Angiotensin-converting enzyme inhibition promotes coronary angiogenesis in the failing heart of Dahl salt-sensitive hypertensive rats.
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T. Murohara | K. Nagata | M. Miyachi | Hiroki Yazawa | Koji Tsuboi | Yosuke Kato | Mayuko Furukawa | Keiji Takahashi | M. Takatsu | M. Ohtake | T. Murase | T. Hattori | Takuya Hattori | Tamayo Murase | Miwa Takatsu
[1] S. Fagan,et al. Angiotensin receptor blockers and angiogenesis: clinical and experimental evidence. , 2011, Clinical science.
[2] U. Laufs,et al. ACE inhibition promotes upregulation of endothelial progenitor cells and neoangiogenesis in cardiac pressure overload. , 2009, Cardiovascular research.
[3] T. Yokoi,et al. Exercise Training Alters Left Ventricular Geometry and Attenuates Heart Failure in Dahl Salt-Sensitive Hypertensive Rats , 2009, Hypertension.
[4] Hong Wang,et al. Compensatory function of bradykinin B1 receptor in the inhibitory effect of captopril on cardiomyocyte hypertrophy and cardiac fibroblast proliferation in neonatal rats. , 2008, Chinese medical journal.
[5] K. Okumura,et al. Role of Bradykinin, Nitric Oxide, and Angiotensin II Type 2 Receptor in Imidapril-Induced Angiogenesis , 2008, Hypertension.
[6] J. Norman,et al. VEGF regulates the mobilization of VEGFR2/KDR from an intracellular endothelial storage compartment. , 2006, Blood.
[7] T. Kato,et al. Mineralocorticoid Receptor Antagonism Attenuates Cardiac Hypertrophy and Failure in Low-Aldosterone Hypertensive Rats , 2006, Hypertension.
[8] K. Nagata,et al. Pravastatin increases survival and suppresses an increase in myocardial matrix metalloproteinase activity in a rat model of heart failure. , 2006, Cardiovascular research.
[9] T. Kato,et al. Mineralocorticoid Receptor Antagonism Ameliorates Left Ventricular Diastolic Dysfunction and Myocardial Fibrosis in Mildly Symptomatic Patients With Idiopathic Dilated Cardiomyopathy: A Pilot Study , 2005, Circulation.
[10] T. Naoe,et al. Nicorandil Promotes Myocardial Capillary and Arteriolar Growth in the Failing Heart of Dahl Salt-Sensitive Hypertensive Rats , 2005, Hypertension.
[11] T. Gustafsson,et al. Antagonism of the renin-angiotensin system can counteract cardiac angiogenic vascular endothelial growth factor gene therapy and myocardial angiogenesis in the normal heart. , 2005, American journal of hypertension.
[12] I. Shiojima,et al. Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure. , 2005, The Journal of clinical investigation.
[13] I. Inuwa,et al. Long-term exercise training and angiotensin-converting enzyme inhibition differentially enhance myocardial capillarization in the spontaneously hypertensive rat , 2005, Journal of hypertension.
[14] B. Lévy,et al. Dual Effect of Angiotensin-Converting Enzyme Inhibition on Angiogenesis in Type 1 Diabetic Mice , 2004, Arteriosclerosis, thrombosis, and vascular biology.
[15] M. Hori,et al. AT1 Receptor Blocker Added to ACE Inhibitor Provides Benefits at Advanced Stage of Hypertensive Diastolic Heart Failure , 2004, Hypertension.
[16] J. Toblli,et al. Angiotensin-converting enzyme inhibition and angiogenesis in myocardium of obese Zucker rats. , 2004, American journal of hypertension.
[17] E. Olson,et al. Cardiac hypertrophy: the good, the bad, and the ugly. , 2003, Annual review of physiology.
[18] Y. Yoshikawa,et al. Beneficial effects of angiotensin-converting enzyme inhibition on sarcoplasmic reticulum function in the failing heart of the Dahl rat. , 2003, Circulation journal : official journal of the Japanese Circulation Society.
