Amyloid P-Component Expression of C-Reactive Protein and Serum Complement-Dependent Acute-Phase

[1]  H. Colten,et al.  Genetic disruption of the murine complement C3 promoter region generates deficient mice with extrahepatic expression of C3 mRNA. , 1999, Immunopharmacology.

[2]  N. Davoust,et al.  Human T cells express the C5a receptor and are chemoattracted to C5a. , 1999, Journal of immunology.

[3]  I. Kushner,et al.  Acute-phase proteins and other systemic responses to inflammation. , 1999, The New England journal of medicine.

[4]  U. Höpken,et al.  Expression of C5a receptor in mouse brain: role in signal transduction and neurodegeneration , 1999, Neuroscience.

[5]  J. Volanakis,et al.  Testosterone and IL-6 requirements for human C-reactive protein gene expression in transgenic mice. , 1998, Journal of immunology.

[6]  T. Hugli,et al.  Characterization of Complement Anaphylatoxins and Their Biological Responses , 1998 .

[7]  F. Rosen,et al.  Endotoxin shock in antibody-deficient mice: unraveling the role of natural antibody and complement in the clearance of lipopolysaccharide. , 1997, Journal of immunology.

[8]  C. Janeway,et al.  Innate immunity: impact on the adaptive immune response. , 1997, Current opinion in immunology.

[9]  J. Volanakis,et al.  Role of complement in C-reactive-protein-mediated protection of mice from Streptococcus pneumoniae , 1996, Infection and immunity.

[10]  R. Locksley,et al.  The Instructive Role of Innate Immunity in the Acquired Immune Response , 1996, Science.

[11]  J. Volanakis,et al.  Human C-reactive protein is protective against fatal Streptococcus pneumoniae infection in transgenic mice. , 1995, Journal of immunology.

[12]  Dongxiao Zhang,et al.  The effect of interleukin-1 on C-reactive protein expression in Hep3B cells is exerted at the transcriptional level. , 1995, The Biochemical journal.

[13]  T. Hugli,et al.  Expression of functional receptors for human C5a anaphylatoxin (CD88) on the human hepatocellular carcinoma cell line HepG2. Stimulation of acute-phase protein-specific mRNA and protein synthesis by human C5a anaphylatoxin. , 1995, Journal of immunology.

[14]  D. Haviland,et al.  N-formylpeptide and complement C5a receptors are expressed in liver cells and mediate hepatic acute phase gene regulation , 1995, The Journal of experimental medicine.

[15]  A. Shaw,et al.  Resistance to fever induction and impaired acute-phase response in interleukin-1 beta-deficient mice. , 1995, Immunity.

[16]  S. Dalrymple,et al.  Interleukin-6-deficient mice are highly susceptible to Listeria monocytogenes infection: correlation with inefficient neutrophilia , 1995, Infection and immunity.

[17]  W. Parks,et al.  Cellular expression of the C5a anaphylatoxin receptor (C5aR): demonstration of C5aR on nonmyeloid cells of the liver and lung. , 1995, Journal of immunology.

[18]  Y. Takakuwa,et al.  Changes in serum concentration and mRNA level of rat C-reactive protein. , 1994, Biochimica et biophysica acta.

[19]  R. Zinkernagel,et al.  Impaired immune and acute-phase responses in interleukin-6-deficient mice , 1994, Nature.

[20]  T. Yoshida,et al.  Endotoxin‐induced cytokine gene expression and excretion in the liver , 1994, Hepatology.

[21]  Y. Taktak,et al.  Solid Phase Enzyme Immunoassays for the Quantification of Serum Amyloid P (SAP) and Complement Component 3 (C3) Proteins in Acute-Phase Mouse Sera , 1992, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.

[22]  I. Kushner,et al.  Effect of combinations of cytokines and hormones on synthesis of serum amyloid A and C-reactive protein in Hep 3B cells. , 1991, Journal of immunology.

