John A Todd

发表

Fine-mapping, trans-ancestral, and genomic analyses identify causal variants, cells, genes, and drug targets for type 1 diabetes

Amy S. Shah, Catherine C. Robertson, P. Deloukas, 2020, Nature Genetics.

Congenic mapping of the insulin-dependent diabetes (Idd) gene, Idd10, localizes two genes mediating the Idd10 effect and eliminates the candidate Fcgr1.

J. Todd, P. Lyons, P. Denny, 1997, Journal of immunology.

SARS-CoV-2 infection induces a long-lived pro-inflammatory transcriptional profile

R. Ferreira, L. Wicker, J. Whalley, 2023, Genome Medicine.

This information is current as Diabetes Gene in Nonobese Diabetic Mice Is an Autoimmune Cd 101 Evidence

C. Steward, O. Burren, E. Masteller, 2011 .

Congenic mapping of the type 1 diabetes locus, Idd3, to a 780-kb region of mouse chromosome 3: identification of a candidate segment of ancestral DNA by haplotype mapping.

J. Todd, P. Lyons, P. Denny, 2000, Genome research.

Resistance alleles at two non-major histocompatibility complex-linked insulin-dependent diabetes loci on chromosome 3, Idd3 and Idd10, protect nonobese diabetic mice from diabetes

J. Todd, L. Wicker, L. Peterson, 1994, The Journal of experimental medicine.

NOD Idd5 locus controls insulitis and diabetes and overlaps the orthologous CTLA4/IDDM12 and NRAMP1 loci in humans.

J. Todd, P. Lyons, L. Wicker, 2000, Diabetes.

The NOD Idd9 genetic interval influences the pathogenicity of insulitis and contains molecular variants of Cd30, Tnfr2, and Cd137.

J. Todd, M. Phillips, P. Lyons, 2000, Immunity.

COVID-19 due to the B.1.617.2 (Delta) variant compared to B.1.1.7 (Alpha) variant of SARS-CoV-2: a prospective observational cohort study

Scott M Elliott, Kevin R. Cole, Robert M. Davies, 2022, Scientific Reports.