Xiao-Jing Wang

发表

Distinct roles of individual Smads in skin carcinogenesis

Shi-Long Lu, S. Bornstein, G. Han, 2007, Molecular carcinogenesis.

Overexpression of Smad7 results in severe pathological alterations in multiple epithelial tissues

M. Goumans, Xiao‐Jing Wang, P. Dijke, 2002, The EMBO journal.

MicroRNA-31 Is Overexpressed in Psoriasis and Modulates Inflammatory Cytokine and Chemokine Production in Keratinocytes via Targeting Serine/Threonine Kinase 40

A. Pivarcsi, C. Söderberg-Nauclér, E. Sonkoly, 2013, The Journal of Immunology.

Involvement of IL-9 in Th17-Associated Inflammation and Angiogenesis of Psoriasis

P. Wolf, M. Schön, A. Gruber‐Wackernagel, 2013, PloS one.

The Pro-inflammatory Role of TGFβ1: A Paradox?

P. Wolf, G. Han, Fulun Li, 2012, International journal of biological sciences.

8-Methoxypsoralen Plus Ultraviolet A Therapy Acts via Inhibition of the IL-23/Th17 Axis and Induction of Foxp3+ Regulatory T Cells Involving CTLA4 Signaling in a Psoriasis-Like Skin Disorder

P. Wolf, M. Schön, B. Rinner, 2010, The Journal of Immunology.

Latent TGFβ1 overexpression in keratinocytes results in a severe psoriasis‐like skin disorder

Xin-Hua Feng, Allen Li, Allen G Li, 2004, The EMBO journal.

Keratin promoter based gene manipulation in the murine conducting airway

S. Malkoski, Shi-Long Lu, Timothy G. Cleaver, 2010, International Journal on Biological Sciences.

Smad4 loss promotes lung cancer formation but increases sensitivity to DNA topoisomerase inhibitors

S. Malkoski, Xiao-Jing Wang, S. Haeger, 2015, Oncogene.

Role of PTEN in basal cell derived lung carcinogenesis

S. Malkoski, Shi-Long Lu, S. Haeger, 2014, Molecular carcinogenesis.

Loss of Transforming Growth Factor Beta Type II Receptor Increases Aggressive Tumor Behavior and Reduces Survival in Lung Adenocarcinoma and Squamous Cell Carcinoma

W. Franklin, A. Baron, H. Ijichi, 2012, Clinical Cancer Research.

Human Cancer Biology Loss of Transforming Growth Factor Beta Type II Receptor Increases Aggressive Tumor Behavior and Reduces Survival in Lung Adenocarcinoma and Squamous Cell Carcinoma

W. Franklin, H. Ijichi, S. Malkoski, 2012 .

Smad4 loss in mice causes spontaneous head and neck cancer with increased genomic instability and inflammation.

C. Deng, S. Malkoski, S. Olson, 2009, The Journal of clinical investigation.

Inducible Activation of Oncogenic K-ras Results in Tumor Formation in the Oral Cavity

D. Roop, C. Caulin, Thao Nguyen, 2004, Cancer Research.

Genome-Wide Expression Profiling of Five Mouse Models Identifies Similarities and Differences with Human Psoriasis

Andrew Johnston, William R. Swindell, Johann E. Gudjonsson, 2011, PloS one.

CIP2A Inhibits PP2A in Human Malignancies

R. Sears, J. Westermarck, R. Grenman, 2007, Cell.

An Inducible Mouse Model for Epidermolysis Bullosa Simplex

D. Roop, M. Longley, T. Cao, 2001, The Journal of cell biology.

The role of Smad7 in oral mucositis

P. Garl, L. Bian, G. Han, 2015, Protein & Cell.

Preventive and therapeutic effects of Smad7 on radiation-induced oral mucositis

James B. Mitchell, D. Raben, P. Dijke, 2013, Nature Medicine.

Acute Lymphoblastic Leukemia Cells Inhibit the Differentiation of Bone Mesenchymal Stem Cells into Osteoblasts In Vitro by Activating Notch Signaling

Gui-cun Yang, You-hua Xu, Xiao-Jing Wang, 2015, Stem cells international.

HGF upregulation contributes to angiogenesis in mice with keratinocyte-specific Smad2 deletion.

E. Bottinger, Qinghong Zhang, Masako Oka, 2010, The Journal of clinical investigation.

Keratinocyte-specific Smad2 ablation results in increased epithelial-mesenchymal transition during skin cancer formation and progression.

E. Bottinger, W. Ju, M. Kulesz-Martin, 2008, The Journal of clinical investigation.