G1 arrest and p53-independent apoptosis influence the radiobiologic response of glioblastoma.

W. Dewey, M. Israel, M. Haas, 1996, International journal of radiation oncology, biology, physics.

Ionizing radiation inhibits chemotherapy-induced apoptosis in cultured glioma cells: implications for combined modality therapy.

K. Aldape, M. Israel, D. Haas-Kogan, 1998, Cancer research.

p53 function influences the effect of fractionated radiotherapy on glioblastoma tumors.

M. Israel, M. Haas, D. Haas-Kogan, 1999, International journal of radiation oncology, biology, physics.