Macrophage HIV-1 infection in duodenal tissue of patients on long term HAART.

[1]  M. Dikal,et al.  Role of CD64, p53 and p21 proteins in the pathogenesis of the tubulo-interstitial syndrome in Masugi chronic nephritis , 2007, Bulletin of Experimental Biology and Medicine.

[2]  P. Cahn,et al.  The intestinal mucosa as a reservoir of HIV-1 infection after successful HAART , 2007, AIDS.

[3]  Christine Hogan,et al.  Primary HIV-1 Infection Is Associated with Preferential Depletion of CD4+ T Lymphocytes from Effector Sites in the Gastrointestinal Tract , 2004, The Journal of experimental medicine.

[4]  G. Shaw,et al.  Macrophage HIV‐1 infection and the gastrointestinal tract reservoir , 2003, Journal of leukocyte biology.

[5]  J. Flamm,et al.  Severe CD4+ T-Cell Depletion in Gut Lymphoid Tissue during Primary Human Immunodeficiency Virus Type 1 Infection and Substantial Delay in Restoration following Highly Active Antiretroviral Therapy , 2003, Journal of Virology.

[6]  G. Shaw,et al.  Lamina propria lymphocytes, not macrophages, express CCR5 and CXCR4 and are the likely target cell for human immunodeficiency virus type 1 in the intestinal mucosa. , 2000, The Journal of infectious diseases.

[7]  G. Shaw,et al.  Biological parameters of HIV‐1 infection in primary intestinal lymphocytes and macrophages , 2000, Journal of leukocyte biology.

[8]  J. Mascola,et al.  Development of Calibrated Viral Load Standards for Group M Subtypes of Human Immunodeficiency Virus Type 1 and Performance of an Improved AMPLICOR HIV-1 MONITOR Test with Isolates of Diverse Subtypes , 1999, Journal of Clinical Microbiology.

[9]  R P Johnson,et al.  Gastrointestinal tract as a major site of CD4+ T cell depletion and viral replication in SIV infection. , 1998, Science.

[10]  G. Shaw,et al.  Infection of gastrointestinal tract macrophages by HIV‐1 , 1997, Journal of leukocyte biology.

[11]  A. Bertoli,et al.  Clinical implications of HIV dynamics and drug resistance in macrophages. , 1998, Journal of biological regulators and homeostatic agents.