Impact of nuclear YAP1 expression in residual cancer after neoadjuvant chemohormonal therapy with docetaxel for high-risk localized prostate cancer
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T. Habuchi | N. Tsuchiya | S. Satoh | M. Saito* | H. Nanjo | T. Inoue | S. Narita | K. Numakura | Y. Hiroshima | A. Koizumi | T. Nara | H. Sato | Huang Mingguo | S. Kanda | Takamitsu Inoue | Y. Matsuda | M. Saito*
[1] T. Hour,et al. CD44 Promotes Migration and Invasion of Docetaxel-Resistant Prostate Cancer Cells Likely via Induction of Hippo-Yap Signaling , 2019, Cells.
[2] Conor A. Bradley. AR-V7 — repress to impress , 2019, Nature Reviews Urology.
[3] T. Habuchi,et al. Radical Prostatectomy With and Without Neoadjuvant Chemohormonal Pretreatment for High‐Risk Localized Prostate Cancer: A Comparative Propensity Score Matched Analysis , 2019, Clinical genitourinary cancer.
[4] Paula Esther Moraga-Serrano. Global, Regional, and National Cancer Incidence, Mortality, Years of Life Lost, Years Lived With Disability, and Disability-Adjusted Life-Years for 29 Cancer Groups, 1990 to 2016:A Systematic Analysis for the Global Burden of Disease Study , 2018 .
[5] Xiao-li Zhang,et al. Functional significance of Hippo/YAP signaling for drug resistance in colorectal cancer , 2018, Molecular carcinogenesis.
[6] Mohammad Hossein Khosravi,et al. Global, Regional, and National Cancer Incidence, Mortality, Years of Life Lost, Years Lived With Disability, and Disability-Adjusted Life-Years for 29 Cancer Groups, 1990 to 2016 , 2018, JAMA oncology.
[7] Rongsheng Tong,et al. Multifaceted regulation and functions of YAP/TAZ in tumors , 2018, Oncology reports.
[8] Christian N. Cunningham,et al. Targeting the Hippo Pathway and Cancer through the TEAD Family of Transcription Factors , 2018, Cancers.
[9] G. Castoria,et al. Estrogens and Their Receptors in Prostate Cancer: Therapeutic Implications , 2018, Front. Oncol..
[10] D. Jäger,et al. Overexpression of nuclear AR-V7 protein in primary prostate cancer is an independent negative prognostic marker in men with high-risk disease receiving adjuvant therapy. , 2017, Urologic oncology.
[11] C. Collins,et al. Impact of Therapy on Genomics and Transcriptomics in High-Risk Prostate Cancer Treated with Neoadjuvant Docetaxel and Androgen Deprivation Therapy , 2017, Clinical Cancer Research.
[12] W. Hong,et al. ETS (E26 transformation-specific) up-regulation of the transcriptional co-activator TAZ promotes cell migration and metastasis in prostate cancer , 2017, The Journal of Biological Chemistry.
[13] Alan D. Lopez,et al. Global, Regional, and National Cancer Incidence, Mortality, Years of Life Lost, Years Lived With Disability, and Disability-Adjusted Life-years for 32 Cancer Groups, 1990 to 2015: A Systematic Analysis for the Global Burden of Disease Study , 2017, JAMA oncology.
[14] Min Hwan Kim,et al. Role of YAP/TAZ transcriptional regulators in resistance to anti-cancer therapies , 2016, Cellular and Molecular Life Sciences.
[15] N. Kyprianou,et al. Mechanisms of Therapeutic Resistance in Prostate Cancer , 2017, Current Oncology Reports.
[16] M. Nevalainen,et al. miR-375 induces docetaxel resistance in prostate cancer by targeting SEC23A and YAP1 , 2016, Molecular Cancer.
[17] R. Laing,et al. Addition of docetaxel, zoledronic acid, or both to first-line long-term hormone therapy in prostate cancer (STAMPEDE): survival results from an adaptive, multiarm, multistage, platform randomised controlled trial , 2016, The Lancet.
[18] Jun Yu,et al. International Journal of Molecular Sciences the Tead Family and Its Oncogenic Role in Promoting Tumorigenesis , 2022 .
[19] Bin Zhao,et al. Hippo Pathway in Organ Size Control, Tissue Homeostasis, and Cancer , 2015, Cell.
[20] R. Walther,et al. Differential expression of the multidrug resistance 1 (MDR1) protein in prostate cancer cells is independent from anticancer drug treatment and Y box binding protein 1 (YB-1) activity , 2015, World Journal of Urology.
[21] I. Garraway,et al. YAP1 and AR interactions contribute to the switch from androgen-dependent to castration-resistant growth in prostate cancer , 2015, Nature Communications.
[22] David F Jarrard,et al. Chemohormonal Therapy in Metastatic Hormone-Sensitive Prostate Cancer. , 2015, The New England journal of medicine.
[23] C. Evans,et al. Mechanisms of resistance in castration-resistant prostate cancer (CRPC) , 2015, Translational andrology and urology.
[24] E. Richardsen,et al. High Progesterone Receptor Expression in Prostate Cancer Is Associated with Clinical Failure , 2015, PloS one.
