S. I. Levin

发表

Causes and frequency of failures on gas mains in the USSR

S. I. Levin, V. V. Kharionovsky, 1993 .

A Novel Epilepsy Mutation in the Sodium Channel SCN1A Identifies a Cytoplasmic Domain for β Subunit Interaction

S. I. Levin, A. L. Goldin, I. Soltesz, 2004, The Journal of Neuroscience.

Molecular and pathological effects of a modifier gene on deficiency of the sodium channel Scn8a (Na(v)1.6).

S. I. Levin, R. Albin, G. de Haan, 2002, Human molecular genetics.

Impact of anaesthetics and analgesics on fetal growth in the mouse

S. I. Levin, L. Thaete, A. Dudley, 2013, Laboratory animals.

Floxed allele for conditional inactivation of the voltage‐gated sodium channel Scn8a (Nav1.6)

S. I. Levin, M. Meisler, Stephen I. Levin, 2004, Genesis.

TECHNOLOGY REPORT Floxed Allele for Conditional Inactivation of the Voltage-Gated Sodium Channel Scn8a (Na V 1.6)

S. I. Levin, M. Meisler, Stephen I. Levin, 2004 .

Biology and Diseases of Dogs

S. I. Levin, J. Nemzek, M. Moalli, 2002, Laboratory Animal Medicine.

Management, Husbandry, and Colony Health

Stephen I. Levin, Diana M. Palila Berger, S. I. Levin, 2012 .

Impaired motor function in mice with cell-specific knockout of sodium channel Scn8a (NaV1.6) in cerebellar purkinje neurons and granule cells.

S. I. Levin, Zayd M. Khaliq, I. Raman, 2006, Journal of neurophysiology.

Inactivation of sodium channel Scn8A (Na-sub(v)1.6) in Purkinje neurons impairs learning in Morris water maze and delay but not trace eyeblink classical conditioning.

S. I. Levin, M. Meisler, D. Woodruff-Pak, 2006, Behavioral neuroscience.

Cerebellar Purkinje Neurons and Granule Cells 1.6) in V (Na Scn8a Knockout of Sodium Channel Impaired Motor Function in Mice With Cell-Specific

S. I. Levin, Zayd M. Khaliq, C. Semsarian, 2014 .