The association of the TEL-AML1 chromosomal translocation with the accumulation of methotrexate polyglutamates in lymphoblasts and with ploidy in childhood B-progenitor cell acute lymphoblastic leukemia: a Pediatric Oncology Group study
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A. Look | B. Camitta | L. Cooley | J. Shuster | D. Pullen | M. Vuchich | S. Lauer | D. Mahoney | V. Whitehead | M. Bernstein | C. Payment
[1] C. Pui,et al. Outcome of treatment in children with Philadelphia chromosome-positive acute lymphoblastic leukemia. , 2000, The New England journal of medicine.
[2] J. Shuster,et al. Intensification with intermediate-dose intravenous methotrexate is effective therapy for children with lower-risk B-precursor acute lymphoblastic leukemia: A Pediatric Oncology Group study. , 2000, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[3] S. Raimondi,et al. Reduced folate carrier expression in acute lymphoblastic leukemia: a mechanism for ploidy but not lineage differences in methotrexate accumulation. , 1999, Blood.
[4] A. Look,et al. Accumulation of methotrexate polyglutamates, ploidy and trisomies of both chromosomes 4 and 10 in lymphoblasts from children with B-progenitor cell acute lymphoblastic leukemia: a Pediatric Oncology Group Study. , 1998, Leukemia & lymphoma.
[5] Y. Ravindranath,et al. Reduced folate carrier gene expression in childhood acute lymphoblastic leukemia: relationship to immunophenotype and ploidy. , 1998, Clinical cancer research : an official journal of the American Association for Cancer Research.
[6] M. Rhee,et al. Characterization of human cellular gamma-glutamyl hydrolase. , 1998, Molecular pharmacology.
[7] A. Borkhardt,et al. Incidence of TEL/AML1 fusion gene analyzed consecutively in children with acute lymphoblastic leukemia in relapse. , 1997, Blood.
[8] J. Bertino,et al. gamma-Glutamyl hydrolase and folylpolyglutamate synthetase activities predict polyglutamylation of methotrexate in acute leukemias. , 1997, Oncology research.
[9] Pui,et al. Childhood Acute Lymphoblastic Leukemia. , 1997, The oncologist.
[10] M. Relling,et al. Differences in folylpolyglutamate synthetase and dihydrofolate reductase expression in human B-lineage versus T-lineage leukemic lymphoblasts: mechanisms for lineage differences in methotrexate polyglutamylation and cytotoxicity. , 1997, Molecular pharmacology.
[11] J. Downing,et al. TEL gene rearrangement in acute lymphoblastic leukemia: a new genetic marker with prognostic significance. , 1997, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[12] J. Downing,et al. Case-control study suggests a favorable impact of TEL rearrangement in patients with B-lineage acute lymphoblastic leukemia treated with antimetabolite-based therapy: a Pediatric Oncology Group study. , 1997, Blood.
[13] H P Koeffler,et al. TEL/AML-1 dimerizes and is associated with a favorable outcome in childhood acute lymphoblastic leukemia. , 1996, Blood.
[14] P. Marynen,et al. The 12;21 translocation involving TEL and deletion of the other TEL allele: two frequently associated alterations found in childhood acute lymphoblastic leukemia. , 1996, Blood.
[15] E. Macintyre,et al. High frequency of t(12;21) in childhood B-lineage acute lymphoblastic leukemia. , 1995, Blood.
[16] S. Shurtleff,et al. TEL/AML1 fusion resulting from a cryptic t(12;21) is the most common genetic lesion in pediatric ALL and defines a subgroup of patients with an excellent prognosis. , 1995, Leukemia.
[17] P. Meltzer,et al. Isolation of a gene encoding a human reduced folate carrier (RFC1) and analysis of its expression in transport-deficient, methotrexate-resistant human breast cancer cells. , 1995, Cancer research.
[18] E. Soeda,et al. Alternative splicing and genomic structure of the AML1 gene involved in acute myeloid leukemia. , 1995, Nucleic acids research.
[19] C. Reynolds,et al. Frequent loss of heterozygosity at the TEL gene locus in acute lymphoblastic leukemia of childhood. , 1995, Blood.
[20] D. Le Paslier,et al. The t(12;21) of acute lymphoblastic leukemia results in a tel-AML1 gene fusion. , 1995, Blood.
[21] D C Ward,et al. Fusion of the TEL gene on 12p13 to the AML1 gene on 21q22 in acute lymphoblastic leukemia. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[22] J. Rowley,et al. Identification of cytogenetically undetected 12p13 translocations and associated deletions with fluorescence in situ hybridization , 1995, Genes, chromosomes & cancer.
[23] R. Kucherlapati,et al. Fluorescence in situ hybridization mapping of translocations and deletions involving the short arm of human chromosome 12 in malignant hematologic diseases. , 1994, Blood.
