Heterogeneity of neoplastic stem cells: theoretical, functional, and clinical implications.
暂无分享,去创建一个
C. Eaves | M. Andreeff | M. Copland | P. Valent | D. Bonnet | C. Chomienne | S. Wöhrer
[1] M. Esteller,et al. Epigenetic alterations involved in cancer stem cell reprogramming , 2012, Molecular oncology.
[2] H. Johnsen,et al. Cancer stem cell definitions and terminology: the devil is in the details , 2012, Nature Reviews Cancer.
[3] S. Yamanaka. Induced pluripotent stem cells: past, present, and future. , 2012, Cell stem cell.
[4] P. Varlet,et al. High frequency of germline SUFU mutations in children with desmoplastic/nodular medulloblastoma younger than 3 years of age. , 2012, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[5] A. Van Keymeulen,et al. Tracing epithelial stem cells during development, homeostasis, and repair , 2012, The Journal of cell biology.
[6] S. Beà,et al. SNP-based mapping arrays reveal high genomic complexity in monoclonal gammopathies, from MGUS to myeloma status , 2012, Leukemia.
[7] David A. Williams,et al. Rac2-MRC-cIII-generated ROS cause genomic instability in chronic myeloid leukemia stem cells and primitive progenitors. , 2012, Blood.
[8] K. Polyak,et al. Intra-tumour heterogeneity: a looking glass for cancer? , 2012, Nature Reviews Cancer.
[9] Christine A Iacobuzio-Donahue,et al. A new branch on the tree: next-generation sequencing in the study of cancer evolution. , 2012, Seminars in cell & developmental biology.
[10] Huanming Yang,et al. Single-Cell Exome Sequencing and Monoclonal Evolution of a JAK2-Negative Myeloproliferative Neoplasm , 2012, Cell.
[11] M. Mimeault,et al. Pathobiological Implications of the Expression of EGFR, pAkt, NF-κB and MIC-1 in Prostate Cancer Stem Cells and Their Progenies , 2012, PloS one.
[12] J. Opitz,et al. Germline mutations in DIS3L2 cause the Perlman syndrome of overgrowth and Wilms tumor susceptibility , 2012, Nature Genetics.
[13] T. Suda,et al. Regulation of reactive oxygen species in stem cells and cancer stem cells , 2012, Journal of cellular physiology.
[14] Samuel Aparicio,et al. Opening Pandora's Box--the new biology of driver mutations and clonal evolution in cancer as revealed by next generation sequencing. , 2012, Current opinion in genetics & development.
[15] Carlo C. Maley,et al. Clonal evolution in cancer , 2012, Nature.
[16] D. Tang,et al. Understanding cancer stem cell heterogeneity and plasticity , 2012, Cell Research.
[17] N. Talley,et al. Prevalence of colonic neoplasia and advanced lesions in the normal population: a prospective population-based colonoscopy study , 2012, Scandinavian journal of gastroenterology.
[18] Peter Dirks,et al. Cancer stem cells: an evolving concept , 2012, Nature Reviews Cancer.
[19] T. Graf,et al. Historical origins of transdifferentiation and reprogramming. , 2011, Cell stem cell.
[20] M. Greaves,et al. Developmental origins and impact of BCR-ABL1 fusion and IKZF1 deletions in monozygotic twins with Ph+ acute lymphoblastic leukemia. , 2011, Blood.
[21] Christopher J. R. Illingworth,et al. Distinguishing Driver and Passenger Mutations in an Evolutionary History Categorized by Interference , 2011, Genetics.
[22] Peter A. Jones,et al. A decade of exploring the cancer epigenome — biological and translational implications , 2011, Nature Reviews Cancer.
[23] R. Levine,et al. Molecular biology of myelodysplastic syndromes. , 2011, Seminars in oncology.
[24] Guy S. Salvesen,et al. SnapShot: Caspases , 2011, Cell.
[25] Paul A. Wiggins,et al. Normal and neoplastic nonstem cells can spontaneously convert to a stem-like state , 2011, Proceedings of the National Academy of Sciences.