[25] Nicholas Y. Palermo,et al. The Hippo Pathway Effector YAP Regulates Motility, Invasion, and Castration-Resistant Growth of Prostate Cancer Cells , 2015, Molecular and Cellular Biology.
[26] M. Rubin,et al. ERG induces taxane resistance in castration-resistant prostate cancer , 2014, Nature Communications.
[27] J. Olsen,et al. In vivo quantitative phosphoproteomic profiling identifies novel regulators of castration-resistant prostate cancer growth , 2014, Oncogene.
[28] M. Gleave,et al. Prostate Stromal Cells Express the Progesterone Receptor to Control Cancer Cell Mobility , 2014, PloS one.
[29] N. Sharifi. Steroid receptors aplenty in prostate cancer. , 2014, The New England journal of medicine.
[30] H. Ji,et al. A peptide mimicking VGLL4 function acts as a YAP antagonist therapy against gastric cancer. , 2014, Cancer cell.
[31] D. Zheng,et al. Glucocorticoid Receptor Confers Resistance to Antiandrogens by Bypassing Androgen Receptor Blockade , 2013, Cell.
[32] B. Mellado,et al. Nuclear factor‐kappa B and interleukin‐6 related docetaxel resistance in castration‐resistant prostate cancer , 2013, The Prostate.
[33] H. Klocker,et al. Epithelial-to-mesenchymal transition leads to docetaxel resistance in prostate cancer and is mediated by reduced expression of miR-200c and miR-205. , 2012, The American journal of pathology.
[34] C. Cordon-Cardo,et al. Suppression of acquired docetaxel resistance in prostate cancer through depletion of notch- and hedgehog-dependent tumor-initiating cells. , 2012, Cancer cell.
[35] T. Habuchi,et al. Short-term clinicopathological outcome of neoadjuvant chemohormonal therapy comprising complete androgen blockade, followed by treatment with docetaxel and estramustine phosphate before radical prostatectomy in Japanese patients with high-risk localized prostate cancer , 2012, World Journal of Surgical Oncology.
[36] L. O’Driscoll,et al. Characterisation and manipulation of docetaxel resistant prostate cancer cell lines , 2011, Molecular Cancer.
[37] P. Troncoso,et al. Persistent, biologically meaningful prostate cancer after 1 year of androgen ablation and docetaxel treatment. , 2011, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[38] F. Désarnaud,et al. Gene expression profiling of the androgen independent prostate cancer cells demonstrates complex mechanisms mediating resistance to docetaxel , 2011, Cancer biology & therapy.
[39] R. Eeles,et al. TEAD1 and c-Cbl are novel prostate basal cell markers that correlate with poor clinical outcome in prostate cancer , 2008, British Journal of Cancer.
[40] Jiandie D. Lin,et al. TEAD mediates YAP-dependent gene induction and growth control. , 2008, Genes & development.
[41] T. Habuchi,et al. Candidate genes involved in enhanced growth of human prostate cancer under high fat feeding identified by microarray analysis , 2008, The Prostate.
[42] E. Sacco,et al. Long‐term follow‐up of a neoadjuvant chemohormonal taxane‐based phase II trial before radical prostatectomy in patients with non‐metastatic high‐risk prostate cancer , 2007, BJU international.
[43] Mikhail Teverovskiy,et al. Improved prediction of prostate cancer recurrence through systems pathology. , 2007, The Journal of clinical investigation.
[44] Young In Park,et al. Comparison of prostate cancer cell lines for androgen receptor-mediated reporter gene assays. , 2006, Toxicology in vitro : an international journal published in association with BIBRA.
[45] P. Febbo,et al. Neoadjuvant Docetaxel before Radical Prostatectomy in Patients with High-Risk Localized Prostate Cancer , 2005, Clinical Cancer Research.
[46] C. Tangen,et al. Docetaxel and estramustine compared with mitoxantrone and prednisone for advanced refractory prostate cancer. , 2004, The New England journal of medicine.
[47] I. Tannock,et al. Docetaxel plus prednisone or mitoxantrone plus prednisone for advanced prostate cancer. , 2004, The New England journal of medicine.
[48] M. Pfaffl,et al. Characterisation of gene expression patterns in 22RV1 cells for determination of environmental androgenic/antiandrogenic compounds , 2003, The Journal of Steroid Biochemistry and Molecular Biology.
[49] C. Tepper,et al. Characterization of a novel androgen receptor mutation in a relapsed CWR22 prostate cancer xenograft and cell line. , 2002, Cancer research.
[50] A. Jemal,et al. Cancer statistics, 2018 , 2018, CA: a cancer journal for clinicians.
[51] W. Ellis,et al. Docetaxel and mitoxantrone before radical prostatectomy in men with high-risk prostate cancer: 10-year follow-up and immune correlates , 2017, Anti-cancer drugs.
[52] Desok Kim,et al. Androgen receptor expression and cellular proliferation during transition from androgen-dependent to recurrent growth after castration in the CWR22 prostate cancer xenograft. , 2002, The American journal of pathology.