Caffeic acid phenethyl ester (CAPE) possesses pro-hypoxia and anti-stress activities: bioinformatics and experimental evidences
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S. Kaul | R. Wadhwa | Y. Ishida | K. Terao | D. Sundar | Anjani Kumari | J. Putri | Priyanshu Bhargava
[1] S. Matsugo,et al. 3,4‐dihydroxybenzalacetone and caffeic acid phenethyl ester induce preconditioning ER stress and autophagy in SH‐SY5Y cells , 2018, Journal of cellular physiology.
[2] Y. Padwad,et al. HIF-1 in cancer therapy: two decade long story of a transcription factor , 2017, Acta oncologica.
[3] K. Pantel,et al. Circulating and disseminated tumour cells — mechanisms of immune surveillance and escape , 2017, Nature Reviews Clinical Oncology.
[4] H. Jane Dyson,et al. Hypersensitive termination of the hypoxic response by a disordered protein switch , 2017, Nature.
[5] S. Z. Abdel‐Rahman,et al. Caffeic Acid Phenethyl Ester: A Review of Its Antioxidant Activity, Protective Effects against Ischemia-reperfusion Injury and Drug Adverse Reactions , 2016, Critical reviews in food science and nutrition.
[6] S. Kaul,et al. Molecular Characterization and Enhancement of Anticancer Activity of Caffeic Acid Phenethyl Ester by γ Cyclodextrin , 2016, Journal of Cancer.
[7] Ashley I Bush,et al. Iron neurochemistry in Alzheimer's disease and Parkinson's disease: targets for therapeutics , 2016, Journal of neurochemistry.
[8] Michael H. Schwartz,et al. Reversible, Specific, Active Aggregates of Endogenous Proteins Assemble upon Heat Stress , 2015, Cell.
[9] Wei Li,et al. HIF-1α pathway: role, regulation and intervention for cancer therapy , 2015, Acta pharmaceutica Sinica. B.
[10] Won‐Kyo Jung,et al. Caffeic acid phenethyl ester reduces the secretion of vascular endothelial growth factor through the inhibition of the ROS, PI3K and HIF-1α signaling pathways in human retinal pigment epithelial cells under hypoxic conditions. , 2015, International journal of molecular medicine.
[11] G. Baldwin,et al. HIF1α expression under normoxia in prostate cancer--which pathways to target? , 2015, The Journal of urology.
[12] G. Murtaza,et al. Caffeic Acid Phenethyl Ester and Therapeutic Potentials , 2014, BioMed research international.
[13] Susan Lindquist,et al. Mechanisms of protein-folding diseases at a glance , 2014, Disease Models & Mechanisms.
[14] S. Z. Abdel‐Rahman,et al. Caffeic acid phenethyl ester, a promising component of propolis with a plethora of biological activities: A review on its anti‐inflammatory, neuroprotective, hepatoprotective, and cardioprotective effects , 2013, IUBMB life.
[15] Woody Sherman,et al. Protein and ligand preparation: parameters, protocols, and influence on virtual screening enrichments , 2013, Journal of Computer-Aided Molecular Design.
[16] H. Car,et al. Propolis changes the anticancer activity of temozolomide in U87MG human glioblastoma cell line , 2013, BMC Complementary and Alternative Medicine.
[17] Clara Navarrete,et al. Arsenite interferes with protein folding and triggers formation of protein aggregates in yeast , 2012, Journal of Cell Science.
[18] Honglian Shi,et al. Hypoxia inducible factor-1 as a target for neurodegenerative diseases. , 2011, Current medicinal chemistry.
[19] A. Vollmar,et al. Caffeic acid phenethyl ester inhibits PDGF-induced proliferation of vascular smooth muscle cells via activation of p38 MAPK, HIF-1α, and heme oxygenase-1. , 2011, Journal of natural products.
[20] Daniel Gallichan,et al. Neuroprotection by Dimethyloxalylglycine following Permanent and Transient Focal Cerebral Ischemia in Rats , 2011, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[21] Bernd Bukau,et al. Cellular strategies for controlling protein aggregation , 2010, Nature Reviews Molecular Cell Biology.
[22] Yunjin Jung,et al. Caffeic acid phenethyl ester is a potent inhibitor of HIF prolyl hydroxylase: structural analysis and pharmacological implication. , 2010, The Journal of nutritional biochemistry.
[23] Giovanni Coppola,et al. HIF prolyl hydroxylase inhibitors prevent neuronal death induced by mitochondrial toxins: therapeutic implications for Huntington's disease and Alzheimer's disease. , 2010, Antioxidants & redox signaling.
[24] R. Moreno-Sánchez,et al. HIF-1alpha modulates energy metabolism in cancer cells by inducing over-expression of specific glycolytic isoforms. , 2009, Mini reviews in medicinal chemistry.
[25] U. Dirnagl,et al. Preconditioning and tolerance against cerebral ischaemia: from experimental strategies to clinical use , 2009, The Lancet Neurology.
