Decreased expression of microRNA-125a-3p upregulates interleukin-23 receptor in patients with Hashimoto’s thyroiditis
暂无分享,去创建一个
Yue Zhang | Huaxi Xu | Jie Tian | Chaoming Mao | Jun Yang | Huiyong Peng | Xinyi Tang | Shengjun Wang | Jie Ma | Yingzhao Liu | Ke Rui | Li-yuan Lu
[1] Huaxi Xu,et al. MiR-346 regulates CD4+CXCR5+ T cells in the pathogenesis of Graves’ disease , 2015, Endocrine.
[2] Huaxi Xu,et al. Th17/Treg Cells Imbalance and GITRL Profile in Patients with Hashimoto’s Thyroiditis , 2014, International journal of molecular sciences.
[3] B. Rapoport,et al. Graves' hyperthyroidism is antibody-mediated but is predominantly a Th1-type cytokine disease. , 2014, The Journal of clinical endocrinology and metabolism.
[4] Jun Wang,et al. Decreased expression of microRNA-21 correlates with the imbalance of Th17 and Treg cells in patients with rheumatoid arthritis , 2014, Journal of cellular and molecular medicine.
[5] Jie Tian,et al. Roles of miRNAs in regulating the differentiation and maturation of myeloid-derived suppressor cells. , 2014, Medical hypotheses.
[6] M. Etemadifar,et al. miR-326 and miR-26a, two potential markers for diagnosis of relapse and remission phases in patient with relapsing-remitting multiple sclerosis. , 2014, Gene.
[7] F. Trimarchi,et al. Serum interleukin-23 (IL-23) is increased in Hashimoto's thyroiditis. , 2014, Endocrine journal.
[8] P. Wiland,et al. Peripheral blood Th17/Treg imbalance in patients with low-active systemic lupus erythematosus. , 2014, Postepy higieny i medycyny doswiadczalnej.
[9] F. Sallusto,et al. Human Th17 subsets , 2012, European journal of immunology.
[10] Wu-xian Li,et al. Circulating microRNAs as candidate biomarkers in patients with systemic lupus erythematosus. , 2012, Translational research : the journal of laboratory and clinical medicine.
[11] Huaxi Xu,et al. Increased frequency of follicular helper T cells in patients with autoimmune thyroid disease. , 2012, The Journal of clinical endocrinology and metabolism.
[12] S. Brant,et al. IL-23 Receptor Regulation by Let-7f in Human CD4+ Memory T Cells , 2011, The Journal of Immunology.
[13] S. Ahmed,et al. MicroRNA, a new paradigm for understanding immunoregulation, inflammation, and autoimmune diseases. , 2011, Translational research : the journal of laboratory and clinical medicine.
[14] J. Jaume,et al. Hashimoto thyroiditis is more frequent than expected when diagnosed by cytology which uncovers a pre-clinical state , 2010, Thyroid research.
[15] H. Yang,et al. Differentiation Imbalance of Th1/ Th17 in Peripheral Blood Mononuclear Cells Might Contribute to Pathogenesis of Hashimoto’s Thyroiditis , 2010, Scandinavian journal of immunology.
[16] F. Sánchez‐Madrid,et al. Increased circulating pro-inflammatory cytokines and Th17 lymphocytes in Hashimoto's thyroiditis. , 2010, The Journal of clinical endocrinology and metabolism.
[17] Y. Iwatani,et al. Increases of the Th1/Th2 cell ratio in severe Hashimoto's disease and in the proportion of Th17 cells in intractable Graves' disease. , 2009, Thyroid : official journal of the American Thyroid Association.
[18] T. Mcclanahan,et al. The interleukin 23 receptor is essential for the terminal differentiation of interleukin 17–producing effector T helper cells in vivo , 2009, Nature Immunology.
[19] R. D. Hatton,et al. IL-17 family cytokines and the expanding diversity of effector T cell lineages. , 2007, Annual review of immunology.
[20] R. J. Hocking,et al. TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells. , 2006, Immunity.
[21] T. Mcclanahan,et al. A Receptor for the Heterodimeric Cytokine IL-23 Is Composed of IL-12Rβ1 and a Novel Cytokine Receptor Subunit, IL-23R1 , 2002, The Journal of Immunology.