Andrew R. Tee

发表

Oncogenic Signalling through Mechanistic Target of Rapamycin (mTOR): A Driver of Metabolic Transformation and Cancer Progression

J. Murray, A. Tee, Ellie Rad, 2018, Cancers.

Endoplasmic reticulum stress and cell death in mTORC1‐overactive cells is induced by nelfinavir and enhanced by chloroquine

R. Errington, A. Tee, J. Sampson, 2015, Molecular oncology.

Reciprocal signaling between mTORC1 and MNK2 controls cell growth and oncogenesis

Shuye Tian, A. Zannettino, S. Fitter, 2020, Cellular and Molecular Life Sciences.

Tuberous Sclerosis Complex cell‐derived EVs have an altered protein cargo capable of regulating their microenvironment and have potential as disease biomarkers

A. Tee, E. Dunlop, J. Webber, 2023, Journal of extracellular vesicles.

Drug Inhibition of Redox Factor-1 Restores Hypoxia-Driven Changes in Tuberous Sclerosis Complex 2 Deficient Cells

E. Henske, A. Tee, D. M. Davies, 2022, Cancers.

Targeting protein homeostasis with nelfinavir/salinomycin dual therapy effectively induces death of mTORC1 hyperactive cells

R. Errington, A. Tee, M. Wiltshire, 2017, Oncotarget.

Finding a cure for tuberous sclerosis complex: From genetics through to targeted drug therapies.

A. Tee, Lauren J McEneaney, Andrew R. Tee, 2019, Advances in genetics.

The Target of Rapamycin and Mechanisms of Cell Growth

A. Tee, Andrew R Tee, Andrew R. Tee, 2018, International journal of molecular sciences.

Exploiting cancer vulnerabilities: mTOR, autophagy, and homeostatic imbalance.

A. Tee, Charlotte E. Johnson, Andrew R. Tee, 2017, Essays in biochemistry.

Energy Stress-Mediated Cytotoxicity in Tuberous Sclerosis Complex 2-Deficient Cells with Nelfinavir and Mefloquine Treatment

R. Errington, A. Tee, D. M. Davies, 2018, Cancers.