A. Segal

发表

Direct interaction between p47phox and protein kinase C: evidence for targeting of protein kinase C by p47phox in neutrophils.

L. Dekker, D. Pappin, A. Segal, 1999, The Biochemical journal.

The major phosphorylation site of the NADPH oxidase component p67phox is Thr233.

Oanh Truong, Anthony W. Segal, Louisa V. Forbes, 1999, The Biochemical journal.

Reconstitution of neutrophil NADPH oxidase activity in the cell-free system by four components: p67-phox, p47-phox, p21rac1, and cytochrome b-245.

A. Abo, A. Segal, A. Thrasher, 1992, The Journal of biological chemistry.

NADPH oxidase and the respiratory burst.

A. Segal, F. Wientjes, Anthony W. Segal, 1995, Seminars in cell biology.

Chronic granulomatous disease.

A. Segal, A W Segal, Anthony W. Segal, 1994, Biochimica et biophysica acta.

Activation of NADPH oxidase involves the dissociation of p21rac from its inhibitory GDP/GTP exchange protein (rhoGDI) followed by its translocation to the plasma membrane.

A. Abo, A. Segal, M. Webb, 1994, The Biochemical journal.

The function of the NADPH oxidase of phagocytes, and its relationship to other NOXs.

A. Segal, P. Béhé, A. Segal, 2007, Biochemical Society transactions.

Cantharidin blisters: a technique for investigating leukocyte trafficking and cytokine production at sites of inflammation in humans.

A. Forbes, A. Segal, R. Day, 2001, Journal of immunological methods.

The antimicrobial role of the neutrophil leukocyte.

A. Segal, A. Segal, 1981, The Journal of infection.

Autosomal recessive chronic granulomatous disease caused by deletion at a dinucleotide repeat.

D. Bentley, A. Segal, A. Rodaway, 1991, Proceedings of the National Academy of Sciences of the United States of America.

Protein kinase C-beta contributes to NADPH oxidase activation in neutrophils.

L. Dekker, J. Roes, A. Segal, 2000, The Biochemical journal.