Calcium-Dependent Isoforms of Protein Kinase C Mediate Posttetanic Potentiation at the Calyx of Held
暂无分享,去创建一个
[1] T. Südhof,et al. Doc2 Supports Spontaneous Synaptic Transmission by a Ca2+-Independent Mechanism , 2011, Neuron.
[2] W. Ho,et al. Post-tetanic increase in the fast-releasing synaptic vesicle pool at the expense of the slowly releasing pool , 2010, The Journal of general physiology.
[3] L. Niels Cornelisse,et al. Doc2b Is a High-affinity Ca 2+ Sensor for Spontaneous Neurotransmitter Release , 2022 .
[4] T. Südhof,et al. Munc13 C2B-Domain – an Activity-Dependent Ca2+-Regulator of Synaptic Exocytosis , 2010, Nature Structural &Molecular Biology.
[5] Jianhua Xu,et al. Compound vesicle fusion increases quantal size and potentiates synaptic transmission , 2009, Nature.
[6] T. Branco,et al. The probability of neurotransmitter release: variability and feedback control at single synapses , 2009, Nature Reviews Neuroscience.
[7] M. Spira,et al. Activity-dependent calpain activation plays a critical role in synaptic facilitation and post-tetanic potentiation. , 2009, Learning & memory.
[8] Takeshi Sakaba,et al. Multiple Roles of Calcium Ions in the Regulation of Neurotransmitter Release , 2008, Neuron.
[9] W. Catterall,et al. Calcium Channel Regulation and Presynaptic Plasticity , 2008, Neuron.
[10] Nils Brose,et al. Phorbol Esters Modulate Spontaneous and Ca2+-Evoked Transmitter Release via Acting on Both Munc13 and Protein Kinase C , 2008, The Journal of Neuroscience.
[11] W. Ho,et al. Presynaptic Release Probability and Readily Releasable Pool Size Are Regulated by Two Independent Mechanisms during Posttetanic Potentiation at the Calyx of Held Synapse , 2008, The Journal of Neuroscience.
[12] W. Catterall,et al. Regulation of Presynaptic CaV2.1 Channels by Ca2+ Sensor Proteins Mediates Short-Term Synaptic Plasticity , 2008, Neuron.
[13] Doyun Lee,et al. Target Cell-Specific Involvement of Presynaptic Mitochondria in Post-Tetanic Potentiation at Hippocampal Mossy Fiber Synapses , 2007, The Journal of Neuroscience.
[14] R. Schneggenburger,et al. Posttetanic potentiation critically depends on an enhanced Ca2+ sensitivity of vesicle fusion mediated by presynaptic PKC , 2007, Proceedings of the National Academy of Sciences.
[15] M. Ghirardi,et al. Phosphorylation of synapsin domain A is required for post-tetanic potentiation , 2007, Journal of Cell Science.
[16] S. Corbalán-García,et al. The C2 domains of classical PKCs are specific PtdIns(4,5)P2-sensing domains with different affinities for membrane binding. , 2007, Journal of molecular biology.
[17] W. Regehr,et al. Differential Expression of Posttetanic Potentiation and Retrograde Signaling Mediate Target-Dependent Short-Term Synaptic Plasticity , 2007, Neuron.
[18] J. Borst,et al. Dynamics of the readily releasable pool during post‐tetanic potentiation in the rat calyx of Held synapse , 2007, The Journal of physiology.
[19] M. Verhage,et al. Interdependence of PKC-Dependent and PKC-Independent Pathways for Presynaptic Plasticity , 2007, Neuron.
[20] J. Falke,et al. Mechanism of specific membrane targeting by C2 domains: localized pools of target lipids enhance Ca2+ affinity. , 2007, Biochemistry.
[21] J. Borst,et al. An increase in calcium influx contributes to post-tetanic potentiation at the rat calyx of Held synapse. , 2006, Journal of neurophysiology.
[22] W. Regehr,et al. Sustained Elevation of Dendritic Calcium Evokes Widespread Endocannabinoid Release and Suppression of Synapses onto Cerebellar Purkinje Cells , 2006, The Journal of Neuroscience.