[26] F. Sigaux,et al. Clonal selection in xenografted human T cell acute lymphoblastic leukemia recapitulates gain of malignancy at relapse , 2011, The Journal of experimental medicine.
[27] Hans Clevers,et al. The cancer stem cell: premises, promises and challenges , 2011, Nature Medicine.
[28] D. Hanahan,et al. Hallmarks of Cancer: The Next Generation , 2011, Cell.
[29] P. Vyas,et al. Coexistence of LMPP-like and GMP-like leukemia stem cells in acute myeloid leukemia. , 2011, Cancer cell.
[30] B. Kong,et al. A Proposed Model for Endometrial Serous Carcinogenesis , 2011, The American journal of surgical pathology.
[31] S. Morrison,et al. Phenotypic heterogeneity among tumorigenic melanoma cells from patients that is reversible and not hierarchically organized. , 2010, Cancer cell.
[32] C. Eaves,et al. Insights into the stem cells of chronic myeloid leukemia , 2010, Leukemia.
[33] A. Ashworth,et al. BRCA1 basal-like breast cancers originate from luminal epithelial progenitors and not from basal stem cells. , 2010, Cell stem cell.
[34] T. Skorski,et al. Chronic myeloid leukemia: mechanisms of blastic transformation. , 2010, The Journal of clinical investigation.
[35] T. Halazonetis,et al. Genomic instability — an evolving hallmark of cancer , 2010, Nature Reviews Molecular Cell Biology.
[36] T. Enver,et al. Forcing cells to change lineages , 2009, Nature.
[37] E. Laurenti,et al. Balancing dormant and self-renewing hematopoietic stem cells. , 2009, Current opinion in genetics & development.
[38] S. Fox,et al. Aberrant luminal progenitors as the candidate target population for basal tumor development in BRCA1 mutation carriers , 2009, Nature Medicine.
[39] Tao Zhang,et al. Aldehyde dehydrogenase 1 is a marker for normal and malignant human colonic stem cells (SC) and tracks SC overpopulation during colon tumorigenesis. , 2009, Cancer research.
[40] T. Naoe,et al. BCR-ABL-transformed GMP as myeloid leukemic stem cells , 2008, Proceedings of the National Academy of Sciences.
[41] Trachette L. Jackson,et al. Pathways to tumorigenesis--modeling mutation acquisition in stem cells and their progeny. , 2008, Neoplasia.
[42] Jinshui Fan,et al. Genomic instability in myeloid malignancies: increased reactive oxygen species (ROS), DNA double strand breaks (DSBs) and error-prone repair. , 2008, Cancer letters.
[43] R. Kralovics. Genetic complexity of myeloproliferative neoplasms , 2008, Leukemia.
[44] C. Jamieson,et al. Chronic myeloid leukemia stem cells. , 2008, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[45] J. Knoblich,et al. Mechanisms of Asymmetric Stem Cell Division , 2008, Cell.
[46] G. Haas,et al. The worldwide epidemiology of prostate cancer: perspectives from autopsy studies. , 2008, The Canadian journal of urology.
[47] Junia V. Melo,et al. Chronic myeloid leukaemia as a model of disease evolution in human cancer , 2007, Nature Reviews Cancer.
[48] D. Kent,et al. Identification of a new intrinsically timed developmental checkpoint that reprograms key hematopoietic stem cell properties , 2007, Proceedings of the National Academy of Sciences.
[49] Soon-Siong Teo,et al. Leukemic blasts in transformed JAK2-V617F-positive myeloproliferative disorders are frequently negative for the JAK2-V617F mutation. , 2006, Blood.
[50] P. Campbell,et al. Mutation of JAK2 in the myeloproliferative disorders: timing, clonality studies, cytogenetic associations, and role in leukemic transformation. , 2006, Blood.
[51] Hongyue Dai,et al. Gene expression changes associated with progression and response in chronic myeloid leukemia. , 2006, Proceedings of the National Academy of Sciences of the United States of America.
