Sho Yoshida

The phospholipase-A2 reaction leads to increased monocyte adhesion of endothelial cells via the expression of adhesion molecules.

K. Yokote, N. Morisaki, Y. Saito, 1993, European journal of biochemistry.

Type I hyperlipoproteinemia caused by lipoprotein lipase defect in lipid-interface recognition was relieved by administration of medium-chain triglyceride.

H. Inadera, K. Shirai, J. Kobayashi, 1992, Metabolism: clinical and experimental.

A heterozygous mutation (the codon for Ser447----a stop codon) in lipoprotein lipase contributes to a defect in lipid interface recognition in a case with type I hyperlipidemia.

K. Shirai, J. Kobayashi, M. Schotz, 1992, Biochemical and biophysical research communications.

Effects of transforming growth factor-beta 1 on growth of aortic smooth muscle cells. Influences of interaction with growth factors, cell state, cell phenotype, and cell cycle.

N. Morisaki, M. Kawano, Y. Saito, 1991, Atherosclerosis.

Inhibition of intimal thickening of the carotid artery of rabbits and of outgrowth of explants of aorta by probucol.

M. Shinomiya, K. Shirai, Y. Saito, 1992, Atherosclerosis.