[26] Shanta M. Messerli,et al. CAPE (caffeic acid phenethyl ester)‐based propolis extract (Bio 30) suppresses the growth of human neurofibromatosis (NF) tumor xenografts in mice , 2009, Phytotherapy research : PTR.
[27] W. Kaelin. The von Hippel–Lindau tumour suppressor protein: O2 sensing and cancer , 2008, Nature Reviews Cancer.
[28] P. Walter,et al. Signal integration in the endoplasmic reticulum unfolded protein response , 2007, Nature Reviews Molecular Cell Biology.
[29] L. Peso,et al. Hypoxia-inducible factors and cancer , 2007, Clinical & translational oncology : official publication of the Federation of Spanish Oncology Societies and of the National Cancer Institute of Mexico.
[30] M. Simon,et al. Hypoxia-inducible factors: central regulators of the tumor phenotype. , 2007, Current opinion in genetics & development.
[31] Federico D. Sacerdoti,et al. Scalable Algorithms for Molecular Dynamics Simulations on Commodity Clusters , 2006, ACM/IEEE SC 2006 Conference (SC'06).
[32] Matthew P. Repasky,et al. Extra precision glide: docking and scoring incorporating a model of hydrophobic enclosure for protein-ligand complexes. , 2006, Journal of medicinal chemistry.
[33] J. LaManna,et al. Hypoxia-inducible Factor Prolyl 4-Hydroxylase Inhibition , 2005, Journal of Biological Chemistry.
[34] D. Holtzman,et al. Caffeic acid phenethyl ester prevents neonatal hypoxic-ischaemic brain injury. , 2004, Brain : a journal of neurology.
[35] Peter Vaupel,et al. The role of hypoxia-induced factors in tumor progression. , 2004, The oncologist.
[36] M. Simon,et al. Regulation of Transcription and Translation by Hypoxia , 2004, Cancer biology & therapy.
[37] Kyu-Won Kim,et al. Hypoxia-inducible factor (HIF-1)α: its protein stability and biological functions , 2004, Experimental & Molecular Medicine.
[38] A. Goldberg,et al. Protein degradation and protection against misfolded or damaged proteins , 2003, Nature.
[39] C. Dobson. Protein folding and misfolding , 2003, Nature.
[40] A. Mackinnon,et al. Metal-protein attenuation with iodochlorhydroxyquin (clioquinol) targeting Abeta amyloid deposition and toxicity in Alzheimer disease: a pilot phase 2 clinical trial. , 2003, Archives of neurology.
[41] B. Kurganov,et al. Kinetics of heat- and acidification-induced aggregation of firefly luciferase. , 2003, Biophysical chemistry.
[42] Kyu-Won Kim,et al. Hypoxia-induced angiogenesis during carcinogenesis. , 2003, Journal of biochemistry and molecular biology.
[43] J. Elkins,et al. Structure of Factor-inhibiting Hypoxia-inducible Factor (HIF) Reveals Mechanism of Oxidative Modification of HIF-1α* , 2003, The Journal of Biological Chemistry.
[44] J. Deisenhofer,et al. Structure of factor-inhibiting hypoxia-inducible factor 1: An asparaginyl hydroxylase involved in the hypoxic response pathway , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[45] P. Maffia,et al. Phytochemical compounds involved in the anti-inflammatory effect of propolis extract. , 2002, Fitoterapia.
[46] Dena Leshkowitz,et al. Identification of a Novel Hypoxia-Inducible Factor 1-Responsive Gene, RTP801, Involved in Apoptosis , 2002, Molecular and Cellular Biology.
[47] D. Peet,et al. Asparagine Hydroxylation of the HIF Transactivation Domain: A Hypoxic Switch , 2002, Science.
[48] L. Huang,et al. Regulation of hypoxia-inducible factor 1α is mediated by an O2-dependent degradation domain via the ubiquitin-proteasome pathway , 1998 .
[49] G. Semenza,et al. Structural and functional analysis of hypoxia-inducible factor 1. , 1997, Kidney international.
[50] F. Lombardo,et al. Experimental and computational approaches to estimate solubility and permeability in drug discovery and development settings , 1997 .
[51] D. McLachlan,et al. Intramuscular desferrioxamine in patients with Alzheimer's disease , 1991, The Lancet.
[52] M. Morange,et al. Protein denaturation during heat shock and related stress. Escherichia coli beta-galactosidase and Photinus pyralis luciferase inactivation in mouse cells. , 1989, The Journal of biological chemistry.
[53] C. Moraes,et al. Mitochondrial alterations during carcinogenesis: a review of metabolic transformation and targets for anticancer treatments. , 2013, Advances in cancer research.
[54] A. Uzun,et al. Inhibitory effect of caffeic acid phenethyl ester on bleomycine-induced lung fibrosis in rats. , 2004, Clinica chimica acta; international journal of clinical chemistry.
[55] M. Paintz,et al. [On the antimicrobial activity of propolis and propolis constituents (author's transl)]. , 1979, Die Pharmazie.