Absence of tumor necrosis factor rescues RelA-deficient mice from embryonic lethality.
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L. Old | Y. Obata | M. Marino | T. Sakakura | T. Doi | T. Yoshida | T. Takahashi | Lloyd J. Old | Y. Obata | Toshitada Takahashi | Toshimichi Yoshida | Teruyo Sakakura | Takahiro S. Doi | Michael W. Marino
[1] L. Kirshenbaum,et al. Bcl-2 Activates the Transcription Factor NFκB through the Degradation of the Cytoplasmic Inhibitor IκBα* , 1998, The Journal of Biological Chemistry.
[2] C. Y. Wang,et al. NF-kappaB antiapoptosis: induction of TRAF1 and TRAF2 and c-IAP1 and c-IAP2 to suppress caspase-8 activation. , 1998, Science.
[3] V. Dixit,et al. Death receptors: signaling and modulation. , 1998, Science.
[4] Z. Ao,et al. IEX-1L, an Apoptosis Inhibitor Involved in NF-κB-Mediated Cell Survival , 1998 .
[5] T. Cotter,et al. The production of a reactive oxygen intermediate during the induction of apoptosis by cytotoxic insult. , 1998, Experimental cell research.
[6] J. Bell,et al. Lymphocyte inhibitor of TRAIL (TNF-related apoptosis-inducing ligand): a new receptor protecting lymphocytes from the death ligand TRAIL. , 1998, Journal of immunology.
[7] John Calvin Reed,et al. The c‐IAP‐1 and c‐IAP‐2 proteins are direct inhibitors of specific caspases , 1997, The EMBO journal.
[8] C. Cordon-Cardo,et al. Lipopolysaccharide Induces Disseminated Endothelial Apoptosis Requiring Ceramide Generation , 1997, The Journal of experimental medicine.
[9] M. Nussenzweig,et al. TRAF2 is essential for JNK but not NF-kappaB activation and regulates lymphocyte proliferation and survival. , 1997, Immunity.
[10] D. Goeddel,et al. Early lethality, functional NF-kappaB activation, and increased sensitivity to TNF-induced cell death in TRAF2-deficient mice. , 1997, Immunity.
[11] M. Malim,et al. Suppression of tumor necrosis factor-induced cell death by inhibitor of apoptosis c-IAP2 is under NF-kappaB control. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[12] R. Gentz,et al. An antagonist decoy receptor and a death domain-containing receptor for TRAIL. , 1997, Science.
[13] W I Wood,et al. Control of TRAIL-induced apoptosis by a family of signaling and decoy receptors. , 1997, Science.
[14] L. Old,et al. Characterization of tumor necrosis factor-deficient mice. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[15] D. Baltimore,et al. Failure of lymphopoiesis after adoptive transfer of NF-kappaB-deficient fetal liver cells. , 1997, Immunity.
[16] Y. Obata,et al. NF-κB RelA-deficient Lymphocytes: Normal Development of T Cells and B Cells, Impaired Production of IgA and IgG1 and Reduced Proliferative Responses , 1997, The Journal of experimental medicine.
[17] P. Baeuerle,et al. NF-kappa B as a frequent target for immunosuppressive and anti-inflammatory molecules. , 1997, Advances in immunology.
[18] Y. Hannun. Functions of Ceramide in Coordinating Cellular Responses to Stress , 1996, Science.
[19] K. Bauer,et al. Apo-3, a new member of the tumor necrosis factor receptor family, contains a death domain and activates apoptosis and NF-κB , 1996, Current Biology.
[20] A. Chinnaiyan,et al. Signal Transduction by DR3, a Death Domain-Containing Receptor Related to TNFR-1 and CD95 , 1996, Science.
[21] Marty W. Mayo,et al. TNF- and Cancer Therapy-Induced Apoptosis: Potentiation by Inhibition of NF-κB , 1996, Science.
[22] David Baltimore,et al. An Essential Role for NF-κB in Preventing TNF-α-Induced Cell Death , 1996, Science.
[23] Seamus J. Martin,et al. Suppression of TNF-α-Induced Apoptosis by NF-κB , 1996, Science.
