L. R. Stewart

Diverse fibrillar peptides directly bind the Alzheimer’s amyloid precursor protein and amyloid precursor-like protein 2 resulting in cellular accumulation

C. Masters, G. Multhaup, K. Beyreuther, 2003, Brain Research.

Neonatal lethality in transgenic mice expressing prion protein with a deletion of residues 105–125

K. Roth, D. Harris, R. Chiesa, 2007, The EMBO journal.

Involvement of the 5‐lipoxygenase pathway in the neurotoxicity of the prion peptide PrP106‐126

C. Masters, K. Beyreuther, R. Cappai, 2001, Journal of neuroscience research.

The Hydrophobic Core Sequence Modulates the Neurotoxic and Secondary Structure Properties of the Prion Peptide 106‐126

Fran Maher, Anna Friedhuber, Roberto Cappai, 1999, Journal of neurochemistry.

Mutant PrP Is Delayed in Its Exit from the Endoplasmic Reticulum, but Neither Wild-type nor Mutant PrP Undergoes Retrotranslocation Prior to Proteasomal Degradation*

R. S. Stewart, D. Harris, R. Chiesa, 2003, Journal of Biological Chemistry.

Prion protein-deficient neurons reveal lower glutathione reductase activity and increased susceptibility to hydrogen peroxide toxicity.

K. Beyreuther, R. Cappai, C. Masters, 1999, The American journal of pathology.

Copper and zinc binding modulates the aggregation and neurotoxic properties of the prion peptide PrP106-126.

A. Bush, R. Cherny, R. Cappai, 2001, Biochemistry.

Cytosolic Prion Protein (PrP) Is Not Toxic in N2a Cells and Primary Neurons Expressing Pathogenic PrP Mutations*

R. S. Stewart, G. Forloni, D. Harris, 2005, Journal of Biological Chemistry.