Stephen G Waxman

发表

Chair's introduction: sodium channels and neuronal dysfunction--emerging concepts, converging themes.

Stephen G Waxman, 2002, Novartis Foundation symposium.

Abnormal sodium channel distribution in optic nerve axons in a model of inflammatory demyelination.

Stephen G Waxman, A. Lo, S. Waxman, 2003, Brain : a journal of neurology.

Voltage-gated Na+ channels in glia: properties and possible functions

Harald Sontheimer, Stephen G Waxman, S. Waxman, 1996, Trends in Neurosciences.

Molecular changes in neurons in multiple sclerosis: altered axonal expression of Nav1.2 and Nav1.6 sodium channels and Na+/Ca2+ exchanger.

Jia Newcombe, Stephen G Waxman, S. Waxman, 2004, Proceedings of the National Academy of Sciences of the United States of America.

Na+ channel expression along axons in multiple sclerosis and its models.

Stephen G Waxman, S. Waxman, Matthew J Craner, 2004, Trends in pharmacological sciences.

Co-localization of sodium channel Nav1.6 and the sodium-calcium exchanger at sites of axonal injury in the spinal cord in EAE.

Stephen G Waxman, A. Lo, S. Waxman, 2004, Brain : a journal of neurology.

Primary cortical motor neurons undergo apoptosis after axotomizing spinal cord injury

Stephen G Waxman, S. Waxman, J. A. Black, 2003, The Journal of comparative neurology.

Alterations in burst firing of thalamic VPL neurons and reversal by Na(v)1.3 antisense after spinal cord injury.

Stephen G Waxman, S. Waxman, B. C. Hains, 2006, Journal of neurophysiology.

Mesenchymal stem cells: therapeutic outlook for stroke.

Osamu Honmou, Stephen G Waxman, S. Waxman, 2012, Trends in molecular medicine.

A Nav1.7 channel mutation associated with hereditary erythromelalgia contributes to neuronal hyperexcitability and displays reduced lidocaine sensitivity

Stephen G Waxman, James O. Jackson, S. Dib-Hajj, 2007, The Journal of physiology.

Regulating excitability of peripheral afferents: emerging ion channel targets

Stephen G Waxman, S. Waxman, Gerald W Zamponi, 2014, Nature Neuroscience.

Intravenous administration of auto serum-expanded autologous mesenchymal stem cells in stroke.

Takuya Matsunaga, Osamu Honmou, Kiyohiro Houkin, 2011, Brain : a journal of neurology.

Temporal Course of Upregulation of Nav1.8 in Purkinje Neurons Parallels the Progression of Clinical Deficit in Experimental Allergic Encephalomyelitis

Stephen G Waxman, A. Lo, S. Waxman, 2003, Journal of neuropathology and experimental neurology.

GTP‐induced tetrodotoxin‐resistant Na+ current regulates excitability in mouse and rat small diameter sensory neurones

Stephen G Waxman, S. Waxman, J. Wood, 2003, The Journal of physiology.

Cerebellar dysfunction in multiple sclerosis: evidence for an acquired channelopathy.

Stephen G Waxman, S. Waxman, 2005, Progress in brain research.

Small-Fiber Neuropathy Nav1.8 Mutation Shifts Activation to Hyperpolarized Potentials and Increases Excitability of Dorsal Root Ganglion Neurons

Yang Yang, Stephen G Waxman, S. Dib-Hajj, 2013, The Journal of Neuroscience.

Long-term protection of central axons with phenytoin in monophasic and chronic-relapsing EAE.

Stephen G Waxman, S. Waxman, J. A. Black, 2006, Brain : a journal of neurology.