AAA+ ATPases Reptin and Pontin as potential diagnostic and prognostic biomarkers in salivary gland cancer - a short report

[1]  J. Bishop,et al.  Newly Described Entities in Salivary Gland Pathology. , 2017, The American journal of surgical pathology.

[2]  Sung-Dae Cho,et al.  Silymarin and its active component silibinin act as novel therapeutic alternatives for salivary gland cancer by targeting the ERK1/2-Bim signaling cascade , 2017, Cellular Oncology.

[3]  Sung-Dae Cho,et al.  Silymarin and its active component silibinin act as novel therapeutic alternatives for salivary gland cancer by targeting the ERK1/2-Bim signaling cascade , 2017, Cellular Oncology.

[4]  E. Wardelmann,et al.  Prognostic relevance of epithelial–mesenchymal transition and proliferation in surgically treated primary parotid gland cancer , 2016, Journal of Clinical Pathology.

[5]  M. Odenthal,et al.  MDM2 and CDK4 amplifications are rare events in salivary duct carcinomas , 2016, Oncotarget.

[6]  M. McGurk,et al.  Management of Salivary Gland Tumours: United Kingdom National Multidisciplinary Guidelines , 2016, The Journal of Laryngology & Otology.

[7]  M. Odenthal,et al.  Targeted next generation sequencing of parotid gland cancer uncovers genetic heterogeneity , 2015, Oncotarget.

[8]  Anindya Dutta,et al.  The AAA+ proteins Pontin and Reptin enter adult age: from understanding their basic biology to the identification of selective inhibitors , 2015, Front. Mol. Biosci..

[9]  Dawei Li,et al.  Cytoplasmic Expression of Pontin in Renal Cell Carcinoma Correlates with Tumor Invasion, Metastasis and Patients’ Survival , 2015, PloS one.

[10]  F. Persson,et al.  Diagnostic and therapeutic implications of new molecular biomarkers in salivary gland cancers. , 2014, Oral oncology.

[11]  S. Bras,et al.  Pontin is a critical regulator for AML1-ETO-induced leukemia , 2014, Leukemia.

[12]  Dawei Li,et al.  Overexpression of reptin in renal cell carcinoma contributes to tumor malignancies and its inhibition triggers senescence of cancer cells. , 2013, Urologic oncology.

[13]  H. Buhr,et al.  Increased Pontin expression is a potential predictor for outcome in sporadic colorectal carcinoma. , 2012, Oncology reports.

[14]  T. Zander,et al.  Definition of a fluorescence in-situ hybridization score identifies high- and low-level FGFR1 amplification types in squamous cell lung cancer , 2012, Modern Pathology.

[15]  Michel Laguerre,et al.  First identification of small-molecule inhibitors of Pontin by combining virtual screening and enzymatic assay. , 2012, The Biochemical journal.

[16]  Aude Grigoletto,et al.  The multifaceted proteins Reptin and Pontin as major players in cancer. , 2011, Biochimica et biophysica acta.

[17]  T. Hupp,et al.  A divergent substrate-binding loop within the pro-oncogenic protein anterior gradient-2 forms a docking site for Reptin. , 2010, Journal of molecular biology.

[18]  Aude Grigoletto,et al.  In vivo silencing of Reptin blocks the progression of human hepatocellular carcinoma in xenografts and is associated with replicative senescence. , 2010, Journal of hepatology.

[19]  B. Perez-Ordonez,et al.  Mammary Analogue Secretory Carcinoma of Salivary Glands, Containing the ETV6-NTRK3 Fusion Gene: A Hitherto Undescribed Salivary Gland Tumor Entity , 2010, The American journal of surgical pathology.

[20]  M. McGurk,et al.  Major and minor salivary gland tumors. , 2003, Critical reviews in oncology/hematology.

[21]  J. Zucman‐Rossi,et al.  Adenosine triphosphatase pontin is overexpressed in hepatocellular carcinoma and coregulated with reptin through a new posttranslational mechanism , 2009, Hepatology.

[22]  Anindya Dutta,et al.  RVB1/RVB2: running rings around molecular biology. , 2009, Molecular cell.

[23]  O. Huber,et al.  Pontin and reptin, two related ATPases with multiple roles in cancer. , 2008, Cancer research.

[24]  Jörn Gröne,et al.  Increased pontin expression in human colorectal cancer tissue. , 2007, Human pathology.

[25]  A. Shimomura,et al.  Pitfalls in the Detection of t(11;22) Translocation by Fluorescence In Situ Hybridization and RT-PCR: A Single-blinded Study , 2006, Diagnostic molecular pathology : the American journal of surgical pathology, part B.

[26]  O. Huber,et al.  The histidine triad protein Hint1 interacts with Pontin and Reptin and inhibits TCF–β-catenin-mediated transcription , 2005, Journal of Cell Science.

[27]  A. Cancellieri,et al.  Prognostic relevance of cell proliferation in major salivary gland carcinomas. , 2005, Acta otorhinolaryngologica Italica : organo ufficiale della Societa italiana di otorinolaringologia e chirurgia cervico-facciale.

[28]  Hee June Choi,et al.  Transcriptional regulation of a metastasis suppressor gene by Tip60 and β-catenin complexes , 2005, Nature.

[29]  B. Gumbiner,et al.  Distinct molecular forms of β-catenin are targeted to adhesive or transcriptional complexes , 2004, The Journal of cell biology.

[30]  S. Prescott,et al.  Regulation of COX-2 transcription in a colon cancer cell line by Pontin52/TIP49a , 2003, Molecular Cancer.

[31]  W. Rottbauer,et al.  Reptin and Pontin Antagonistically Regulate Heart Growth in Zebrafish Embryos , 2002, Cell.

[32]  R. Zarbo Salivary Gland Neoplasia: A Review for the Practicing Pathologist , 2002, Modern Pathology.

[33]  B. Gumbiner,et al.  E-Cadherin Suppresses Cellular Transformation by Inhibiting β-Catenin Signaling in an Adhesion-Independent Manner , 2001, The Journal of cell biology.

[34]  A. Bauer,et al.  Pontin52 and Reptin52 function as antagonistic regulators of β‐catenin signalling activity , 2000, The EMBO journal.

[35]  Frank McCormick,et al.  β-Catenin regulates expression of cyclin D1 in colon carcinoma cells , 1999, Nature.

[36]  A. Bauer,et al.  Pontin52, an interaction partner of beta-catenin, binds to the TATA box binding protein. , 1998, Proceedings of the National Academy of Sciences of the United States of America.

[37]  R. Spiro,et al.  Malignant tumors of major salivary gland origin. A matched-pair analysis of the role of combined surgery and postoperative radiotherapy. , 1990, Archives of otolaryngology--head & neck surgery.