Human renal tubular cells contain CD24/CD133 progenitor cell populations: Implications for tubular regeneration after toxicant induced damage using cadmium as a model

[1]  T. Riemer,et al.  Short-term overfeeding of zebrafish with normal or high-fat diet as a model for the development of metabolically healthy versus unhealthy obesity , 2017, BMC Physiology.

[2]  M. Buser,et al.  Urinary and blood cadmium and lead and kidney function: NHANES 2007-2012. , 2016, International journal of hygiene and environmental health.

[3]  Paul Jennings,et al.  Development of an in vitro renal epithelial disease state model for xenobiotic toxicity testing. , 2015, Toxicology in vitro : an international journal published in association with BIBRA.

[4]  S. Garrett,et al.  Cadherin Expression, Vectorial Active Transport, and Metallothionein Isoform 3 Mediated EMT/MET Responses in Cultured Primary and Immortalized Human Proximal Tubule Cells , 2015, PloS one.

[5]  M. Schwartz,et al.  Heptinstall's Pathology of the Kidney , 2014 .

[6]  G. Remuzzi,et al.  CD133+ renal stem cells always co-express CD24 in adult human kidney tissue. , 2014, Stem cell research.

[7]  Paul Jennings,et al.  Delineation of the Key Aspects in the Regulation of Epithelial Monolayer Formation , 2013, Molecular and Cellular Biology.

[8]  P. Boor,et al.  Proximal tubular cells contain a phenotypically distinct, scattered cell population involved in tubular regeneration , 2013, The Journal of pathology.

[9]  F. Schena,et al.  Human renal stem/progenitor cells repair tubular epithelial cell injury through TLR2-driven inhibin-A and microvesicle-shuttled decorin. , 2013, Kidney international.

[10]  G. Remuzzi,et al.  Renal progenitors: an evolutionary conserved strategy for kidney regeneration , 2013, Nature Reviews Nephrology.

[11]  J. Sommar,et al.  End-stage renal disease and low level exposure to lead, cadmium and mercury; a population-based, prospective nested case-referent study in Sweden , 2013, Environmental Health.

[12]  M. Satoh,et al.  Cadmium renal toxicity via apoptotic pathways. , 2012, Biological & pharmaceutical bulletin.

[13]  Joshua R Edwards,et al.  Mechanisms of Cadmium-Induced Proximal Tubule Injury: New Insights with Implications for Biomonitoring and Therapeutic Interventions , 2012, Journal of Pharmacology and Experimental Therapeutics.

[14]  Pam Tucker,et al.  Toxicological Profile for Cadmium , 2012 .

[15]  M. Carini,et al.  Characterization of Renal Progenitors Committed Toward Tubular Lineage and Their Regenerative Potential in Renal Tubular Injury , 2012, Stem cells.

[16]  Xiaoyan Li,et al.  V-ATPase promotes transforming growth factor-β-induced epithelial-mesenchymal transition of rat proximal tubular epithelial cells. , 2012, American journal of physiology. Renal physiology.

[17]  B. Molitoris.  Acute Kidney Injury , 2012 .

[18]  K. Jirström,et al.  Isolation and characterization of progenitor-like cells from human renal proximal tubules. , 2011, The American journal of pathology.

[19]  J. Dubernard,et al.  [The kidney]. , 2011, Bulletin de l'Academie nationale de medecine.

[20]  Y. Liu,et al.  Multiple roles of cadmium in cell death and survival. , 2010, Chemico-biological interactions.

[21]  S. Garrett,et al.  Keratin 6 expression correlates to areas of squamous differentiation in multiple independent isolates of As+3‐induced bladder cancer , 2010, Journal of applied toxicology : JAT.

[22]  G. Gambaro,et al.  Low level exposure to cadmium increases the risk of chronic kidney disease: analysis of the NHANES 1999-2006 , 2010, BMC public health.

[23]  A. Kribben,et al.  N-cadherin is depleted from proximal tubules in experimental and human acute kidney injury , 2010, Histochemistry and Cell Biology.

[24]  M. Rosner,et al.  Acute kidney injury. , 2009, Current drug targets.

[25]  E. Guallar,et al.  Blood cadmium and lead and chronic kidney disease in US adults: a joint analysis. , 2009, American journal of epidemiology.

[26]  Jie Liu,et al.  Expression of kidney injury molecule-1 (Kim-1) in relation to necrosis and apoptosis during the early stages of Cd-induced proximal tubule injury. , 2009, Toxicology and applied pharmacology.

[27]  M. Carini,et al.  Regeneration of glomerular podocytes by human renal progenitors. , 2009, Journal of the American Society of Nephrology : JASN.

[28]  Paul Jennings,et al.  hTERT alone immortalizes epithelial cells of renal proximal tubules without changing their functional characteristics. , 2008, American journal of physiology. Renal physiology.

[29]  S. Garrett,et al.  Cadmium, vectorial active transport, and MT-3-dependent regulation of cadherin expression in human proximal tubular cells. , 2008, Toxicological sciences : an official journal of the Society of Toxicology.

