Role and possible mechanisms of clenbuterol in enhancing reverse remodelling during mechanical unloading in murine heart failure
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M. Yacoub | P. Barton | L. Felkin | C. Terracciano | U. Siedlecka | M. A. Stagg | Gopal K. R. Soppa | Joon-Soek Lee | S. Youssef | Gopal K R Soppa
[1] Mahesh P. Gupta,et al. Factors controlling cardiac myosin-isoform shift during hypertrophy and heart failure. , 2007, Journal of molecular and cellular cardiology.
[2] M. Yacoub,et al. Clenbuterol affects rat ventricular myocyte contractility via an inhibitory G protein-mediated pathway , 2007 .
[3] O H Frazier,et al. Cardiac Improvement During Mechanical Circulatory Support: A Prospective Multicenter Study of the LVAD Working Group , 2007, Circulation.
[4] S. Day,et al. Tuning cardiac performance in ischemic heart disease and failure by modulating myofilament function , 2007, Journal of Molecular Medicine.
[5] Masashi Komeda,et al. Determination of optimal duration of mechanical unloading for failing hearts to achieve bridge to recovery in a rat heterotopic heart transplantation model. , 2007, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.
[6] Magdi H Yacoub,et al. A quantitative gene expression profile of matrix metalloproteinases (MMPS) and their inhibitors (TIMPS) in the myocardium of patients with deteriorating heart failure requiring left ventricular assist device support. , 2006, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.
[7] P. Royle,et al. Clinical and cost-effectiveness of left ventricular assist devices as a bridge to heart transplantation for people with end-stage heart failure: a systematic review and economic evaluation. , 2006, European heart journal.
[8] Magdi H Yacoub,et al. Left ventricular assist device and drug therapy for the reversal of heart failure. , 2006, The New England journal of medicine.
[9] D. Bers,et al. Regulation of Ca2+ and Na+ in Normal and Failing Cardiac Myocytes , 2006, Annals of the New York Academy of Sciences.
[10] Allison R. Schulman,et al. Effect of clenbuterol on cardiac and skeletal muscle function during left ventricular assist device support. , 2006, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.
[11] Allison R. Schulman,et al. β2-Adrenergic Stimulation Attenuates Left Ventricular Remodeling, Decreases Apoptosis, and Improves Calcium Homeostasis in a Rodent Model of Ischemic Cardiomyopathy , 2006, Journal of Pharmacology and Experimental Therapeutics.
[12] Atsushi Takeda,et al. Reverse remodeling following insertion of left ventricular assist devices (LVAD): a review of the morphological and molecular changes. , 2005, Cardiovascular research.
[13] E. Lakatta,et al. Pharmacological Stimulation of β 2-adrenergic Receptors (β 2AR) Enhances Therapeutic Effectiveness of β 1AR Blockade in Rodent Dilated Ischemic Cardiomyopathy , 2005, Heart Failure Reviews.
[14] I. Pinto,et al. [Left ventricular hypertrophy of athletes: adaptative physiologic response of the heart]. , 2005, Arquivos brasileiros de cardiologia.
[15] I. Pinto,et al. Hipertrofia ventricular esquerda do atleta: resposta adaptativa fisiológica do coração , 2005 .
[16] S. Matsuoka,et al. Does the β2-Agonist Clenbuterol Help to Maintain Myocardial Potential to Recover During Mechanical Unloading? , 2005, Circulation.
[17] R. Hetzer,et al. Long-Term Results in Patients With Idiopathic Dilated Cardiomyopathy After Weaning From Left Ventricular Assist Devices , 2005, Circulation.
[18] J. Schulz-Menger,et al. Letter regarding article by Sharkey et al, "Acute and reversible cardiomyopathy provoked by stress in women from the United States". , 2005, Circulation.
[19] S. Raj,et al. Letter regarding article by Adamson et al, "Continuous autonomic assessment in patients with symptomatic heart failure: prognostic value of heart rate variability measured by an implanted cardiac resynchronization device". , 2005, Circulation.
[20] D. Goldspink,et al. beta2-Adrenergic receptor stimulation in vivo induces apoptosis in the rat heart and soleus muscle. , 2005, Journal of applied physiology.
[21] S. Kong,et al. Genetic expression profiles during physiological and pathological cardiac hypertrophy and heart failure in rats. , 2005, Physiological genomics.
[22] M. Yacoub,et al. Effects of chronic administration of clenbuterol on function and metabolism of adult rat cardiac muscle. , 2005, American journal of physiology. Heart and circulatory physiology.
[23] M. Komeda,et al. Heterotopic Transplantation of the Failing Rat Heart as a Model of Left Ventricular Mechanical Unloading Toward Recovery , 2005, ASAIO journal.
[24] E. Lakatta,et al. Pharmacological stimulation of beta2-adrenergic receptors (beta2AR) enhances therapeutic effectiveness of beta1AR blockade in rodent dilated ischemic cardiomyopathy. , 2005, Heart Failure Reviews.
[25] E. Lakatta,et al. Beneficial Effects of Chronic Pharmacological Manipulation of &bgr;-Adrenoreceptor Subtype Signaling in Rodent Dilated Ischemic Cardiomyopathy , 2004, Circulation.
[26] A. Khaghani,et al. Clinical Recovery From End-Stage Heart Failure Using Left-Ventricular Assist Device and Pharmacological Therapy Correlates With Increased Sarcoplasmic Reticulum Calcium Content but Not With Regression of Cellular Hypertrophy , 2004, Circulation.
