Oncogenic cooperation between H-Twist and N-Myc overrides failsafe programs in cancer cells.
暂无分享,去创建一个
A. Puisieux | V. Combaret | P. Leissner | A. Krause | S. Valsesia-Wittmann | Anne-Catherine Jallas | Élisabeth Garin | S. Dupasquier | M. Magdeleine
[1] S. Ramaswamy,et al. Twist, a Master Regulator of Morphogenesis, Plays an Essential Role in Tumor Metastasis , 2004, Cell.
[2] M. Hogarty. The requirement for evasion of programmed cell death in neuroblastomas with MYCN amplification. , 2003, Cancer letters.
[3] G. Brodeur. Neuroblastoma: biological insights into a clinical enigma , 2003, Nature Reviews Cancer.
[4] E. Olson,et al. Twist Regulates Cytokine Gene Expression through a Negative Feedback Loop that Represses NF-κB Activity , 2003, Cell.
[5] Birgit Luber,et al. Differential expression of the epithelial-mesenchymal transition regulators snail, SIP1, and twist in gastric cancer. , 2002, The American journal of pathology.
[6] R. Behringer,et al. Twist plays an essential role in FGF and SHH signal transduction during mouse limb development. , 2002, Developmental biology.
[7] V. El Ghouzzi,et al. Twist haploinsufficiency in Saethre-Chotzen syndrome induces calvarial osteoblast apoptosis due to increased TNFalpha expression and caspase-2 activation. , 2002, Human molecular genetics.
[8] P. Catalano,et al. c-myc/p53 interaction determines sensitivity of human colon carcinoma cells to 5-fluorouracil in vitro and in vivo. , 2001, Cancer research.
[9] L. Kedes,et al. Twist is a potential oncogene that inhibits apoptosis. , 1999, Genes & development.
[10] M. Schwab,et al. MycN sensitizes neuroblastoma cells for drug-induced apoptosis , 1999, Oncogene.
[11] J L Cleveland,et al. Myc signaling via the ARF tumor suppressor regulates p53-dependent apoptosis and immortalization. , 1998, Genes & development.
[12] L. Kedes,et al. The basic domain of myogenic basic helix-loop-helix (bHLH) proteins is the novel target for direct inhibition by another bHLH protein, Twist , 1997, Molecular and cellular biology.
[13] M. Schwab,et al. Conditional expression of N-myc in human neuroblastoma cells increases expression of alpha-prothymosin and ornithine decarboxylase and accelerates progression into S-phase early after mitogenic stimulation of quiescent cells. , 1996, Oncogene.
[14] L. Donehower,et al. The MMTV/c-myc transgene and p53 null alleles collaborate to induce T-cell lymphomas, but not mammary carcinomas in transgenic mice. , 1995, Oncogene.
[15] R. Behringer,et al. twist is required in head mesenchyme for cranial neural tube morphogenesis. , 1995, Genes & development.
[16] E. Füchtbauer,et al. M-twist is an inhibitor of muscle differentiation. , 1994, Developmental biology.
[17] H. Hermeking,et al. Mediation of c-Myc-induced apoptosis by p53. , 1994, Science.
[18] J. Pelletier,et al. Absence of p53 gene mutations in primary neuroblastomas. , 1993, Cancer research.
[19] Gerard I. Evan,et al. Induction of apoptosis in fibroblasts by c-myc protein , 1992, Cell.
[20] A. Strasser,et al. Novel primitive lymphoid tumours induced in transgenic mice by cooperation between myc and bcl-2 , 1990, Nature.
[21] M. Leptin,et al. Cell shape changes during gastrulation in Drosophila. , 1990, Development.
[22] H. Varmus,et al. Human N-myc gene contributes to neoplastic transformation of mammalian cells in culture , 1985, Nature.
[23] H. Varmus,et al. Amplification of N-myc in untreated human neuroblastomas correlates with advanced disease stage. , 1984, Science.
[24] E. Green,et al. Mutations in TWIST, a basic helix–loop–helix transcription factor, in Saethre-Chotzen syndrome , 1997, Nature Genetics.
[25] P. Bénit,et al. Mutations of the TWIST gene in the Saethre-Chotzene syndrome , 1997, Nature Genetics.
[26] E. Olson,et al. bHLH factors in muscle development: dead lines and commitments, what to leave in and what to leave out. , 1994, Genes & development.