Repeated Negative DC Deflections in Rat Cortex following Middle Cerebral Artery Occlusion are Abolished by MK-801: Effect on Volume of Ischemic Injury

[1]  K. Hossmann Disturbances of cerebral protein synthesis and ischemic cell death. , 1993, Progress in brain research.

[2]  G. Mies,et al.  Ischemic Thresholds of Cerebral Protein Synthesis and Energy State following Middle Cerebral Artery Occlusion in Rat , 1991, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[3]  H. Weiss,et al.  Cerebral Blood Flow and Oxygen Consumption in Cortical Spreading Depression , 1991, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[4]  M. Reivich,et al.  Mechanism Underlying Protective Effect of MK-801 against NMDA-Induced Neuronal Injury in vivo , 1991, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[5]  嶋田 延光 Differences in ischemia-induced accumulation of amino acids in the cat cortex , 1991 .

[6]  U. Dirnagl,et al.  Pre- and post-treatment with MK-801 but not pretreatment alone reduces neocortical damage after focal cerebral ischemia in the rat , 1990, Brain Research.

[7]  M. Kocher Journal of Cerebral Blood Flow and Metabolism Metabolic and Hemodynamic Activation of Postischemic Rat Brain by Cortical Spreading Depression , 2022 .

[8]  B. Meldrum,et al.  Protection by NMDA Antagonists against Selective Cell Loss following Transient Ischaemia , 1990, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[9]  D. Corbett,et al.  MK-801 reduced cerebral ischemic injury by inducing hypothermia , 1990, Brain Research.

[10]  R. Auer,et al.  Regional Neuroprotective Effects of the NMDA Receptor Antagonist MK-801 (Dizocilpine) in Hypoglycemic Brain Damage , 1990, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[11]  W. Pulsinelli,et al.  Hypothermia but not the N-methyl-D-aspartate antagonist, MK-801, attenuates neuronal damage in gerbils subjected to transient global ischemia , 1990, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[12]  K. Marquis,et al.  Comparative electroencephalographic and behavioral effects of phencyclidine, (+)-SKF-10,047 and MK-801 in rats. , 1989, The Journal of pharmacology and experimental therapeutics.

[13]  J. Mcculloch,et al.  Effect of the NMDA Antagonist MK-801 on Local Cerebral Blood Flow in Focal Cerebral Ischaemia in the Rat , 1989, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[14]  G. Mies,et al.  Ischemic threshold of brain protein synthesis after unilateral carotid artery occlusion in gerbils. , 1989, Stroke.

[15]  I. Kass,et al.  The N-methyl-d-aspartate antagonists aminophosphonovaleric acid and MK-801 reduce anoxic damage to dentate granule and CA1 pyramidal cells in the rat hippocampal slice , 1989, Experimental Neurology.

[16]  J. Mcculloch,et al.  The glutamate antagonist MK‐801 reduces focal ischemic brain damage in the rat , 1988, Annals of neurology.

[17]  Roland Willems,et al.  Evidence for a role of the N-methyl-d-aspartate (NMDA) receptor in cortical spreading depression in the rat , 1988, Brain Research.

[18]  M. Nedergaard,et al.  Brain ion homeostasis in cerebral ischemia. , 1988, Neurochemical pathology.

[19]  A. Tamura,et al.  Behavioral changes after focal cerebral ischemia by left middle cerebral artery occlusion in rats , 1988, Brain Research.

[20]  A. Foster,et al.  MK-801 is neuroprotective in gerbils when administered during the post-ischaemic period , 1988, Neuroscience.

[21]  Maiken Nedergaard,et al.  Spreading depression is not associated with neuronal injury in the normal brain , 1988, Brain Research.

[22]  D. Graham,et al.  Protective Effect of the Glutamate Antagonist, MK-801 in Focal Cerebral Ischemia in the Cat , 1988, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[23]  J Astrup,et al.  Infarct Rim: Effect of Hyperglycemia on Direct Current Potential and [14C]2-Deoxyglucose Phosphorylation , 1986, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[24]  W. Paschen,et al.  Regional changes of blood flow, glucose, and ATP content determined on brain sections during a single passage of spreading depression in rat brain cortex , 1984, Experimental Neurology.

[25]  W D Heiss,et al.  Functional recovery of cortical neurons as related to degree and duration of ischemia , 1983, Annals of neurology.

[26]  C. Steer Barbiturate Therapy in the Management of Cerebral Ischaemia , 1982, Developmental medicine and child neurology.

[27]  B. Siesjö,et al.  Thresholds in cerebral ischemia - the ischemic penumbra. , 1981, Stroke.

[28]  L. Sokoloff,et al.  Cerebral glucose utilization: local changes during and after recovery from spreading cortical depression. , 1979, Science.

[29]  J. Křivánek On the mechanism of the inhibitory effect of potasium ions on amino acid incorporation into rat cerebral cortex proteins in vivo. , 1978, Physiologia Bohemoslovaca.

[30]  N M Branston,et al.  Cortical Evoked Potential and Extracellular K+ and H+ at Critical Levels of Brain Ischemia , 1977, Stroke.

[31]  J. Křivánek,et al.  Double labelling study of the spreading depression effect on the incorporation of labelled leucine into the proteins of the brain mitochondrial subfractions , 1973, Journal of neurochemistry.

[32]  A. van Harreveld,et al.  Mechanisms involved in spreading depression. , 1973, Journal of neurobiology.