[27] Martin J. Lohse,et al. What Is the Role of &bgr;-Adrenergic Signaling in Heart Failure? , 2003, Circulation research.
[28] D. Schulze,et al. Protein Kinase A Hyperphosphorylation Increases Basal Current but Decreases &bgr;-Adrenergic Responsiveness of the Sarcolemmal Na+-Ca2+ Exchanger in Failing Pig Myocytes , 2003, Circulation research.
[29] S. Houser,et al. [Na+]i handling in the failing human heart. , 2003, Cardiovascular research.
[30] Kenta Ito,et al. Contractile Reserve and Calcium Regulation Are Depressed in Myocytes From Chronically Unloaded Hearts , 2003, Circulation.
[31] S. Silver,et al. Heart Failure , 1937, The New England journal of medicine.
[32] K. Margulies. Reversal mechanisms of left ventricular remodeling: lessons from left ventricular assist device experiments. , 2002, Journal of cardiac failure.
[33] Ø. Ellingsen,et al. Cardiomyocyte contractility and calcium handling partially recover after early deterioration during post-infarction failure in rat. , 2002, Acta physiologica Scandinavica.
[34] W. Koch,et al. Myocardial Gene Transfer and Overexpression of &bgr;2-Adrenergic Receptors Potentiates the Functional Recovery of Unloaded Failing Hearts , 2002, Circulation.
[35] M C Oz,et al. Long-term use of a left ventricular assist device for end-stage heart failure. , 2001, The New England journal of medicine.
[36] P. Steendijk,et al. Influence of clenbuterol treatment during six weeks of chronic right ventricular pressure overload as studied with pressure-volume analysis. , 2001, The Journal of thoracic and cardiovascular surgery.
[37] M. Yacoub,et al. Protection From Reperfusion Injury After Cardiac Transplantation by Inhibition of Adenosine Metabolism and Nucleotide Precursor Supply , 2001, Circulation.
[38] N. Smedira,et al. Mechanical Unloading Restores &bgr;-Adrenergic Responsiveness and Reverses Receptor Downregulation in the Failing Human Heart , 2001, Circulation.
[39] M. Yacoub. A novel strategy to maximize the efficacy of left ventricular assist devices as a bridge to recovery. , 2001, European heart journal.
[40] O. Carretero,et al. Echocardiographic assessment of cardiac function in conscious and anesthetized mice. , 1999, American journal of physiology. Heart and circulatory physiology.
[41] T. Myers,et al. Left ventricular assist system as a bridge to myocardial recovery. , 1999, The Annals of thoracic surgery.
[42] M. Yacoub,et al. Clenbuterol increases stroke power and contractile speed of skeletal muscle for cardiac assist. , 1999, Circulation.
[43] M Flannery,et al. Low incidence of myocardial recovery after left ventricular assist device implantation in patients with chronic heart failure. , 1998, Circulation.
[44] K. Boheler,et al. Clenbuterol induces cardiac hypertrophy with normal functional, morphological and molecular features. , 1998, Cardiovascular research.
[45] S. Chugh,et al. Isolated myocyte contractile function is normal in postinfarct remodeled rat heart with systolic dysfunction. , 1997, Circulation.
[46] K. Boheler,et al. Pharmacological modulation of pressure-overload cardiac hypertrophy: changes in ventricular function, extracellular matrix, and gene expression. , 1997, Circulation.
[47] S. Houser,et al. Cellular basis of contractile derangements of hypertrophied feline ventricular myocytes. , 1997, Journal of molecular and cellular cardiology.
[48] K Rakusan,et al. Transplantation-induced atrophy of normal and hypertrophic rat hearts: effect on cardiac myocytes and capillaries. , 1997, Journal of molecular and cellular cardiology.
[49] D. Tweedie,et al. The effects of changes to action potential duration on the calcium content of the sarcoplasmic reticulum in isolated guinea-pig ventricular myocytes , 1997, Pflügers Archiv.
[50] K. Spitzer,et al. Effect on the indo‐1 transient of applying Ca2+ channel blocker for a single beat in voltage‐clamped guinea‐pig cardiac myocytes. , 1996, The Journal of physiology.
[51] K. Boheler,et al. Clenbuterol induces hypertrophy of the latissimus dorsi muscle and heart in the rat with molecular and phenotypic changes. , 1995, Circulation.
[52] E. Sonnenblick,et al. Myocyte performance during evolution of myocardial infarction in rats: effects of propionyl-L-carnitine. , 1995, American Journal of Physiology.
[53] R A Bassani,et al. Calibration of indo-1 and resting intracellular [Ca]i in intact rabbit cardiac myocytes. , 1995, Biophysical journal.
[54] C. Vahl,et al. Intracellular calcium transient of working human myocardium of seven patients transplanted for congestive heart failure. , 1994, Circulation research.
[55] L. Toll,et al. L-type cardiac calcium channels in doxorubicin cardiomyopathy in rats morphological, biochemical, and functional correlations. , 1991, The Journal of clinical investigation.
[56] W Grossman,et al. Abnormal intracellular calcium handling in myocardium from patients with end-stage heart failure. , 1987, Circulation research.
[57] S. Hering,et al. Sodium current in freshly isolated and in cultured single rat myocardial cells: frequency and voltage-dependent block by mexiletine. , 1983, Journal of molecular and cellular cardiology.
[58] K Ono,et al. Improved technique of heart transplantation in rats. , 1969, The Journal of thoracic and cardiovascular surgery.