Katharina Baebler

Intestinal Activation of pH-Sensing Receptor OGR1 [GPR68] Contributes to Fibrogenesis

G. Rogler, M. Hausmann, C. Wagner, 2018, Journal of Crohn's & colitis.

Biomarkers, Models and Mechanisms of Intestinal Fibrosis

M. Hausmann, I. Frey-Wagner, B. Misselwitz, 2022 .

Iron Prevents Hypoxia-Associated Inflammation Through the Regulation of Nuclear Factor-κB in the Intestinal Epithelium

G. Rogler, S. Vavricka, M. Hausmann, 2018, Cellular and molecular gastroenterology and hepatology.

Activation of pH-Sensing Receptor OGR1 (GPR68) Induces ER Stress Via the IRE1α/JNK Pathway in an Intestinal Epithelial Cell Model

G. Rogler, M. Hausmann, L. Hering, 2020, Scientific Reports.

New Therapeutic Approach for Intestinal Fibrosis Through Inhibition of pH-Sensing Receptor GPR4.

G. Rogler, K. Seuwen, S. Mueller, 2021, Inflammatory bowel diseases.

Commensal Clostridiales strains mediate effective anti-cancer immune response against solid tumors.

G. Zeller, Nicola Zamboni, M. Krauthammer, 2021, Cell host & microbe.

Lack of the pH-sensing Receptor TDAG8 [GPR65] in Macrophages Plays a Detrimental Role in Murine Models of Inflammatory Bowel Disease

G. Russo, G. Rogler, K. Seuwen, 2018, Journal of Crohn's & colitis.

TiO2 nanoparticles abrogate the protective effect of the Crohn’s disease-associated variation within the PTPN22 gene locus

G. Rogler, S. Zeissig, A. Moor, 2022, Gut.

pH-Sensing G Protein-Coupled Receptor OGR1 (GPR68) Expression and Activation Increases in Intestinal Inflammation and Fibrosis

G. Rogler, K. Seuwen, M. Hausmann, 2022, International journal of molecular sciences.