Heparan sulfate expression is affected by inflammatory stimuli in primary human endothelial cells
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[1] J. Zaia,et al. WT1-dependent sulfatase expression maintains the normal glomerular filtration barrier. , 2011, Journal of the American Society of Nephrology : JASN.
[2] E. Lewis,et al. Reactive oxygen species mediate high glucose-induced heparanase-1 production and heparan sulphate proteoglycan degradation in human and rat endothelial cells: a potential role in the pathogenesis of atherosclerosis , 2011, Diabetologia.
[3] J E Tooke,et al. High glucose causes dysfunction of the human glomerular endothelial glycocalyx. , 2011, American journal of physiology. Renal physiology.
[4] U. V. von Andrian,et al. Endothelial heparan sulfate controls chemokine presentation in recruitment of lymphocytes and dendritic cells to lymph nodes. , 2010, Immunity.
[5] T. Jenssen,et al. Serglycin Is a Major Proteoglycan in Polarized Human Endothelial Cells and Is Implicated in the Secretion of the Chemokine GROα/CXCL1* , 2010, The Journal of Biological Chemistry.
[6] S. Kennel,et al. The Heparan Sulfate Motif (GlcNS6S-IdoA2S)3, Common in Heparin, Has a Strict Topography and Is Involved in Cell Behavior and Disease* , 2010, The Journal of Biological Chemistry.
[7] M. Romero,et al. Novel mechanisms of endothelial dysfunction in diabetes , 2010, Journal of cardiovascular disease research.
[8] R. Esper,et al. Endothelial dysfunction in normal and abnormal glucose metabolism. , 2008, Advances in cardiology.
[9] Oscar Ochoa,et al. Chemokines and Diabetic Wound Healing , 2007, Vascular.
[10] Farhad R Danesh,et al. Contribution of proteoglycans towards the integrated functions of renal glomerular capillaries: a historical perspective. , 2007, The American journal of pathology.
[11] N. Nagai,et al. Mice Deficient in Heparan Sulfate 6-O-Sulfotransferase-1 Exhibit Defective Heparan Sulfate Biosynthesis, Abnormal Placentation, and Late Embryonic Lethality* , 2007, Journal of Biological Chemistry.
[12] P. Little,et al. Vascular wall proteoglycan synthesis and structure as a target for the prevention of atherosclerosis , 2007, Vascular health and risk management.
[13] C. Parish. The role of heparan sulphate in inflammation , 2006, Nature Reviews Immunology.
[14] Ann Marie Schmidt,et al. Advanced glycation end products: sparking the development of diabetic vascular injury. , 2006, Circulation.
[15] J. Borrebæk,et al. Effect of hyperglycemic condition on proteoglycan secretion in cultured human endothelial cells , 2006, European journal of nutrition.
[16] S. O. Kolset,et al. High glucose and Nε-(carboxymethyl) lysine bovine serum albumin modulate release of matrix metalloproteinases in cultured human endothelial cells , 2006, European journal of nutrition.
[17] Christopher J. Robinson,et al. VEGF165-binding Sites within Heparan Sulfate Encompass Two Highly Sulfated Domains and Can Be Liberated by K5 Lyase* , 2006, Journal of Biological Chemistry.
[18] K. Kimata,et al. Heparin Regulates Vascular Endothelial Growth Factor165-dependent Mitogenic Activity, Tube Formation, and Its Receptor Phosphorylation of Human Endothelial Cells , 2005, Journal of Biological Chemistry.
[19] S. Tyagi,et al. The central role of vascular extracellular matrix and basement membrane remodeling in metabolic syndrome and type 2 diabetes : the matrix preloaded , 2015 .
[20] I. Edwards,et al. High-glucose-induced structural changes in the heparan sulfate proteoglycan, perlecan, of cultured human aortic endothelial cells. , 2004, Biochimica et biophysica acta.
[21] C. Emerson,et al. QSulf1 remodels the 6-O sulfation states of cell surface heparan sulfate proteoglycans to promote Wnt signaling , 2003, The Journal of cell biology.
[22] B. Sumpio,et al. Cells in focus: endothelial cell. , 2002, The international journal of biochemistry & cell biology.
[23] A. Varki,et al. Heparin's anti-inflammatory effects require glucosamine 6-O-sulfation and are mediated by blockade of L- and P-selectins. , 2002, The Journal of clinical investigation.
[24] A. Groffen,et al. Decreased Glomerular Expression of Agrin in Diabetic Nephropathy and Podocytes, Cultured in High Glucose Medium , 2001, Nephron Experimental Nephrology.
[25] K. Prydz,et al. The AGE product Nɛ–(carboxymethyl)lysine serum albumin is a modulator of proteoglycan expression in polarized cultured kidney epithelial cells , 2001, Diabetologia.
[26] M. Salmivirta,et al. Selectively Desulfated Heparin Inhibits Fibroblast Growth Factor-induced Mitogenicity and Angiogenesis* , 2000, The Journal of Biological Chemistry.
[27] T. Ranheim,et al. Stimulation of serglycin and CD44 mRNA expression in endothelial cells exposed to TNF-α and IL-1α , 1999 .
[28] T. Ranheim,et al. Stimulation of serglycin and CD44 mRNA expression in endothelial cells exposed to TNF-alpha and IL-1alpha. , 1999, Biochimica et biophysica acta.
[29] U. Lindahl,et al. Domain Structure of Heparan Sulfates from Bovine Organs* , 1996, The Journal of Biological Chemistry.
[30] S. O. Kolset,et al. Blood platelets stimulate the expression of chondroitin sulfate proteoglycan in human monocytes. , 1992, Blood.
[31] M. Farquhar,et al. Increased permeability of the glomerular basement membrane to ferritin after removal of glycosaminoglycans (heparan sulfate) by enzyme digestion , 1980, The Journal of cell biology.
[32] J. Shively,et al. Formation of anhydrosugars in the chemical depolymerization of heparin. , 1976, Biochemistry.
[33] E. Jaffe,et al. Culture of human endothelial cells derived from umbilical veins. Identification by morphologic and immunologic criteria. , 1973, The Journal of clinical investigation.
[34] T BITTER,et al. A modified uronic acid carbazole reaction. , 1962, Analytical biochemistry.