[52] C. Eaves,et al. Different subsets of primary chronic myeloid leukemia stem cells engraft immunodeficient mice and produce a model of the human disease , 2005, Leukemia.
[53] K. Kinzler,et al. Cancer genes and the pathways they control , 2004, Nature Medicine.
[54] J. Dick,et al. Acute myeloid leukemia originates from a hierarchy of leukemic stem cell classes that differ in self-renewal capacity , 2004, Nature Immunology.
[55] C. Eaves,et al. Engraftment of NOD/SCID-beta2 microglobulin null mice with multilineage neoplastic cells from patients with myelodysplastic syndrome. , 2004, Blood.
[56] S. Tavaré,et al. Pretumor progression: clonal evolution of human stem cell populations. , 2004, The American journal of pathology.
[57] Gonzalez,et al. Insights into the multistep transformation of MGUS to myeloma using microarray expression analysis. , 2003, Blood.
[58] Miae Lee,et al. Acute lymphoblastic leukemia without the Philadelphia chromosome occurring in chronic myelogenous leukemia with the Philadelphia chromosome , 2003, American journal of hematology.
[59] A. Strife,et al. Chronic myelogenous leukemia as a paradigm of early cancer and possible curative strategies , 2003, Leukemia.
[60] R. Liang,et al. Philadelphia (Ph) chromosome‐positive chronic myeloid leukaemia relapsing as Ph‐negative leukaemia after allogeneic bone marrow transplantation , 2001, British journal of haematology.
[61] K. Kinzler,et al. Genetic instability and darwinian selection in tumours. , 1999, Trends in cell biology.
[62] D. van der Kooy,et al. In vivo clonal analyses reveal the properties of endogenous neural stem cell proliferation in the adult mammalian forebrain. , 1998, Development.
[63] J. Dick,et al. Human acute myeloid leukemia is organized as a hierarchy that originates from a primitive hematopoietic cell , 1997, Nature Medicine.
[64] P. Nowell. The clonal evolution of tumor cell populations. , 1976, Science.
[65] G. B. Pierce,et al. Differentiation of malignant to benign cells. , 1971, Cancer research.
[66] For April. , 1903 .
[67] R. Arceci. Developmental origins and impact of BCR-ABL1 fusion and IKZF1 deletions in monozygotic twins with Ph+ acute lymphoblastic leukemia , 2012 .
[68] R. Arceci. Intratumor Heterogeneity and Branched Evolution Revealed by Multiregion Sequencing , 2012 .
[69] Martin Krapcho,et al. SEER Cancer Statistics Review, 1975–2009 (Vintage 2009 Populations) , 2012 .
[70] P. Valent. Targeting of leukemia-initiating cells to develop curative drug therapies: straightforward but nontrivial concept. , 2011, Current cancer drug targets.
[71] R. Arceci. Evolution of human BCR–ABL1 lymphoblastic leukaemia-initiating cells , 2011 .
[72] P. Valent,et al. Idiopathic bone marrow dysplasia of unknown significance (IDUS): definition, pathogenesis, follow up, and prognosis. , 2011, American journal of cancer research.
[73] G. Ghanem,et al. Genotypic and gene expression studies in congenital melanocytic nevi: insight into initial steps of melanotumorigenesis. , 2009, The Journal of investigative dermatology.
[74] E. Feuer,et al. SEER Cancer Statistics Review, 1975-2003 , 2006 .
[75] J. Goldman,et al. Normal and chronic phase CML hematopoietic cells repopulate NOD/SCID bone marrow with different kinetics and cell lineage representation. , 2000, The hematology journal : the official journal of the European Haematology Association.
[76] C. Eaves,et al. The biology of normal and neoplastic stem cells in CML. , 1993, Leukemia & lymphoma.
[77] Wenjun Guo,et al. Slug and Sox 9 Cooperatively Determine the Mammary Stem Cell State Citation , 2022 .