[23] Takeshi Sakaba,et al. Roles of the Fast-Releasing and the Slowly Releasing Vesicles in Synaptic Transmission at the Calyx of Held , 2006, The Journal of Neuroscience.
[24] Y. Sara,et al. Phorbol Esters Target the Activity-Dependent Recycling Pool and Spare Spontaneous Vesicle Recycling , 2005, The Journal of Neuroscience.
[25] R. Schneggenburger,et al. Allosteric modulation of the presynaptic Ca2+ sensor for vesicle fusion , 2005, Nature.
[26] R. Schneggenburger,et al. Presynaptic Ca2+ Requirements and Developmental Regulation of Posttetanic Potentiation at the Calyx of Held , 2005, The Journal of Neuroscience.
[27] J. Borst,et al. Post‐tetanic potentiation in the rat calyx of Held synapse , 2005, The Journal of physiology.
[28] D. Storm,et al. The role of calmodulin as a signal integrator for synaptic plasticity , 2005, Nature Reviews Neuroscience.
[29] Christian Rosenmund,et al. Calmodulin and Munc13 Form a Ca2+ Sensor/Effector Complex that Controls Short-Term Synaptic Plasticity , 2004, Cell.
[30] Wade G Regehr,et al. Presynaptic calcium measurements at physiological temperatures using a new class of dextran-conjugated indicators. , 2004, Journal of neurophysiology.
[31] Scott M Thompson,et al. Activity-dependent activation of presynaptic protein kinase C mediates post-tetanic potentiation , 2003, Nature Neuroscience.
[32] Nils Brose,et al. Move over protein kinase C, you've got company: alternative cellular effectors of diacylglycerol and phorbol esters , 2002, Journal of Cell Science.
[33] Joseph J. Falke,et al. C2 Domains of Protein Kinase C Isoforms α, β, and γ: Activation Parameters and Calcium Stoichiometries of the Membrane-Bound State , 2002 .
[34] M. Sajan,et al. Knockout of PKC Enhances Insulin Signaling Through PI3K , 2002 .
[35] Thomas C. Südhof,et al. β Phorbol Ester- and Diacylglycerol-Induced Augmentation of Transmitter Release Is Mediated by Munc13s and Not by PKCs , 2002, Cell.
[36] P. Jonas,et al. PTP and LTP at a hippocampal mossy fiber-interneuron synapse , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[37] Tomoyuki Takahashi,et al. Activation of the epsilon isoform of protein kinase C in the mammalian nerve terminal , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[38] Xin-sheng Wu,et al. Protein Kinase C Increases the Apparent Affinity of the Release Machinery to Ca2+ by Enhancing the Release Machinery Downstream of the Ca2+ Sensor , 2001, The Journal of Neuroscience.
[39] A. Newton,et al. Protein kinase C: structural and spatial regulation by phosphorylation, cofactors, and macromolecular interactions. , 2001, Chemical reviews.
[40] Edwin J. Weeber,et al. A Role for the β Isoform of Protein Kinase C in Fear Conditioning , 2000, The Journal of Neuroscience.
[41] B. Walmsley,et al. Phosphorylation regulates spontaneous and evoked transmitter release at a giant terminal in the rat auditory brainstem , 2000, The Journal of physiology.
[42] K. Svoboda,et al. Estimating intracellular calcium concentrations and buffering without wavelength ratioing. , 2000, Biophysical journal.
[43] Y. Takai,et al. Presynaptic Mechanism for Phorbol Ester-Induced Synaptic Potentiation , 1999, The Journal of Neuroscience.
[44] S. Corbalán-García,et al. Determination of the calcium-binding sites of the C2 domain of protein kinase Calpha that are critical for its translocation to the plasma membrane. , 1999, The Biochemical journal.
[45] L. Maler,et al. Differential roles of Ca2+/calmodulin-dependent kinases in posttetanic potentiation at input selective glutamatergic pathways. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[46] E R Kandel,et al. Involvement of Pre- and Postsynaptic Mechanisms in Posttetanic Potentiation at Aplysia Synapses , 1997, Science.
