Simvastatin inhibits Staphylococcus aureus alpha toxin mediated polymorphonuclear leukocyte-induced vasocontraction and endothelial dysfunction
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W. Seeger | F. Grimminger | K. Werdan | M. Buerke | U. Sibelius | U. Grandel | R. Prondzinsky | U. Buerke | W. Seeger | M. Russ | H. Schmidt | K. Werdan | R. Prondzinsky | U. Buerke | H. Schmidt | U. Grandel
[1] W. Seeger,et al. Simvastatin Inhibits Inflammatory Properties of Staphylococcus aureus &agr;-Toxin , 2002, Circulation.
[2] W. Seeger,et al. Staphylococcus Aureus Alpha Toxin Mediates Polymorphonuclear Leukocyte-Induced Vasocontraction and Endothelial Dysfunction , 2002, Shock.
[3] A. M. Lefer,et al. A new HMG‐CoA reductase inhibitor, rosuvastatin, exerts anti‐inflammatory effects on the microvascular endothelium: the role of mevalonic acid , 2001, British journal of pharmacology.
[4] W. Seeger,et al. Staphylococcal alpha-toxin provokes coronary vasoconstriction and loss in myocardial contractility in perfused rat hearts: role of thromboxane generation. , 2000, Circulation.
[5] A. M. Lefer,et al. Simvastatin inhibits leukocyte-endothelial cell interactions and protects against inflammatory processes in normocholesterolemic rats. , 1999, Arteriosclerosis, thrombosis, and vascular biology.
[6] A. M. Lefer,et al. Simvastatin preserves the ischemic-reperfused myocardium in normocholesterolemic rat hearts. , 1999, Circulation.
[7] M. Moskowitz,et al. Stroke protection by 3-hydroxy-3-methylglutaryl (HMG)-CoA reductase inhibitors mediated by endothelial nitric oxide synthase. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[8] U. Laufs,et al. Upregulation of endothelial nitric oxide synthase by HMG CoA reductase inhibitors. , 1998, Circulation.
[9] A. Cheung,et al. Hyperproduction of alpha-toxin by Staphylococcus aureus results in paradoxically reduced virulence in experimental endocarditis: a host defense role for platelet microbicidal proteins , 1997, Infection and immunity.
[10] W. Seeger,et al. Human endothelial cell activation and mediator release in response to the bacterial exotoxins Escherichia coli hemolysin and staphylococcal alpha-toxin. , 1997, Journal of immunology.
[11] N. Suttorp,et al. Escherichia coli hemolysin and Staphylococcus aureas alpha-toxin potently induce neutrophil adhesion to cultured human endothelial cells. , 1996, Journal of immunology.
[12] A. M. Lefer,et al. Lysophosphatidylcholine promotes P-selectin expression in platelets and endothelial cells. Possible involvement of protein kinase C activation and its inhibition by nitric oxide donors. , 1996, Circulation research.
[13] A. M. Lefer,et al. Polymorphonuclear leukocyte-induced vasocontraction and endothelial dysfunction. Role of selectins. , 1994, Arteriosclerosis and thrombosis : a journal of vascular biology.
[14] N. Suttorp,et al. Stimulation of PAF-synthesis in pulmonary artery endothelial cells by Staphylococcus aureus alpha-toxin. , 1992, Thrombosis research.
[15] S Bhakdi,et al. Alpha-toxin of Staphylococcus aureus. , 1991, Microbiological reviews.
[16] G. Zimmerman,et al. Coexpression of GMP-140 and PAF by endothelium stimulated by histamine or thrombin: a juxtacrine system for adhesion and activation of neutrophils , 1991, The Journal of cell biology.
[17] R. McEver. Leukocyte Interactions Mediated by Selectins , 1991, Thrombosis and Haemostasis.
[18] A. M. Lefer,et al. Neutrophil-mediated vasoconstriction and endothelial dysfunction in low-flow perfusion-reperfused cat coronary artery. , 1991, Circulation research.
[19] P. Kubes,et al. Nitric oxide: an endogenous modulator of leukocyte adhesion. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[20] B. Fleming,et al. A method for isolation and fluorescent labeling of rat neutrophils for intravital microvascular studies. , 1990, Microvascular research.
[21] J. Goldstein,et al. Regulation of the mevalonate pathway , 1990, Nature.
[22] E. Ohlstein,et al. Rabbit Polymorphonuclear Neutrophils Elicit Endothelium-Dependent Contraction in Vascular Smooth Muscle , 1989, Circulation research.
[23] S. Weiss. Tissue destruction by neutrophils. , 1989, The New England journal of medicine.
[24] S. Bhakdi,et al. Damage to mammalian cells by proteins that form transmembrane pores. , 1987, Reviews of physiology, biochemistry and pharmacology.
[25] P. Vanhoutte,et al. Oxygen-derived free radicals, endothelium, and responsiveness of vascular smooth muscle. , 1986, The American journal of physiology.
[26] R. Paul,et al. Eicosonoid metabolism and beta-adrenergic mechanisms in coronary arterial smooth muscle: potential compartmentation of cAMP. , 1986, The American journal of physiology.
[27] R. Furchgott. Role of endothelium in responses of vascular smooth muscle. , 1983, Circulation research.
[28] P. Ward,et al. Role of oxygen-derived free radicals and metabolites in leukocyte-dependent inflammatory reactions. , 1982, The American journal of pathology.
[29] A. Bøyum,et al. Separation of leukocytes from blood and bone marrow. Introduction. , 1968 .
[30] A. Bøyum,et al. Isolation of mononuclear cells and granulocytes from human blood. , 1968 .
[31] C. Mogensen. The glomerular permeability determined by dextran clearance using Sephadex gel filtration. , 1968, Scandinavian journal of clinical and laboratory investigation.