Low-level laser facilitates alternatively activated macrophage/microglia polarization and promotes functional recovery after crush spinal cord injury in rats
暂无分享,去创建一个
Xueyu Hu | Zhuowen Liang | Shuang Wang | Kun Li | Chen Dai | Xuefeng Shen | Yuzhe Gong | Zhe Wang | Ji Wei Song
[1] Zhong Chen,et al. N-acetylcysteine treatment following spinal cord trauma reduces neural tissue damage and improves locomotor function in mice , 2015, Molecular medicine reports.
[2] M. Gaestel,et al. TNF and Increased Intracellular Iron Alter Macrophage Polarization to a Detrimental M1 Phenotype in the Injured Spinal Cord , 2014, Neuron.
[3] Xi Chen,et al. Gift-Giving and Network Structure in Rural China: Utilizing Long-Term Spontaneous Gift Records , 2014, PloS one.
[4] V. Yong,et al. Immune modulatory therapies for spinal cord injury – Past, present and future , 2014, Experimental Neurology.
[5] J. Gensel,et al. IL-4 Signaling Drives a Unique Arginase+/IL-1β+ Microglia Phenotype and Recruits Macrophages to the Inflammatory CNS: Consequences of Age-Related Deficits in IL-4Rα after Traumatic Spinal Cord Injury , 2014, The Journal of Neuroscience.
[6] L. Shea,et al. Lentivirus delivery of IL‐10 to promote and sustain macrophage polarization towards an anti‐inflammatory phenotype , 2014, Biotechnology and Bioengineering.
[7] Rafael J. Yáñez-Muñoz,et al. Large-Scale Chondroitin Sulfate Proteoglycan Digestion with Chondroitinase Gene Therapy Leads to Reduced Pathology and Modulates Macrophage Phenotype following Spinal Cord Contusion Injury , 2014, The Journal of Neuroscience.
[8] J. Bonventre,et al. Cytosolic Phospholipase A2 Protein as a Novel Therapeutic Target for Spinal Cord Injury , 2014, Annals of neurology.
[9] Deborah S. Barkauskas,et al. High-resolution intravital imaging reveals that blood-derived macrophages but not resident microglia facilitate secondary axonal dieback in traumatic spinal cord injury , 2014, Experimental Neurology.
[10] Mao‐Hsiung Huang,et al. Effect of Low Level Laser Therapy on Chronic Compression of the Dorsal Root Ganglion , 2014, PloS one.
[11] 王晶,et al. 激光照射对脊髓损伤大鼠TNF-α、IL-6和IL-10表达的影响 , 2014 .
[12] W. Young,et al. Managing Inflammation after Spinal Cord Injury through Manipulation of Macrophage Function , 2013, Neural plasticity.
[13] D. Hwang,et al. Contribution of Macrophages to Enhanced Regenerative Capacity of Dorsal Root Ganglia Sensory Neurons by Conditioning Injury , 2013, The Journal of Neuroscience.
[14] Minoru Obara,et al. Low-level laser therapy for spinal cord injury in rats: effects of polarization , 2013, Journal of biomedical optics.
[15] J. Henley,et al. Cellular Neuroinflammation in a Lateral Forceps Compression Model of Spinal Cord Injury , 2013, Anatomical record.
[16] A. Villaverde,et al. Effects of low-level laser therapy (LLLT) on bone repair in rats: optical densitometry analysis , 2013, Lasers in Medical Science.
[17] Daniela Vecchio,et al. Transcranial low level laser (light) therapy for traumatic brain injury , 2012, Journal of biophotonics.
[18] H. Baba,et al. Transplantation of mesenchymal stem cells promotes an alternative pathway of macrophage activation and functional recovery after spinal cord injury. , 2012, Journal of neurotrauma.
[19] N. Rosenthal,et al. An Abundant Tissue Macrophage Population in the Adult Murine Heart with a Distinct Alternatively-Activated Macrophage Profile , 2012, PloS one.
[20] T. Atsumi,et al. Interleukin-1 participates in the classical and alternative activation of microglia/macrophages after spinal cord injury , 2012, Journal of Neuroinflammation.
[21] H. Baba,et al. Blockade of interleukin-6 signaling inhibits the classic pathway and promotes an alternative pathway of macrophage activation after spinal cord injury in mice , 2012, Journal of Neuroinflammation.
[22] Tianhong Dai,et al. The Nuts and Bolts of Low-level Laser (Light) Therapy , 2011, Annals of Biomedical Engineering.
[23] T. Wynn,et al. Protective and pathogenic functions of macrophage subsets , 2011, Nature Reviews Immunology.
[24] S. David,et al. Repertoire of microglial and macrophage responses after spinal cord injury , 2011, Nature Reviews Neuroscience.
[25] S. Cuzzocrea,et al. Anti-TNF therapy in the injured spinal cord. , 2011, Trends in pharmacological sciences.
[26] K. Horn,et al. Multipotent Adult Progenitor Cells Prevent Macrophage-Mediated Axonal Dieback and Promote Regrowth after Spinal Cord Injury , 2011, The Journal of Neuroscience.
