Alba Llop-Guevara

发表

A GM-CSF/IL-33 Pathway Facilitates Allergic Airway Responses to Sub-Threshold House Dust Mite Exposure

M. Jordana, P. O'Byrne, M. Stämpfli, 2014, PloS one.

Indigenous enteric eosinophils control DCs to initiate a primary Th2 immune response in vivo

M. Jordana, C. Verschoor, K. McCoy, 2014, The Journal of experimental medicine.

In Vivo-to-In Silico Iterations to Investigate Aeroallergen-Host Interactions

Miroslav Lovric, M. Jordana, P. O'Byrne, 2008, PloS one.

A Mouse Model of Airway Disease: Oncostatin M-Induced Pulmonary Eosinophilia, Goblet Cell Hyperplasia, and Airway Hyperresponsiveness Are STAT6 Dependent, and Interstitial Pulmonary Fibrosis Is STAT6 Independent

M. Jordana, W. Khan, A. Llop-Guevara, 2011, The Journal of Immunology.

Interleukin 3, Interleukin 5, and Granulocyte–Macrophage Colony‐Stimulating Factor

M. Jordana, A. Llop-Guevara, R. Fattouh, 2011 .

Mechanisms of allergy and clinical immunology IL-33 , but not thymic stromal lymphopoietin or IL-25 , is central to mite and peanut allergic sensitization

M. Jordana, J. Boudreau, D. Chu, 2022 .

Fibrosis Is STAT6 Independent Dependent, and Interstitial Pulmonary Airway Hyperresponsiveness Are STAT6 Eosinophilia, Goblet Cell Hyperplasia, and Oncostatin M-Induced Pulmonary A Mouse Model of Airway Disease:

M. Jordana, W. Khan, A. Llop-Guevara, 2010 .

T helper cell IL-4 drives intestinal Th2 priming to oral peanut antigen, under the control of OX40L and independent of innate-like lymphocytes

M. Jordana, W. Khan, A. Ashkar, 2014, Mucosal Immunology.

Modeling responses to respiratory house dust mite exposure.

M. Jordana, A. Llop-Guevara, R. Fattouh, 2007, Contributions to microbiology.