[24] Michael Karin,et al. Dissection of TNF Receptor 1 Effector Functions: JNK Activation Is Not Linked to Apoptosis While NF-κB Activation Prevents Cell Death , 1996, Cell.
[25] G. Kollias,et al. Immune and inflammatory responses in TNF alpha-deficient mice: a critical requirement for TNF alpha in the formation of primary B cell follicles, follicular dendritic cell networks and germinal centers, and in the maturation of the humoral immune response , 1996, The Journal of experimental medicine.
[26] D. Goeddel,et al. The tumor necrosis factor-inducible zinc finger protein A20 interacts with TRAF1/TRAF2 and inhibits NF-kappaB activation. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[27] A. Strasser,et al. Mice lacking the c-rel proto-oncogene exhibit defects in lymphocyte proliferation, humoral immunity, and interleukin-2 expression. , 1995, Genes & development.
[28] David Baltimore,et al. Embryonic lethality and liver degeneration in mice lacking the RelA component of NF-κB , 1995, Nature.
[29] D. Goeddel,et al. Monoclonal antibodies specific for murine p55 and p75 tumor necrosis factor receptors: identification of a novel in vivo role for p75 , 1995, The Journal of experimental medicine.
[30] Kohchi Chie,et al. Constitutive expression of tnf-α and -β genes in mouse embryo: roles of cytokines as regulator and effector on development , 1994 .
[31] R. Zinkernagel,et al. Mice lacking the tumour necrosis factor receptor 1 are resistant to IMF-mediated toxicity but highly susceptible to infection by Listeria monocytogenes , 1993, Nature.
[32] T. Mak,et al. Mice deficient for the 55 kd tumor necrosis factor receptor are resistant to endotoxic shock, yet succumb to L. monocytogenes infection , 1993, Cell.
[33] V. Dixit,et al. The A20 zinc finger protein protects cells from tumor necrosis factor cytotoxicity. , 1992, The Journal of biological chemistry.
[34] P. Dobrzanski,et al. RelB, a new Rel family transcription activator that can interact with p50-NF-kappa B , 1992, Molecular and cellular biology.
[35] J. Safrit,et al. Synergistic effect of tumor necrosis factor-alpha- and diphtheria toxin-mediated cytotoxicity in sensitive and resistant human ovarian tumor cell lines. , 1991, Journal of immunology.
[36] G. Nolan,et al. DNA binding and IκB inhibition of the cloned p65 subunit of NF-κB, a rel-related polypeptide , 1991, Cell.
[37] T. Hunter,et al. The mouse c-rel protein has an N-terminal regulatory domain and a C-terminal transcriptional transactivation domain , 1990, Molecular and cellular biology.
[38] D. Niesel,et al. Bacteria-infected fibroblasts have enhanced susceptibility to the cytotoxic action of tumor necrosis factor. , 1990, Journal of immunology.
[39] R. Lunn,et al. Correlation between the anticellular and DNA fragmenting activities of tumor necrosis factor. , 1988, Cancer research.
[40] D. Stites,et al. In vitro anti-human immunodeficiency virus activities of tumor necrosis factor-alpha and interferon-gamma. , 1988, Journal of immunology.
[41] F. Balkwill,et al. DNA fragmentation and cytotoxicity caused by tumor necrosis factor is enhanced by interferon‐γ , 1987, European journal of immunology.
[42] Koff Wc,et al. Human tumor necrosis factor-alpha kills herpesvirus-infected but not normal cells. , 1986 .
[43] L. Old,et al. Human tumor necrosis factor produced by human B-cell lines: synergistic cytotoxic interaction with human interferon. , 1983, Proceedings of the National Academy of Sciences of the United States of America.
[44] W. Rutter,et al. Isolation of biologically active ribonucleic acid from sources enriched in ribonuclease. , 1979, Biochemistry.
[45] W. Reutter,et al. Galactosamine-induced sensitization to the lethal effects of endotoxin. , 1979, Proceedings of the National Academy of Sciences of the United States of America.
[46] N. Blin,et al. A general method for isolation of high molecular weight DNA from eukaryotes. , 1976, Nucleic acids research.
[47] R L Kassel,et al. An endotoxin-induced serum factor that causes necrosis of tumors. , 1975, Proceedings of the National Academy of Sciences of the United States of America.