Structural organization of the inactive X chromosome in the mouse

[1]  William Stafford Noble,et al.  Bipartite structure of the inactive mouse X chromosome , 2015, Genome Biology.

[2]  Jeannie T. Lee,et al.  Chromosomes. A comprehensive Xist interactome reveals cohesin repulsion and an RNA-directed chromosome conformation. , 2015, Science.

[3]  D. Gallie.  Faculty Opinions recommendation of The Xist lncRNA interacts directly with SHARP to silence transcription through HDAC3. , 2015 .

[4]  J. Dekker,et al.  Condensin-Driven Remodeling of X-Chromosome Topology during Dosage Compensation , 2015, Nature.

[5]  Qiangfeng Cliff Zhang,et al.  Systematic Discovery of Xist RNA Binding Proteins , 2015, Cell.

[6]  William Stafford Noble,et al.  The lncRNA Firre anchors the inactive X chromosome to the nucleolus by binding CTCF and maintains H3K27me3 methylation , 2015, Genome Biology.

[7]  William Stafford Noble,et al.  Escape from X Inactivation Varies in Mouse Tissues , 2015, PLoS genetics.

[8]  D. Odom,et al.  Comparative Hi-C Reveals that CTCF Underlies Evolution of Chromosomal Domain Architecture , 2015, Cell reports.

[9]  Noam Kaplan,et al.  The Hitchhiker's guide to Hi-C analysis: practical guidelines. , 2015, Methods.

[10]  Neva C. Durand,et al.  A 3D Map of the Human Genome at Kilobase Resolution Reveals Principles of Chromatin Looping , 2014, Cell.

[11]  Carolyn J. Brown,et al.  Variable escape from X-chromosome inactivation: Identifying factors that tip the scales towards expression , 2014, BioEssays : news and reviews in molecular, cellular and developmental biology.

[12]  Jan H Bergmann,et al.  Random monoallelic gene expression increases upon embryonic stem cell differentiation. , 2014, Developmental cell.

[13]  E. Barillot,et al.  Developmental dynamics and disease potential of random monoallelic gene expression. , 2014, Developmental cell.

[14]  N. Blüthgen,et al.  The two active X chromosomes in female ESCs block exit from the pluripotent state by modulating the ESC signaling network. , 2014, Cell stem cell.

[15]  Jesse R. Dixon,et al.  Cohesin and CTCF differentially affect chromatin architecture and gene expression in human cells , 2013, Proceedings of the National Academy of Sciences.

[16]  Howard Y. Chang,et al.  Transposition of native chromatin for fast and sensitive epigenomic profiling of open chromatin, DNA-binding proteins and nucleosome position , 2013, Nature Methods.

[17]  Bing Ren,et al.  Whole-genome haplotype reconstruction using proximity-ligation and shotgun sequencing , 2013, Nature Biotechnology.

[18]  G. Schroth,et al.  Cohesin-mediated interactions organize chromosomal domain architecture , 2013, The EMBO journal.

[19]  Job Dekker,et al.  Organization of the Mitotic Chromosome , 2013, Science.

[20]  J. L. Mateo,et al.  Epigenomic enhancer annotation reveals a key role for NFIX in neural stem cell quiescence , 2013, Genes & development.

[21]  E. Lander,et al.  The Xist lncRNA Exploits Three-Dimensional Genome Architecture to Spread Across the X Chromosome , 2013, Science.

[22]  E. Heard,et al.  Unusual chromatin status and organization of the inactive X chromosome in murine trophoblast giant cells , 2013, Development.

[23]  L. Mirny,et al.  Iterative Correction of Hi-C Data Reveals Hallmarks of Chromosome Organization , 2012, Nature Methods.

[24]  T. Magnuson,et al.  The mouse DXZ4 homolog retains Ctcf binding and proximity to Pls3 despite substantial organizational differences compared to the primate macrosatellite , 2012, Genome Biology.

[25]  J. Sedat,et al.  Spatial partitioning of the regulatory landscape of the X-inactivation centre , 2012, Nature.

[26]  Jesse R. Dixon,et al.  Topological Domains in Mammalian Genomes Identified by Analysis of Chromatin Interactions , 2012, Nature.

[27]  T. Cremer,et al.  A top-down analysis of Xa- and Xi-territories reveals differences of higher order structure at ≥ 20 Mb genomic length scales , 2011, Nucleus.

[28]  W. V. van IJcken,et al.  The inactive X chromosome adopts a unique three-dimensional conformation that is dependent on Xist RNA. , 2011, Genes & development.

[29]  N. Gilbert,et al.  Analysis of Active and Inactive X Chromosome Architecture Reveals the Independent Organization of 30 nm and Large-Scale Chromatin Structures , 2010, Molecular cell.

[30]  I. Amit,et al.  Comprehensive mapping of long range interactions reveals folding principles of the human genome , 2011 .

[31]  E. Heard,et al.  Dynamic changes in paternal X-chromosome activity during imprinted X-chromosome inactivation in mice , 2009, Proceedings of the National Academy of Sciences.

[32]  B. Chadwick,et al.  DXZ4 chromatin adopts an opposing conformation to that of the surrounding chromosome and acquires a novel inactive X-specific role involving CTCF and antisense transcripts. , 2008, Genome research.

[33]  E. Heard,et al.  Combined immunofluorescence, RNA fluorescent in situ hybridization, and DNA fluorescent in situ hybridization to study chromatin changes, transcriptional activity, nuclear organization, and X-chromosome inactivation. , 2008, Methods in molecular biology.

[34]  E. Heard,et al.  A novel role for Xist RNA in the formation of a repressive nuclear compartment into which genes are recruited when silenced. , 2006, Genes & development.

[35]  Rudolf Jaenisch,et al.  Chromosomal silencing and localization are mediated by different domains of Xist RNA , 2002, Nature Genetics.

[36]  R. Jaenisch,et al.  A shift from reversible to irreversible X inactivation is triggered during ES cell differentiation. , 2000, Molecular cell.

[37]  R Eils,et al.  Three-dimensional reconstruction of painted human interphase chromosomes: active and inactive X chromosome territories have similar volumes but differ in shape and surface structure , 1996, The Journal of cell biology.

[38]  U. Francke,et al.  A novel GC–rich human macrosatellite VNTR in Xq24 is differentially methylated on active and inactive X chromosomes , 1992, Nature Genetics.