[27] Seung U. Kim,et al. Human neural stem cells genetically modified to overexpress brain‐derived neurotrophic factor promote functional recovery and neuroprotection in a mouse stroke model , 2010, Journal of neuroscience research.
[28] U. Namgung,et al. Endogenous expression of interleukin‐4 regulates macrophage activation and confines cavity formation after traumatic spinal cord injury , 2010, Journal of neuroscience research.
[29] S. Gordon,et al. Alternative activation of macrophages: mechanism and functions. , 2010, Immunity.
[30] Jessica K. Alexander,et al. Identification of Two Distinct Macrophage Subsets with Divergent Effects Causing either Neurotoxicity or Regeneration in the Injured Mouse Spinal Cord , 2009, The Journal of Neuroscience.
[31] K. Horn,et al. Overcoming Macrophage-Mediated Axonal Dieback Following CNS Injury , 2009, The Journal of Neuroscience.
[32] J. Gensel,et al. Macrophages Promote Axon Regeneration with Concurrent Neurotoxicity , 2009, The Journal of Neuroscience.
[33] Michael R. Hamblin,et al. Low-Level Laser Therapy Activates NF-kB via Generation of Reactive Oxygen Species in Mouse Embryonic Fibroblasts , 2009, BiOS.
[34] Jackson Streeter,et al. 810 nm Wavelength light: An effective therapy for transected or contused rat spinal cord , 2009, Lasers in surgery and medicine.
[35] K. Horn,et al. Another Barrier to Regeneration in the CNS: Activated Macrophages Induce Extensive Retraction of Dystrophic Axons through Direct Physical Interactions , 2008, The Journal of Neuroscience.
[36] V. Yong,et al. Dynamics of the inflammatory response after murine spinal cord injury revealed by flow cytometry , 2008, Journal of neuroscience research.
[37] W. Tetzlaff,et al. A graded forceps crush spinal cord injury model in mice. , 2008, Journal of neurotrauma.
[38] David A Ramsay,et al. The cellular inflammatory response in human spinal cords after injury. , 2006, Brain : a journal of neurology.
[39] M. Pacheco,et al. Low-level laser therapy induces dose-dependent reduction of TNFalpha levels in acute inflammation. , 2006, Photomedicine and laser surgery.
[40] S. Gordon,et al. Monocyte and macrophage heterogeneity , 2005, Nature Reviews Immunology.
[41] R. Waynant,et al. Light promotes regeneration and functional recovery and alters the immune response after spinal cord injury , 2005, Lasers in surgery and medicine.
[42] Silvano Sozzani,et al. The chemokine system in diverse forms of macrophage activation and polarization. , 2004, Trends in immunology.
[43] J. Steeves,et al. Minocycline Treatment Reduces Delayed Oligodendrocyte Death, Attenuates Axonal Dieback, and Improves Functional Outcome after Spinal Cord Injury , 2004, The Journal of Neuroscience.
[44] Z. Werb,et al. Blood‐spinal cord barrier after spinal cord injury: Relation to revascularization and wound healing , 2003, Journal of neuroscience research.
[45] J. Wrathall,et al. Temporal–Spatial Pattern of Acute Neuronal and Glial Loss after Spinal Cord Contusion , 2001, Experimental Neurology.
[46] Phillip G. Popovich,et al. Depletion of Hematogenous Macrophages Promotes Partial Hindlimb Recovery and Neuroanatomical Repair after Experimental Spinal Cord Injury , 1999, Experimental Neurology.
[47] Sonia L. Carlson,et al. Acute Inflammatory Response in Spinal Cord Following Impact Injury , 1998, Experimental Neurology.
[48] M. Jacquin,et al. Neuronal and Glial Apoptosis after Traumatic Spinal Cord Injury , 1997, The Journal of Neuroscience.
[49] B. Stokes,et al. A Quantitative Spatial Analysis of the Blood–Spinal Cord Barrier I. Permeability Changes after Experimental Spinal Contusion Injury , 1996, Experimental Neurology.
[50] J. A. Gruner,et al. Histological and functional evaluation of experimental spinal cord injury: evidence of a stepwise response to graded compression , 1996, Brain Research.
[51] M. Schwab,et al. Degeneration and regeneration of axons in the lesioned spinal cord. , 1996, Physiological reviews.
[52] D. Basso,et al. A sensitive and reliable locomotor rating scale for open field testing in rats. , 1995, Journal of neurotrauma.
[53] Dan Zhu,et al. M2 Macrophage Transplantation Ameliorates Cognitive Dysfunction in Amyloid-β-Treated Rats Through Regulation of Microglial Polarization. , 2016, Journal of Alzheimer's disease : JAD.
[54] Sookja K. Chung,et al. Aldose Reductase Regulates Microglia/Macrophages Polarization Through the cAMP Response Element-Binding Protein After Spinal Cord Injury in Mice , 2014, Molecular Neurobiology.
[55] C. Oyinbo. Secondary injury mechanisms in traumatic spinal cord injury: a nugget of this multiply cascade. , 2011, Acta neurobiologiae experimentalis.
[56] 王. W. Jian,et al. Positive Effect of Low Power Laser Irradiation on Neuron Regeneration after Acute Spinal Cord Injury , 2009 .