David L. Allen

Growth hormone/IGF-I and/or resistive exercise maintains myonuclear number in hindlimb unweighted muscles.

R E Grindeland, V R Edgerton, R R Roy, 1997, Journal of applied physiology.

Apoptosis: a mechanism contributing to remodeling of skeletal muscle in response to hindlimb unweighting.

R E Grindeland, V R Edgerton, R R Roy, 1997, The American journal of physiology.

Modulation of myonuclear number in functionally overloaded and exercised rat plantaris fibers.

V R Edgerton, R R Roy, V. Edgerton, 1999, Journal of applied physiology.

Maintenance of myonuclear domain size in rat soleus after overload and growth hormone/IGF-I treatment.

V. Edgerton, D. Allen, R. Roy, 1998, Journal of applied physiology.