[23]  K. Yancey,et al.  C5a, cutaneous mast cells, and inflammation: in vitro and in vivo studies in a murine model. , 1991, The Journal of investigative dermatology.

[24]  H. Baumann,et al.  Interaction of cytokine- and glucocorticoid-response elements of acute-phase plasma protein genes. Importance of glucocorticoid receptor level and cell type for regulation of the elements from rat alpha 1-acid glycoprotein and beta-fibrinogen genes. , 1990, The Journal of biological chemistry.

[25]  D. Noonan,et al.  C5a-mediated release of interleukin 6 by human monocytes. , 1990, Clinical immunology and immunopathology.

[26]  G. Ciliberto,et al.  Regulation of the human C-reactive protein gene, a major marker of inflammation and cancer. , 1990, Molecular biology & medicine.

[27]  J. Gauldie,et al.  Regulation of hepatic acute phase plasma protein genes by hepatocyte stimulating factors and other mediators of inflammation. , 1990, Molecular biology & medicine.

[28]  D. Haviland,et al.  Deficiency of the murine fifth complement component (C5). A 2-base pair gene deletion in a 5'-exon. , 1990, The Journal of biological chemistry.

[29]  G. Ciliberto,et al.  Dual control of C‐reactive protein gene expression by interleukin‐1 and interleukin‐6. , 1989, The EMBO journal.

[30]  C. Dinarello,et al.  C5a stimulates secretion of tumor necrosis factor from human mononuclear cells in vitro. Comparison with secretion of interleukin 1 beta and interleukin 1 alpha , 1988, The Journal of experimental medicine.

[31]  E. Wagner,et al.  Inducible and tissue‐specific expression of human C‐reactive protein in transgenic mice. , 1987, The EMBO journal.

[32]  C. Dinarello,et al.  C5a induction of human interleukin 1. Synergistic effect with endotoxin or interferon-gamma. , 1987, Journal of immunology.

[33]  J. Kearney,et al.  Demonstration of calcium-induced conformational change(s) in C-reactive protein by using monoclonal antibodies. , 1982, Molecular immunology.

[34]  M. Muller,et al.  Acute phase reactants of mice. I. Isolation of serum amyloid P-component (SAP) and its induction by a monokine. , 1982, Journal of immunology.

[35]  A. Feinstein,et al.  BIOLOGY OF SERUM AMYLOID P COMPONENT * , 1982, Annals of the New York Academy of Sciences.

[36]  I. Kushner.  THE PHENOMENON OF THE ACUTE PHASE RESPONSE * , 1982, Annals of the New York Academy of Sciences.

[37]  I. von Zabern,et al.  Comparison of Naja n. naja and Naja h. haje cobra-venom factors: correlation between binding affinity for the fifth component of complement and mediation of its cleavage. , 1980, Immunobiology.

[38]  M. Pepys,et al.  Complement-independence of the acute-phase production of serum amyloijd P-component (SAP) in mice. , 1980, British journal of experimental pathology.

[39]  R. Stroud,et al.  Rabbit CRP Production After Inulin Injection 1 , 1979, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

[40]  J. Volanakis,et al.  C-reactive protein: purification by affinity chromatography and physicochemical characterization , 1978 .

[41]  R. Siboo,et al.  A fluorescent immunoassay for the quantification of C-reactive protein. , 1978, Journal of immunological methods.

[42]  R. Siboo,et al.  Isolation of mouse C-reactive protein from liver and serum. , 1977, Journal of immunology.

[43]  H. Gewurz,et al.  Characterization of C-reactive protein and the complement subcomponent C1t as homologous proteins displaying cyclic pentameric symmetry (pentraxins). , 1977, Proceedings of the National Academy of Sciences of the United States of America.

[44]  L. Kline,et al.  Activation of the classical and properdin pathways of complement by bacterial lipopolysaccharides (LPS). , 1977, Journal of immunology.

[45]  I. Kushner,et al.  The acute phase response: an overview. , 1988, Methods in enzymology.