[47] Paul W. Frankland,et al. Impaired learning in mice with abnormal short-lived plasticity , 1996, Current Biology.
[48] A. Tarakhovsky,et al. Immunodeficiency in Protein Kinase Cβ-Deficient Mice , 1996, Science.
[49] Alcino J. Silva,et al. The α-Ca2+/calmodulin kinase II: A bidirectional modulator of presynaptic plasticity , 1995, Neuron.
[50] D W Tank,et al. A quantitative measurement of the dependence of short-term synaptic enhancement on presynaptic residual calcium , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[51] L. Eliot,et al. Modulation of spontaneous transmitter release during depression and posttetanic potentiation of Aplysia sensory-motor neuron synapses isolated in culture , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[52] D W Tank,et al. The role of presynaptic calcium in short-term enhancement at the hippocampal mossy fiber synapse , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[53] D. Madison,et al. Phorbol esters enhance synaptic transmission by a presynaptic, calcium‐dependent mechanism in rat hippocampus. , 1993, The Journal of physiology.
[54] W. Griffith,et al. Voltage-clamp analysis of posttetanic potentiation of the mossy fiber to CA3 synapse in hippocampus. , 1990, Journal of neurophysiology.
[55] R S Zucker,et al. Calcium in motor nerve terminals associated with posttetanic potentiation , 1989, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[56] Hiroshi Kase,et al. Inhibitors of calmodulin and protein kinase C block different phases of hippocampal long-term potentiation , 1988, Brain Research.
[57] Klaus G. Reymann,et al. Polymyxin B, an inhibitor of protein kinase C, prevents the maintenance of synaptic long-term potentiation in hippocampal CA1 neurons , 1988, Brain Research.
[58] R. Rahamimoff,,et al. Ionic basis of tetanic and post‐tetanic potentiation at a mammalian neuromuscular junction. , 1988, The Journal of physiology.
[59] Robert C. Malenka,et al. Potentiation of synaptic transmission in the hippocampus by phorbol esters , 1986, Nature.
[60] R. Zucker,et al. Post-tetanic decay of evoked and spontaneous transmitter release and a residual-calcium model of synaptic facilitation at crayfish neuromuscular junctions , 1983, The Journal of general physiology.
[61] A. Lev-Tov,et al. A study of tetanic and post‐tetanic potentiation of miniature end‐plate potentials at the frog neuromuscular junction. , 1980, The Journal of physiology.
[62] K L Magleby,et al. A quantitative description of tetanic and post‐tetanic potentiation of transmitter release at the frog neuromuscular junction. , 1975, The Journal of physiology.
[63] J. Eccles,et al. Presynaptic changes associated with post‐tetanic potentiation in the spinal cord , 1959, The Journal of physiology.
[64] B. Katz,et al. Statistical factors involved in neuromuscular facilitation and depression , 1954, The Journal of physiology.
[65] L. Abbott,et al. Synaptic computation , 2004, Nature.
[66] J. Falke,et al. C2 domains of protein kinase C isoforms alpha, beta, and gamma: activation parameters and calcium stoichiometries of the membrane-bound state. , 2002, Biochemistry.
[67] M. Sajan,et al. Knockout of PKC alpha enhances insulin signaling through PI3K. , 2002, Molecular endocrinology.
[68] W. Regehr,et al. Short-term synaptic plasticity. , 2002, Annual review of physiology.
[69] J. Sweatt,et al. A role for the beta isoform of protein kinase C in fear conditioning. , 2000, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[70] A. Tarakhovsky,et al. Immunodeficiency in protein kinase cbeta-deficient mice. , 1996, Science.
[71] P. Chapman,et al. The alpha-Ca2+/calmodulin kinase II: a bidirectional modulator of presynaptic plasticity. , 1995, Neuron.
[72] S. Silberberg,et al. Activation of protein kinase C augments evoked transmitter release , 1987, Nature.
[73] K L Magleby,et al. Facilitation, augmentation, and potentiation of transmitter release. , 1979, Progress in brain research.