Ki-Yon Kim

Extracellular signal-regulated protein kinase, but not c-Jun N-terminal kinase, is activated by type II gonadotropin-releasing hormone involved in the inhibition of ovarian cancer cell proliferation.

P. Leung, N. Auersperg, Ki-Yon Kim, 2005, The Journal of clinical endocrinology and metabolism.

Mechanism of gonadotropin-releasing hormone (GnRH)-I and -II-induced cell growth inhibition in ovarian cancer cells: role of the GnRH-I receptor and protein kinase C pathway.

P. Leung, N. Auersperg, Ki-Yon Kim, 2006, Endocrine-related cancer.

Type II gonadotropin-releasing hormone stimulates p38 mitogen-activated protein kinase and apoptosis in ovarian cancer cells.

P. Leung, N. Auersperg, Ki-Yon Kim, 2004, The Journal of clinical endocrinology and metabolism.

Conditional knockout of brca1/2 and p53 in mouse ovarian surface epithelium: Do they play a role in ovarian carcinogenesis?

Ki-Yon Kim, Kyung-Chul Choi, E. Jeung, 2010, Journal of veterinary science.

Cell growth of ovarian cancer cells is stimulated by xenoestrogens through an estrogen-dependent pathway, but their stimulation of cell growth appears not to be involved in the activation of the mitogen-activated protein kinases ERK-1 and p38.

P. Leung, Jung-Hye Choi, B. An, 2009, The Journal of reproduction and development.

Influence of the prodrugs 5‐fluorocytosine and CPT‐11 on ovarian cancer cells using genetically engineered stem cells: tumor‐tropic potential and inhibition of ovarian cancer cell growth

P. Leung, Ki-Yon Kim, Kyung-Chul Choi, 2010, Cancer science.