Salidroside alleviates cognitive impairment by inhibiting ferroptosis via activation of the Nrf2/GPX4 axis in SAMP8 mice.
暂无分享,去创建一个
Jiaqi Li | Y. Zeng | Zeping Xie | Wenxu Li | Weidong Cheng | Sixia Yang | Linshuang Wang | Qiaowu Xiong | Tingting Pei | Dongfeng Wei | Yong Wang | Huijuan Wei | Qian Su
[1] Ruihan Yang,et al. Salidroside reduces neuropathology in Alzheimer’s disease models by targeting NRF2/SIRT3 pathway , 2022, Cell & Bioscience.
[2] Yingwei Chen,et al. Salidroside alleviates ulcerative colitis via inhibiting macrophage pyroptosis and repairing the dysbacteriosis‐associated Th17/Treg imbalance , 2022, Phytotherapy research : PTR.
[3] S. Merighi,et al. Microglia and Alzheimer’s Disease , 2022, International journal of molecular sciences.
[4] Lei Zhou,et al. Regulating Nrf2-GPx4 axis by bicyclol can prevent ferroptosis in carbon tetrachloride-induced acute liver injury in mice , 2022, Cell Death Discovery.
[5] Y. Zeng,et al. Salidroside attenuates neuronal ferroptosis by activating the Nrf2/HO1 signaling pathway in Aβ1-42-induced Alzheimer’s disease mice and glutamate-injured HT22 cells , 2022, Chinese Medicine.
[6] Hui-Zhi Long,et al. The Role of Microglia in Alzheimer’s Disease From the Perspective of Immune Inflammation and Iron Metabolism , 2022, Frontiers in Aging Neuroscience.
[7] Han Guo,et al. Forsythoside A Mitigates Alzheimer's-like Pathology by Inhibiting Ferroptosis-mediated Neuroinflammation via Nrf2/GPX4 Axis Activation , 2022, International journal of biological sciences.
[8] W. Zou,et al. CD8+ T cells and fatty acids orchestrate tumor ferroptosis and immunity via ACSL4. , 2022, Cancer cell.
[9] Y. Liu,et al. Salidroside inhibits doxorubicin-induced cardiomyopathy by modulating a ferroptosis-dependent pathway. , 2022, Phytomedicine : international journal of phytotherapy and phytopharmacology.
[10] B. Platt,et al. Sex Differences in Behavior and Molecular Pathology in the 5XFAD Model. , 2021, Journal of Alzheimer's disease : JAD.
[11] N. Chattipakorn,et al. Cell death inhibitors protect against brain damage caused by cardiac ischemia/reperfusion injury , 2021, Cell death discovery.
[12] A. Bush,et al. Ferroptosis and NRF2: an emerging battlefield in the neurodegeneration of Alzheimer's disease. , 2021, Essays in biochemistry.
[13] A. Alexiou,et al. Neuroinflammatory Signaling in the Pathogenesis of Alzheimer’s Disease , 2021, Current neuropharmacology.
[14] Yusheng Cong,et al. Neuroprotective effects of salidroside on ageing hippocampal neurons and naturally ageing mice via the PI3K/Akt/TERT pathway , 2021, Phytotherapy research : PTR.
[15] B. Železná,et al. Aging and high-fat diet feeding lead to peripheral insulin resistance and sex-dependent changes in brain of mouse model of tau pathology THY-Tau22 , 2021, Journal of Neuroinflammation.
[16] Lan Zhang,et al. Tetrahydroxy Stilbene Glucoside Ameliorates Cognitive Impairments and Pathology in APP/PS1 Transgenic Mice , 2021, Current Medical Science.
[17] Yu Zheng,et al. Salidroside: A review of its recent advances in synthetic pathways and pharmacological properties. , 2021, Chemico-biological interactions.
[18] Fengna Chu,et al. Role of Adaptive Immune and Impacts of Risk Factors on Adaptive Immune in Alzheimer’s Disease: Are Immunotherapies Effective or Off-Target? , 2021, The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry.
[19] R. Velazquez,et al. Sex differences in the IntelliCage and the Morris water maze in the APP/PS1 mouse model of amyloidosis , 2021, Neurobiology of Aging.
[20] D. Tang,et al. Characteristics and Biomarkers of Ferroptosis , 2021, Frontiers in Cell and Developmental Biology.
[21] Shih-Ying Wu,et al. Nicotine promotes breast cancer metastasis by stimulating N2 neutrophils and generating pre-metastatic niche in lung , 2021, Nature Communications.
[22] P. Nelson,et al. Loss of ferroportin induces memory impairment by promoting ferroptosis in Alzheimer’s disease , 2021, Cell Death & Differentiation.
[23] Ting Zeng,et al. Salidroside Attenuates Cognitive Dysfunction in Senescence-Accelerated Mouse Prone 8 (SAMP8) Mice and Modulates Inflammation of the Gut-Brain Axis , 2020, Frontiers in Pharmacology.