[19] S. Miura,et al. Transactivation of KDR/Flk-1 by the B2 Receptor Induces Tube Formation in Human Coronary Endothelial Cells , 2003, Hypertension.
[20] H. Izawa,et al. AT1 Receptor Blockade Reduces Cardiac Calcineurin Activity in Hypertensive Rats , 2002, Hypertension.
[21] D. Neglia,et al. Prognostic Role of Myocardial Blood Flow Impairment in Idiopathic Left Ventricular Dysfunction , 2002, Circulation.
[22] B. Lévy,et al. Proangiogenic Effect of Angiotensin-Converting Enzyme Inhibition Is Mediated by the Bradykinin B2 Receptor Pathway , 2001, Circulation research.
[23] H. Izawa,et al. Reduced Myocardial Sarcoplasmic Reticulum Ca2+-ATPase mRNA Expression and Biphasic Force-Frequency Relations in Patients With Hypertrophic Cardiomyopathy , 2001, Circulation.
[24] H. Traxler,et al. Selective Downregulation of VEGF-A165, VEGF-R1, and Decreased Capillary Density in Patients With Dilative but Not Ischemic Cardiomyopathy , 2000, Circulation research.
[25] N. Kobayashi,et al. Effects of imidapril on endothelin-1 and ACE gene expression in failing hearts of salt-sensitive hypertensive rats. , 2000, American journal of hypertension.
[26] J. Cooke,et al. Nitric oxide induces the synthesis of vascular endothelial growth factor by rat vascular smooth muscle cells. , 2000, Arteriosclerosis, thrombosis, and vascular biology.
[27] J. Cohn,et al. Cardiac remodeling--concepts and clinical implications: a consensus paper from an international forum on cardiac remodeling. Behalf of an International Forum on Cardiac Remodeling. , 2000, Journal of the American College of Cardiology.
[28] D. Levy,et al. Congestive heart failure in subjects with normal versus reduced left ventricular ejection fraction: prevalence and mortality in a population-based cohort. , 1999, Journal of the American College of Cardiology.
[29] P. Huang,et al. Nitric oxide synthase modulates angiogenesis in response to tissue ischemia. , 1998, The Journal of clinical investigation.
[30] C. H. Conrad,et al. Effect of angiotensin-converting enzyme inhibition on myocardial fibrosis and function in hypertrophied and failing myocardium from the spontaneously hypertensive rat. , 1997, Circulation.
[31] C. Garlanda,et al. Involvement of endothelial PECAM-1/CD31 in angiogenesis. , 1997, The American journal of pathology.
[32] R. Busse,et al. ACE inhibitor potentiation of bradykinin‐induced venoconstriction , 1997, British journal of pharmacology.
[33] G. B. John Mancini,et al. Angiotensin Converting Enzyme Inhibition, with Quinapril, Improves Endothelial Vasomotor Dysfunction in Patients with Coronary Artery Disease: the TREND Study (Trial on REversing ENdothelial Dysfunction) , 1996 .
[34] S Sasayama,et al. Transition from compensatory hypertrophy to dilated, failing left ventricles in Dahl salt-sensitive rats. , 1994, The American journal of physiology.
[35] D. Campbell,et al. Effects of converting enzyme inhibitors on angiotensin and bradykinin peptides. , 1994, Hypertension.
[36] D. Levy,et al. Prognostic implications of echocardiographically determined left ventricular mass in the Framingham Heart Study. , 1990, The New England journal of medicine.
[37] R. Tomanek. Response of the coronary vasculature to myocardial hypertrophy. , 1990, Journal of the American College of Cardiology.
[38] L. Ignarro,et al. Biological Actions and Properties of Endothelium-Derived Nitric Oxide Formed and Released From Artery and Vein , 1989, Circulation research.
[39] I. Komuro,et al. Gene Expression in Fibroblasts and Fibrosis: Involvement in Cardiac Hypertrophy Roles of Cardiac Transcription Factors in Cardiac Hypertrophy Ras, Akt, and Mechanotransduction in the Cardiac Myocyte G Protein–Coupled Signaling and Gene Expression Genetic Models and Mechanisms of Transcription in Car , 2003 .