Heparan sulfate expression is affected by inflammatory stimuli in primary human endothelial cells

[1]  J. Zaia,et al.  WT1-dependent sulfatase expression maintains the normal glomerular filtration barrier. , 2011, Journal of the American Society of Nephrology : JASN.

[2]  E. Lewis,et al.  Reactive oxygen species mediate high glucose-induced heparanase-1 production and heparan sulphate proteoglycan degradation in human and rat endothelial cells: a potential role in the pathogenesis of atherosclerosis , 2011, Diabetologia.

[3]  J E Tooke,et al.  High glucose causes dysfunction of the human glomerular endothelial glycocalyx. , 2011, American journal of physiology. Renal physiology.

[4]  U. V. von Andrian,et al.  Endothelial heparan sulfate controls chemokine presentation in recruitment of lymphocytes and dendritic cells to lymph nodes. , 2010, Immunity.

[5]  T. Jenssen,et al.  Serglycin Is a Major Proteoglycan in Polarized Human Endothelial Cells and Is Implicated in the Secretion of the Chemokine GROα/CXCL1* , 2010, The Journal of Biological Chemistry.

[6]  S. Kennel,et al.  The Heparan Sulfate Motif (GlcNS6S-IdoA2S)3, Common in Heparin, Has a Strict Topography and Is Involved in Cell Behavior and Disease* , 2010, The Journal of Biological Chemistry.

[7]  M. Romero,et al.  Novel mechanisms of endothelial dysfunction in diabetes , 2010, Journal of cardiovascular disease research.

[8]  R. Esper,et al.  Endothelial dysfunction in normal and abnormal glucose metabolism. , 2008, Advances in cardiology.

[9]  Oscar Ochoa,et al.  Chemokines and Diabetic Wound Healing , 2007, Vascular.

[10]  Farhad R Danesh,et al.  Contribution of proteoglycans towards the integrated functions of renal glomerular capillaries: a historical perspective. , 2007, The American journal of pathology.

[11]  N. Nagai,et al.  Mice Deficient in Heparan Sulfate 6-O-Sulfotransferase-1 Exhibit Defective Heparan Sulfate Biosynthesis, Abnormal Placentation, and Late Embryonic Lethality* , 2007, Journal of Biological Chemistry.

[12]  P. Little,et al.  Vascular wall proteoglycan synthesis and structure as a target for the prevention of atherosclerosis , 2007, Vascular health and risk management.

[13]  C. Parish The role of heparan sulphate in inflammation , 2006, Nature Reviews Immunology.

[14]  Ann Marie Schmidt,et al.  Advanced glycation end products: sparking the development of diabetic vascular injury. , 2006, Circulation.

[15]  J. Borrebæk,et al.  Effect of hyperglycemic condition on proteoglycan secretion in cultured human endothelial cells , 2006, European journal of nutrition.

[16]  S. O. Kolset,et al.  High glucose and Nε-(carboxymethyl) lysine bovine serum albumin modulate release of matrix metalloproteinases in cultured human endothelial cells , 2006, European journal of nutrition.

[17]  Christopher J. Robinson,et al.  VEGF165-binding Sites within Heparan Sulfate Encompass Two Highly Sulfated Domains and Can Be Liberated by K5 Lyase* , 2006, Journal of Biological Chemistry.

[18]  K. Kimata,et al.  Heparin Regulates Vascular Endothelial Growth Factor165-dependent Mitogenic Activity, Tube Formation, and Its Receptor Phosphorylation of Human Endothelial Cells , 2005, Journal of Biological Chemistry.

[19]  S. Tyagi,et al.  The central role of vascular extracellular matrix and basement membrane remodeling in metabolic syndrome and type 2 diabetes : the matrix preloaded , 2015 .

[20]  I. Edwards,et al.  High-glucose-induced structural changes in the heparan sulfate proteoglycan, perlecan, of cultured human aortic endothelial cells. , 2004, Biochimica et biophysica acta.

[21]  C. Emerson,et al.  QSulf1 remodels the 6-O sulfation states of cell surface heparan sulfate proteoglycans to promote Wnt signaling , 2003, The Journal of cell biology.

[22]  B. Sumpio,et al.  Cells in focus: endothelial cell. , 2002, The international journal of biochemistry & cell biology.

[23]  A. Varki,et al.  Heparin's anti-inflammatory effects require glucosamine 6-O-sulfation and are mediated by blockade of L- and P-selectins. , 2002, The Journal of clinical investigation.

[24]  A. Groffen,et al.  Decreased Glomerular Expression of Agrin in Diabetic Nephropathy and Podocytes, Cultured in High Glucose Medium , 2001, Nephron Experimental Nephrology.

[25]  K. Prydz,et al.  The AGE product Nɛ–(carboxymethyl)lysine serum albumin is a modulator of proteoglycan expression in polarized cultured kidney epithelial cells , 2001, Diabetologia.

[26]  M. Salmivirta,et al.  Selectively Desulfated Heparin Inhibits Fibroblast Growth Factor-induced Mitogenicity and Angiogenesis* , 2000, The Journal of Biological Chemistry.

[27]  T. Ranheim,et al.  Stimulation of serglycin and CD44 mRNA expression in endothelial cells exposed to TNF-α and IL-1α , 1999 .

[28]  T. Ranheim,et al.  Stimulation of serglycin and CD44 mRNA expression in endothelial cells exposed to TNF-alpha and IL-1alpha. , 1999, Biochimica et biophysica acta.

[29]  U. Lindahl,et al.  Domain Structure of Heparan Sulfates from Bovine Organs* , 1996, The Journal of Biological Chemistry.

[30]  S. O. Kolset,et al.  Blood platelets stimulate the expression of chondroitin sulfate proteoglycan in human monocytes. , 1992, Blood.

[31]  M. Farquhar,et al.  Increased permeability of the glomerular basement membrane to ferritin after removal of glycosaminoglycans (heparan sulfate) by enzyme digestion , 1980, The Journal of cell biology.

[32]  J. Shively,et al.  Formation of anhydrosugars in the chemical depolymerization of heparin. , 1976, Biochemistry.

[33]  E. Jaffe,et al.  Culture of human endothelial cells derived from umbilical veins. Identification by morphologic and immunologic criteria. , 1973, The Journal of clinical investigation.

[34]  T BITTER,et al.  A modified uronic acid carbazole reaction. , 1962, Analytical biochemistry.