[30]  M. Rotondi,et al.  Regenerative potential of embryonic renal multipotent progenitors in acute renal failure. , 2007, Journal of the American Society of Nephrology : JASN.

[31]  D. Sens,et al.  Variation in the electrical properties of cultured human proximal tubule cells , 1993, In Vitro Cellular & Developmental Biology - Animal.

[32]  M. Carini,et al.  Isolation and characterization of multipotent progenitor cells from the Bowman's capsule of adult human kidneys. , 2006, Journal of the American Society of Nephrology : JASN.

[33]  S. Garrett,et al.  The unique N-terminal sequence of metallothionein-3 is required to regulate the choice between apoptotic or necrotic cell death of human proximal tubule cells exposed to Cd+2. , 2006, Toxicological sciences : an official journal of the Society of Toxicology.

[34]  S. Skerfving,et al.  Tubular and Glomerular Kidney Effects in Swedish Women with Low Environmental Cadmium Exposure , 2005, Environmental health perspectives.

[35]  S. Garrett,et al.  Expression of metallothionein isoform 3 (MT-3) determines the choice between apoptotic or necrotic cell death in Cd+2-exposed human proximal tubule cells. , 2004, Toxicological sciences : an official journal of the Society of Toxicology.

[36]  D. Appelt,et al.  Differential expression of E-cadherin, N-cadherin and beta-catenin in proximal and distal segments of the rat nephron. , 2004, BMC Physiology.

[37]  E. Prigge.  Early signs of oral and inhalative cadmium uptake in rats , 1978, Archives of Toxicology.

[38]  S. Garrett,et al.  Metallothionein isoform 3 and proximal tubule vectorial active transport. , 2002, Kidney international.

[39]  S. Garrett,et al.  Exposure of human proximal tubule cells to cd2+, zn2+, and Cu2+ induces metallothionein protein accumulation but not metallothionein isoform 2 mRNA. , 1998, Environmental health perspectives.

[40]  V. Jha,et al.  Acute renal cortical necrosis--a study of 113 patients. , 1994, Renal failure.

[41]  R. Zager,et al.  HK-2: an immortalized proximal tubule epithelial cell line from normal adult human kidney. , 1994, Kidney international.

[42]  S. Dauwe,et al.  Stage- and segment-specific expression of cell-adhesion molecules N-CAM, A-CAM, and L-CAM in the kidney. , 1993, Kidney international.

[43]  A. Evan,et al.  Proximal tubule characteristics of cultured human renal cortex epithelium. , 1989, The Journal of laboratory and clinical medicine.

[44]  J. G. Blackburn,et al.  Electrophysiology and ultrastructure of cultured human proximal tubule cells. , 1988, Kidney international.

[45]  C. Cha.  A study on the effect of garlic to the heavy metal poisoning of rat. , 1987, Journal of Korean medical science.

[46]  J. Foreman,et al.  Characteristics of cultured human renal cortical epithelia. , 1986, Biochemical medicine and metabolic biology.

[47]  R. Anderson,et al.  Defined human renal tubular epithelia in culture: growth, characterization, and hormonal response. , 1985, The American journal of physiology.

[48]  B. Trump,et al.  Isolation, culture and characterization of human renal tubular cells. , 1985, The Journal of urology.

[49]  S. Spicer,et al.  Tissue culture of human kidney epithelial cells of proximal tubule origin. , 1984, Kidney international.

[50]  M. Taub,et al.  Characterization of primary rabbit kidney cultures that express proximal tubule functions in a hormonally defined medium , 1982, The Journal of cell biology.

[51]  M. Saier,et al.  Continuous growth of proximal tubular kidney epithelial cells in hormone-supplemented serum-free medium , 1982, The Journal of cell biology.

[52]  M. Taub,et al.  Growth of functional primary cultures of kidney epithelial cells in defined medium , 1980, Journal of cellular physiology.

[53]  M H Saier,et al.  Growth of Madin-Darby canine kidney epithelial cell (MDCK) line in hormone-supplemented, serum-free medium. , 1979, Proceedings of the National Academy of Sciences of the United States of America.

[54]  M. Taub,et al.  Growth of kidney epithelial cells in hormone-supplemented, serum-free medium. , 1979, Journal of supramolecular structure.

[55]  F. N. Kotsonis,et al.  The relationship of metallothionein to the toxicity of cadmium after prolonged oral administration to rats. , 1978, Toxicology and applied pharmacology.

[56]  O. Yoshida,et al.  Disturbances in kidney functions and calcium and phosphate metabolism in cadmium-poisoned rats. , 1978, Nephron.

[57]  Y. Itokawa,et al.  Renal and skeletal lesions in experimental cadmium poisoning: histological and biochemical approaches. , 1974, Archives of environmental health.

[58]  D. Beton,et al.  Acute Cadmium Fume Poisoning: Five Cases with one Death from Renal Necrosis , 1966, British journal of industrial medicine.