Brain energization in response to deep brain stimulation of subthalamic nuclei in Parkinson's disease

[1]  A. Bentivoglio,et al.  Motor and cognitive outcome in patients with Parkinson's disease 8 years after subthalamic implants. , 2010, Brain : a journal of neurology.

[2]  Y. Jeong,et al.  Cerebral Glucose Metabolism of Parkinson’s Disease Patients with Mild Cognitive Impairment , 2010, European Neurology.

[3]  Albert Gjedde,et al.  Subcortical elevation of metabolism in Parkinson's disease — A critical reappraisal in the context of global mean normalization , 2009, NeuroImage.

[4]  Marjan Jahanshahi,et al.  PET Functional Imaging of Deep Brain Stimulation in Movement Disorders and Psychiatry , 2009, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[5]  G. Dimitrakopoulos,et al.  Functional architecture , 2009, IEEE Vehicular Technology Magazine.

[6]  Nicola J. Ray,et al.  Anatomy, physiology, and pathophysiology of the pedunculopontine nucleus , 2009, Movement disorders : official journal of the Movement Disorder Society.

[7]  W D Heiss,et al.  Frontal FDG-PET activity correlates with cognitive outcome after STN-DBS in Parkinson disease , 2009, Neurology.

[8]  Alexander Hammers,et al.  SPM-based count normalization provides excellent discrimination of mild Alzheimer's disease and amnestic mild cognitive impairment from healthy aging , 2009, NeuroImage.

[9]  Albert Gjedde,et al.  STN‐stimulation in Parkinson's disease restores striatal inhibition of thalamocortical projection , 2009, Human brain mapping.

[10]  Y. Guan,et al.  Modulation of metabolic brain function by bilateral subthalamic nucleus stimulation in the treatment of Parkinson’s disease , 2008, Journal of Neurology.

[11]  Erwin B. Montgomery,et al.  Mechanisms of action of deep brain stimulation (DBS) , 2008, Neuroscience & Biobehavioral Reviews.

[12]  M. C. Campbell,et al.  Neural correlates of STN DBS-induced cognitive variability in Parkinson disease , 2008, Neuropsychologia.

[13]  J. Mink,et al.  Subthalamic nucleus stimulation-induced regional blood flow responses correlate with improvement of motor signs in Parkinson disease. , 2008, Brain : a journal of neurology.

[14]  W D Heiss,et al.  STN-DBS activates the target area in Parkinson disease , 2008, Neurology.

[15]  M. Taniguchi,et al.  Mechanisms of unilateral STN-DBS in patients with Parkinson’s disease , 2008, Journal of Neurology.

[16]  John Ashburner,et al.  A fast diffeomorphic image registration algorithm , 2007, NeuroImage.

[17]  E. Tolosa,et al.  Clinical diagnostic criteria for dementia associated with Parkinson's disease , 2007, Movement disorders : official journal of the Movement Disorder Society.

[18]  V. Dhawan,et al.  Abnormal Metabolic Network Activity in Parkinson'S Disease: Test—Retest Reproducibility , 2007, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[19]  V. Dhawan,et al.  Network modulation in the treatment of Parkinson's disease. , 2006, Brain : a journal of neurology.

[20]  Anna Barnes,et al.  Network modulation by the subthalamic nucleus in the treatment of Parkinson's disease , 2006, NeuroImage.

[21]  Bernhard Haslinger,et al.  Frequency-correlated decreases of motor cortex activity associated with subthalamic nucleus stimulation in Parkinson's disease , 2005, NeuroImage.

[22]  J. Thie.  Understanding the standardized uptake value, its methods, and implications for usage. , 2004, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[23]  Pierre Payoux,et al.  Subthalamic nucleus stimulation reduces abnormal motor cortical overactivity in Parkinson disease. , 2004, Archives of neurology.

[24]  Paul Cumming,et al.  Focal changes of oxygen consumption in cerebral cortex of patients with Parkinson's disease during subthalamic stimulation , 2004, NeuroImage.

[25]  V. Sturm,et al.  Subthalamic Nucleus Stimulation Restores Glucose Metabolism in Associative and Limbic Cortices and in Cerebellum: Evidence from a FDG-PET Study in Advanced Parkinson's Disease , 2004, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[26]  P. Maquet,et al.  Reproducibility of cerebral glucose utilization measured by PET and the [18F]-2-fluoro-2-deoxy-d-glucose method in resting, healthy human subjects , 2004, European Journal of Nuclear Medicine.

[27]  A. Benabid,et al.  Five-year follow-up of bilateral stimulation of the subthalamic nucleus in advanced Parkinson's disease. , 2003, The New England journal of medicine.

[28]  Jean-Claude Baron,et al.  Resting-state brain glucose utilization as measured by PET is directly related to regional synaptophysin levels: a study in baboons , 2003, NeuroImage.

[29]  Alberto Pupi,et al.  Changes in regional cerebral blood flow caused by deep-brain stimulation of the subthalamic nucleus in Parkinson's disease. , 2002, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[30]  Daniel J Buysse,et al.  Human regional cerebral glucose metabolism during non-rapid eye movement sleep in relation to waking. , 2002, Brain : a journal of neurology.

[31]  M Samii,et al.  Resting regional cerebral glucose metabolism in advanced Parkinson's disease studied in the off and on conditions with [18F]FDG‐PET , 2001, Movement disorders : official journal of the Movement Disorder Society.

[32]  S. Laureys,et al.  Comparison of Impaired Subcortico-Frontal Metabolic Networks in Normal Aging, Subcortico-Frontal Dementia, and Cortical Frontal Dementia , 1999, NeuroImage.

[33]  B Conrad,et al.  A positron emission tomographic study of subthalamic nucleus stimulation in Parkinson disease: enhanced movement-related activity of motor-association cortex and decreased motor cortex resting activity. , 1999, Archives of neurology.

[34]  P. Magistretti,et al.  Cellular mechanisms of brain energy metabolism and their relevance to functional brain imaging. , 1999, Philosophical transactions of the Royal Society of London. Series B, Biological sciences.

[35]  Richard S. J. Frackowiak,et al.  Changes in cerebral activity pattern due to subthalamic nucleus or internal pallidum stimulation in Parkinson's disease , 1997, Annals of neurology.

[36]  T. Ishikawa,et al.  The Metabolic Topography of Parkinsonism , 1994, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[37]  J. Hughes,et al.  Accuracy of clinical diagnosis of idiopathic Parkinson's disease: a clinico-pathological study of 100 cases. , 1992, Journal of neurology, neurosurgery, and psychiatry.

[38]  M. Delong,et al.  Primate models of movement disorders of basal ganglia origin , 1990, Trends in Neurosciences.

[39]  G. E. Alexander,et al.  Functional architecture of basal ganglia circuits: neural substrates of parallel processing , 1990, Trends in Neurosciences.

[40]  Karl J. Friston,et al.  The Relationship between Global and Local Changes in PET Scans , 1990, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[41]  S. Folstein,et al.  "Mini-mental state". A practical method for grading the cognitive state of patients for the clinician. , 1975, Journal of psychiatric research.