[24] O. Nicole,et al. CD8+ T Cell-Mediated Mechanisms Contribute to the Progression of Neurocognitive Impairment in Both Multiple Sclerosis and Alzheimer's Disease? , 2020, Frontiers in Immunology.
[25] A. Bush,et al. Iron and Ferroptosis as Therapeutic Targets in Alzheimer’s Disease , 2020, Neurotherapeutics.
[26] N. S. Yarla,et al. Salidroside - Can it be a multifunctional drug? , 2020, Current drug metabolism.
[27] J. Attems,et al. CD8+ T-cells infiltrate Alzheimer’s disease brains and regulate neuronal- and synapse-related gene expression in APP-PS1 transgenic mice , 2020, Brain, Behavior, and Immunity.
[28] Jie Liu,et al. SAMP8 Mice as a Model of Age-Related Cognition Decline with Underlying Mechanisms in Alzheimer's Disease. , 2020, Journal of Alzheimer's disease : JAD.
[29] Mark M. Davis,et al. Clonally expanded CD8 T cells patrol the cerebrospinal fluid in Alzheimer’s disease , 2020, Nature.
[30] M. Heneka. An immune-cell signature marks the brain in Alzheimer’s disease , 2020, Nature.
[31] David R. Brown,et al. Microglia and the aging brain: are senescent microglia the key to neurodegeneration? , 2019, Journal of neurochemistry.
[32] Nicola Thrupp,et al. The Major Risk Factors for Alzheimer’s Disease: Age, Sex, and Genes Modulate the Microglia Response to Aβ Plaques , 2019, Cell reports.
[33] A. Chinnaiyan,et al. CD8+ T cells regulate tumor ferroptosis during cancer immunotherapy , 2019, Nature.
[34] Tao Wang,et al. Iron Pathophysiology in Alzheimer's Diseases. , 2019, Advances in experimental medicine and biology.
[35] S. Hébert,et al. Peripheral adaptive immunity of the triple transgenic mouse model of Alzheimer’s disease , 2019, Journal of Neuroinflammation.
[36] Lidian Chen,et al. Pharmacological activities, mechanisms of action, and safety of salidroside in the central nervous system , 2018, Drug design, development and therapy.
[37] Feng Yan,et al. Salidroside provides neuroprotection by modulating microglial polarization after cerebral ischemia , 2018, Journal of Neuroinflammation.
[38] R. Nitsch,et al. Extravascular CD3+ T Cells in Brains of Alzheimer Disease Patients Correlate with Tau but Not with Amyloid Pathology: An Immunohistochemical Study , 2018, Neurodegenerative Diseases.
[39] Zhan-You Wang,et al. α-Lipoic acid improves abnormal behavior by mitigation of oxidative stress, inflammation, ferroptosis, and tauopathy in P301S Tau transgenic mice , 2017, Redox biology.
[40] I. Amit,et al. A Unique Microglia Type Associated with Restricting Development of Alzheimer’s Disease , 2017, Cell.
[41] Yun-Sik Choi,et al. Effect of rotation preference on spontaneous alternation behavior on Y maze and introduction of a new analytical method, entropy of spontaneous alternation , 2017, Behavioural Brain Research.
[42] L. Buée,et al. Hippocampal T cell infiltration promotes neuroinflammation and cognitive decline in a mouse model of tauopathy , 2016, Brain : a journal of neurology.
[43] Erin L. Abner,et al. A Comprehensive Behavioral Test Battery to Assess Learning and Memory in 129S6/Tg2576 Mice , 2016, PloS one.
[44] Na Wei,et al. Delineating the relationships among the formation of reactive oxygen species, cell membrane instability and innate autoimmunity in intestinal reperfusion injury. , 2014, Molecular immunology.
[45] Xiaorui Cheng,et al. The behavioral, pathological and therapeutic features of the senescence-accelerated mouse prone 8 strain as an Alzheimer's disease animal model , 2014, Ageing Research Reviews.
[46] D. Selkoe. Alzheimer's disease. , 2011, Cold Spring Harbor perspectives in biology.
[47] Han-Xiong Li,et al. Production of Th1- and Th2-dependent cytokines induced by the Chinese medicine herb, Rhodiola algida, on human peripheral blood monocytes. , 2009, Journal of ethnopharmacology.
[48] C. Belzung,et al. The open field as a paradigm to measure the effects of drugs on anxiety-like behaviors: a review. , 2003, European journal of pharmacology.
[49] C. Carpi,et al. [In vivo anihistidine-decarboxylasic activity of 2-methyl-6,7-methylendioxy-8-methoxy-1-(4', 5', 6',-triothoxy-7-aminophthalidyl)-1,2,3,4 tetrahydroisoquinoline (tritoqualine)]. , 1968, Bollettino della Societa italiana di biologia sperimentale.