COMBUSTION‐DERIVED NANOPARTICLES: MECHANISMS OF PULMONARY TOXICITY
暂无分享,去创建一个
Dominique Balharry | Lata Koshy | K. BéruBé | T. Jones | K. Sexton | Keith Sexton | D. Balharry | Kelly BéruBé | Tim Jones | Lata Koshy
[1] Jenny R. Roberts,et al. Metal composition and solubility determine lung toxicity induced by residual oil fly ash collected from different sites within a power plant , 2004, Molecular and Cellular Biochemistry.
[2] Ben J. Williamson,et al. Physicochemical characterisation of diesel exhaust particles: Factors for assessing biological activity , 1999 .
[3] David M. Brown,et al. Calcium and ROS-mediated activation of transcription factors and TNF-alpha cytokine gene expression in macrophages exposed to ultrafine particles. , 2003, American journal of physiology. Lung cellular and molecular physiology.
[4] J. A. Bond,et al. Pulmonary toxicity of inhaled diesel exhaust and carbon black in chronically exposed rats. Part I: Neoplastic and nonneoplastic lung lesions. , 1994, Research report.
[5] K. BéruBé,et al. Mineralogy and structure of pathogenic particles , 2007 .
[6] Exposure-route-dependent DNA adduct formation by polycyclic aromatic hydrocarbons. , 2000, Carcinogenesis.
[7] C. Rossiter,et al. Respiratory health effects of carbon black: a survey of European carbon black workers. , 1993, British journal of industrial medicine.
[8] M F Hoylaerts,et al. Passage of intratracheally instilled ultrafine particles from the lung into the systemic circulation in hamster. , 2001, American journal of respiratory and critical care medicine.
[9] M. Dennekamp,et al. Hypothesis: Ill health associated with low concentrations of nitrogen dioxide—an effect of ultrafine particles? , 2003, Thorax.
[10] E. Tagaya,et al. Ultrafine carbon black particles stimulate proliferation of human airway epithelium via EGF receptor-mediated signaling pathway. , 2004, American journal of physiology. Lung cellular and molecular physiology.
[11] J L Mauderly,et al. Comparative Pulmonary Toxicities and Carcinogenicities of Chronically Inhaled Diesel Exhaust and Carban Black in F344 Rats , 1995, Fundamental and applied toxicology : official journal of the Society of Toxicology.
[12] W. MacNee,et al. Activation of NF-κB by PM10 Occurs via an Iron-Mediated Mechanism in the Absence of IκB Degradation , 2000 .
[13] W. Burch,et al. Passage of inhaled particles into the blood circulation in humans. , 2002, Circulation.
[14] W. Linak,et al. Differential Pulmonary Inflammation and In Vitro Cytotoxicity of Size-Fractionated Fly Ash Particles from Pulverized Coal Combustion , 2004, Journal of the Air and Waste Management Association.
[15] G. Oberdörster,et al. Nanotoxicology: An Emerging Discipline Evolving from Studies of Ultrafine Particles , 2005, Environmental health perspectives.
[16] G. Hageman,et al. Genotoxicity of coal fly ash, assessed in vitro in Salmonella typhimurium and human lymphocytes, and in vivo in an occupationally exposed population. , 1989, Mutation research.
[17] J. Carson,et al. Biologic effects of oil fly ash. , 2002, Environmental health perspectives.
[18] W. MacNee,et al. Combustion-derived nanoparticles: A review of their toxicology following inhalation exposure , 2005, Particle and Fibre Toxicology.
[19] K. Müller,et al. Exposure to carbon black: a cancer risk? , 1998, International archives of occupational and environmental health.
[20] W. Pryor,et al. Quinoid redox cycling as a mechanism for sustained free radical generation by inhaled airborne particulate matter. , 2001, Free radical biology & medicine.
[21] K Gardiner,et al. Effects on respiratory morbidity of occupational exposure to carbon black: a review. , 1995, Archives of environmental health.
[22] L. Möller,et al. Early oxidative DNA damages and late development of lung cancer in diesel exhaust-exposed rats. , 2000, Environmental research.
[23] D. Costa,et al. Bioavailable transition metals in particulate matter mediate cardiopulmonary injury in healthy and compromised animal models. , 1997, Environmental health perspectives.
[24] O. Creutzenberg,et al. The carcinogenic potency of carbon particles with and without PAH after repeated intratracheal administration in the rat. , 1996, Toxicology letters.
[25] Jenny R. Roberts,et al. Role of metal-induced reactive oxygen species generation in lung responses caused by residual oil fly ash , 2003, Journal of Biosciences.
[26] T. Yoshikawa,et al. Effects of nano particles on antigen-related airway inflammation in mice , 2005, Respiratory research.
[27] A. Ledbetter,et al. Emission-particle-induced ventilatory abnormalities in a rat model of pulmonary hypertension. , 2004, Environmental health perspectives.
[28] T. Moreno,et al. Characterisation of airborne particulate matter and related mechanisms of toxicity: an experimental approach , 2006 .
[29] R. Elton,et al. Particulate air pollution and the blood , 1999, Thorax.
[30] N. Jansen,et al. EXOGENOUS SURFACTANT RESTORES LUNG FUNCTION BUT NOT PERIPHERAL IMMUNOSUPPRESSION IN VENTILATED SURFACTANT-DEFICIENT RATS , 2006, Experimental lung research.
[31] P. Borm,et al. In vitro effects of coal fly ashes: hydroxyl radical generation, iron release, and DNA damage and toxicity in rat lung epithelial cells. , 1999, Inhalation toxicology.
[32] Benoit Nemery,et al. Possible mechanisms of the cardiovascular effects of inhaled particles: systemic translocation and prothrombotic effects. , 2003, Toxicology letters.
[33] K. BéruBé,et al. INFLAMMATION, EDEMA, AND PERIPHERAL BLOOD CHANGES IN LUNG-COMPROMISED RATS AFTER INSTILLATION WITH COMBUSTION-DERIVED AND MANUFACTURED NANOPARTICLES , 2006, Experimental lung research.
[34] H. Chung,et al. Changes of 8-OH-dG levels in DNA and its base excision repair activity in rat lungs after inhalation exposure to hexavalent chromium. , 2003, Mutation research.