Bcl-2 Protection of Mitochondrial Function Following Chemical Hypoxia/Aglycemia
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[1] D. Bredesen,et al. Bcl‐2 Protects Neural Cells from Cyanide/Aglycemia‐Induced Lipid Oxidation, Mitochondrial Injury, and Loss of Viability , 1995, Journal of neurochemistry.
[2] M. Goldberg,et al. Mitochondrial production of reactive oxygen species in cortical neurons following exposure to N-methyl-D-aspartate , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[3] John Calvin Reed,et al. Upregulation of bax protein levels in neurons following cerebral ischemia , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[4] M. Geschwind,et al. Expression of bcl-2 from a defective herpes simplex virus-1 vector limits neuronal death in focal cerebral ischemia. , 1995, Stroke.
[5] E. Preston,et al. Differences in DNA Fragmentation following Transient Cerebral or Decapitation Ischemia in Rats , 1995, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[6] I. Reynolds,et al. Glutamate induces the production of reactive oxygen species in cultured forebrain neurons following NMDA receptor activation , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[7] M. Raff,et al. Programmed cell death and Bcl-2 protection in very low oxygen , 1995, Nature.
[8] Y. Tsujimoto,et al. Prevention of hypoxia-induced cell death by Bcl-2 and Bcl-xL , 1995, Nature.
[9] Y. Uchiyama,et al. Delayed neuronal death in the CA1 pyramidal cell layer of the gerbil hippocampus following transient ischemia is apoptosis , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[10] R. White,et al. Mitochondria and Na+/Ca2+ exchange buffer glutamate-induced calcium loads in cultured cortical neurons , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[11] Jun Chen,et al. bcl-2 is expressed in neurons that survive focal ischemia in the rat. , 1995, Neuroreport.
[12] D. Newmeyer,et al. Cell-free apoptosis in Xenopus egg extracts: Inhibition by Bcl-2 and requirement for an organelle fraction enriched in mitochondria , 1994, Cell.
[13] L. Wang,et al. Ich-1, an Ice/ced-3-related gene, encodes both positive and negative regulators of programmed cell death , 1994, Cell.
[14] K. Gunter,et al. Mitochondrial calcium transport: physiological and pathological relevance. , 1994, The American journal of physiology.
[15] G. Dubyak,et al. Evidence that BCL-2 represses apoptosis by regulating endoplasmic reticulum-associated Ca2+ fluxes. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[16] T. Shiraishi,et al. Visualization of DNA double strand breaks in the gerbil hippocampal CA1 following transient ischemia , 1994, Neuroscience Letters.
[17] A. Ishida,et al. Increase in bcl-2 oncoprotein and the tolerance to ischemia-induced neuronal death in the gerbil hippocampus , 1994, Neuroscience Research.
[18] D. Gilboe,et al. Ischemia‐Induced Changes in Cerebral Mitochondrial Free Fatty Acids, Phospholipids, and Respiration in the Rat , 1994, Journal of neurochemistry.
[19] R. Lin,et al. Internucleosomal DNA fragmentation in gerbil hippocampus following forebrain ischemia , 1994, Neuroscience Letters.
[20] M. Dubois‐Dauphin,et al. Neonatal motoneurons overexpressing the bcl-2 protooncogene in transgenic mice are protected from axotomy-induced cell death. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[21] John Calvin Reed. Bcl-2 and the regulation of programmed cell death , 1994, The Journal of cell biology.
[22] M. Linnik,et al. Evidence Supporting a Role for Programmed Cell Death in Focal Cerebral Ischemia in Rats , 1993, Stroke.
[23] D. Bredesen,et al. Bcl-2 inhibition of neural death: decreased generation of reactive oxygen species. , 1993, Science.
[24] K. Mikoshiba,et al. Internucleosomal DNA cleavage involved in ischemia-induced neuronal death. , 1993, Biochemical and biophysical research communications.
[25] Y. Ben-Ari,et al. Rapid Communication: Regional Variability in DNA Fragmentation After Global Ischemia Evidenced by Combined Histological and Gel Electrophoresis Observations in the Rat Brain , 1993, Journal of neurochemistry.
[26] Z. Oltvai,et al. Bcl-2 functions in an antioxidant pathway to prevent apoptosis , 1993, Cell.
[27] John Calvin Reed,et al. Investigation of the subcellular distribution of the bcl-2 oncoprotein: residence in the nuclear envelope, endoplasmic reticulum, and outer mitochondrial membranes. , 1993, Cancer research.
[28] D. Bredesen,et al. bcl-2 inhibits death of central neural cells induced by multiple agents. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[29] D. Bredesen,et al. The Protooncogene bcl‐2 Inhibits Apoptosis in PC12 Cells , 1993, Journal of neurochemistry.
[30] B. Siesjö. Pathophysiology and treatment of focal cerebral ischemia. Part II: Mechanisms of damage and treatment. , 1992, Journal of neurosurgery.
[31] A. Fabi,et al. Ischemic injury to rat forebrain mitochondria and cellular calcium homeostasis. , 1992, Biochimica et biophysica acta.
[32] A. Cross,et al. Enzymic mechanisms of superoxide production. , 1991, Biochimica et biophysica acta.
[33] Seamus J. Martin,et al. Dose‐dependent induction of apoptosis in human tumour cell lines by widely diverging stimuli , 1991, Cell proliferation.
[34] R. Schreiber,et al. Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell death , 1990, Nature.
[35] T. Gunter,et al. Mechanisms by which mitochondria transport calcium. , 1990, The American journal of physiology.
[36] R. Koehler,et al. Postischemic recovery rate of cerebral ATP, phosphocreatine, pH, and evoked potentials. , 1989, The American journal of physiology.
[37] M. Cleary,et al. The bcl-2 candidate proto-oncogene product is a 24-kilodalton integral-membrane protein highly expressed in lymphoid cell lines and lymphomas carrying the t(14;18) translocation , 1989, Molecular and cellular biology.
[38] T. Buttke,et al. Oxidative stress as a mediator of apoptosis. , 1994, Immunology today.