I Kodama

Mathematical models of action potentials in the periphery and center of the rabbit sinoatrial node.

H Zhang, A V Holden, I Kodama, 2000, American journal of physiology. Heart and circulatory physiology.

Arrhythmogenic changes in action potential configuration in the ventricle induced by DC shocks.

I Kodama, I Sakuma, J Toyama, 1999, Journal of electrocardiology.

Aftereffects of high-intensity DC stimulation on the electromechanical performance of ventricular muscle.

I Kodama, I Sakuma, J Toyama, 1994, The American journal of physiology.

Effects of long-term oral administration of amiodarone on the ventricular repolarization of rabbit hearts.

I Kodama, J Toyama, K Kamiya, 1996, Japanese circulation journal.

Pacemaker Shift in the Rabbit Sinoatrial Node in Response to Vagal Nerve Stimulation

I Kodama, H Honjo, M R Boyett, 2001, Experimental physiology.

Vesnarinone prolongs action potential duration without reverse frequency dependence in rabbit ventricular muscle by blocking the delayed rectifier K+ current.

I Kodama, J Toyama, K. Kamiya, 1997, Circulation.

Pacing-induced heart failure causes a reduction of delayed rectifier potassium currents along with decreases in calcium and transient outward currents in rabbit ventricle.

I Kodama, T Opthof, T. Opthof, 2000, Cardiovascular research.

Carvedilol blocks the repolarizing K+ currents and the L-type Ca2+ current in rabbit ventricular myocytes.

I Kodama, J Toyama, K. Kamiya, 1999, European journal of pharmacology.

Presence of the Kv1.5 K+ Channel in the Sinoatrial Node

I Kodama, M R Boyett, H Dobrzynski, 2000, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.

Open channel block of HERG K(+) channels by vesnarinone.

I Kodama, M. Sanguinetti, K. Kamiya, 2001, Molecular pharmacology.