R. Gilmour

Protein Kinase C α-mediated Negative Feedback Regulation Is Responsible for the Termination of Insulin-like Growth Factor I-induced Activation of Nuclear Phospholipase C β1 in Swiss 3T3 Cells*

A. Xu, Yu Wang, R. Gilmour, 2001, The Journal of Biological Chemistry.

Nuclear phospholipase C and signaling.

S. Rhee, L. Cocco, A. Martelli, 2001, Biochimica et biophysica acta.

Inositides in the nucleus: taking stock of PLCβ1

K. Wirtz, S. Capitani, S. Rhee, 1998 .

Calcium free inositol (1,4,5)-trisphosphate stimulates protein kinase C dependent protein phosphorylation in nuclei isolated from mitogen-treated Swiss 3T3 cells.

A. Martelli, L. Cocco, F. Manzoli, 1990, Biochemical and biophysical research communications.

Mitogen-stimulated phosphorylation of nuclear proteins in Swiss 3T3 cells: evidence for a protein kinase C requirement.

A. Martelli, L. Cocco, F. Manzoli, 1989, Experimental cell research.

Mitogen‐stimulated events in nuclei of Swiss 3T3 cells Evidence for a direct link between changes of inositol lipids, protein kinase C requirement and the onset of DNA synthesis

A. N. Corps, A. Martelli, L. Manzoli, 1991, FEBS letters.

Inositol lipid cycle and autonomous nuclear signalling.

S. Capitani, L. Cocco, N. Maraldi, 1996, Advances in enzyme regulation.

Expression and immunohistochemical localization of eight phospholipase C isoforms in adult male mouse cerebellar cortex.

A. Martelli, L. Cocco, R. Gilmour, 1996, Acta histochemica.