George Battaglia

Evidence That Gz-Proteins Couple to Hypothalamic 5-HT1A Receptors In Vivo

L. D. van de Kar, G. Battaglia, F. García, 2000, The Journal of Neuroscience.

Effect of prenatal fluoxetine (Prozac) exposure on brain serotonin neurons in prepubescent and adult male rat offspring.

G. Battaglia, F. García, T. M. Cabrera-Vera, 1997, The Journal of pharmacology and experimental therapeutics.

Is dopamine a neurotransmitter within the ventral pallidum/substantia innominata?

G. Battaglia, T. Napier, R. Maslowski, 1991, Advances in experimental medicine and biology.

Daily injections of fluoxetine induce dose-dependent desensitization of hypothalamic 5-HT1A receptors: reductions in neuroendocrine responses to 8-OH-DPAT and in levels of Gz and Gi proteins.

L. D. van de Kar, G. Battaglia, F. García, 1999, The Journal of pharmacology and experimental therapeutics.

Fluoxetine gradually increases [125I]DOI-labelled 5-HT2A/2C receptors in the hypothalamus without changing the levels of Gq- and G11-proteins

G. Battaglia, Q. Li, L. Kar, 1997, Brain Research.

Methylphenidate and pemoline do not cause depletion of rat brain monoamine markers similar to that observed with methamphetamine.

J. Contrera, E. D. De Souza, G. Battaglia, 1989, Toxicology and applied pharmacology.

Activation of the JAK-STAT pathway by olanzapine is necessary for desensitization of serotonin2A receptor-stimulated phospholipase C signaling in rat frontal cortex but not serotonin2A receptor-stimulated hormone release

G. Battaglia, F. García, G. Carrasco, 2010, Journal of psychopharmacology.