[24] M. Relling,et al. Blast cell methotrexate-polyglutamate accumulation in vivo differs by lineage, ploidy, and methotrexate dose in acute lymphoblastic leukemia. , 1994, The Journal of clinical investigation.
[25] R. Berger,et al. t( 12;21): A new recurrent translocation in acute lymphoblastic leukemia , 1994, Genes, chromosomes & cancer.
[26] A. Admon,et al. Expression cloning of a human cDNA encoding folylpoly(gamma-glutamate) synthetase and determination of its primary structure. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[27] A. Look,et al. Accumulation of high levels of methotrexate polyglutamates in lymphoblasts from children with hyperdiploid (greater than 50 chromosomes) B-lineage acute lymphoblastic leukemia: a Pediatric Oncology Group study. , 1992, Blood.
[28] A. Look,et al. Trisomy of leukemic cell chromosomes 4 and 10 identifies children with B-progenitor cell acute lymphoblastic leukemia with a very low risk of treatment failure: a Pediatric Oncology Group study. , 1992, Blood.
[29] L. Frankel,et al. Ploidy of lymphoblasts is the strongest predictor of treatment outcome in B-progenitor cell acute lymphoblastic leukemia of childhood: a Pediatric Oncology Group study. , 1992, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[30] F. Behm,et al. Philadelphia chromosome positive childhood acute lymphoblastic leukemia: clinical and cytogenetic characteristics and treatment outcome. A Pediatric Oncology Group study , 1990 .
[31] J. Shuster,et al. Accumulation of methotrexate and methotrexate polyglutamates in lymphoblasts at diagnosis of childhood acute lymphoblastic leukemia: a pilot prognostic factor analysis. , 1990, Blood.
[32] Jonathan J. Shuster,et al. Poor prognosis of children with pre-B acute lymphoblastic leukemia is associated with the t(1;19)(q23;p13): a Pediatric Oncology Group study , 1990 .
[33] S. Raimondi,et al. Nonrandom involvement of the 12p12 breakpoint in chromosome abnormalities of childhood acute lymphoblastic leukemia , 1986 .
[34] C. Pui,et al. Prognostic importance of blast cell DNA content in childhood acute lymphoblastic leukemia. , 1985, Blood.
[35] A. Look,et al. Aneuploidy and percentage of S-phase cells determined by flow cytometry correlate with cell phenotype in childhood acute leukemia. , 1982, Blood.
[36] S. Sallan,et al. The childhood leukemias. , 1981, The Journal of pediatrics.
[37] Mitchell J. Mergenthaler. Nonparametrics: Statistical Methods Based on Ranks , 1979 .
[38] V. Barnett,et al. Applied Linear Statistical Models , 1975 .
[39] J. Shuster,et al. Accumulation of High Levels of Methotrexate Polyglutamates in Lymphoblasts From Children With Hyperdiploid ( > 50 chromosomes ) B-Lineage Acute Lymphoblastic Leukemia : A Pediatric Oncology Group Study , 2003 .
[40] J. Downing,et al. Low frequency of TEL-AML1 in relapsed acute lymphoblastic leukemia supports a favorable prognosis for this genetic subgroup , 1999, Leukemia.
[41] A. Look,et al. Translocations involving chromosome 12p11-13, methotrexate metabolism, and outcome in childhood B-progenitor cell acute lymphoblastic leukemia: a Pediatric Oncology Group study. , 1998, Clinical cancer research : an official journal of the American Association for Cancer Research.
[42] N. Winick,et al. Intermediate-dose intravenous methotrexate with intravenous mercaptopurine is superior to repetitive low-dose oral methotrexate with intravenous mercaptopurine for children with lower-risk B-lineage acute lymphoblastic leukemia: a Pediatric Oncology Group phase III trial. , 1998, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[43] M. Relling,et al. Accumulation of methotrexate polyglutamates in lymphoblasts is a determinant of antileukemic effects in vivo. A rationale for high-dose methotrexate. , 1996, The Journal of clinical investigation.
[44] C. Pui. Medical progress : childhood leukemias , 1995 .
[45] B. Schweitzer,et al. Basis for natural resistance to methotrexate in human acute non-lymphocytic leukemia. , 1991, Leukemia research.
[46] D. Rosenblatt,et al. Methotrexate polyglutamate synthesis in lymphoblasts from children with acute lymphoblastic leukemia. , 1987, Developmental pharmacology and therapeutics.
[47] D. Rosenblatt,et al. Methotrexate metabolism by bone marrow cells from patients with leukemia. , 1983, Advances in experimental medicine and biology.
[48] A. Witte,et al. Synthesis of methotrexate polyglutamates by bone marrow cells from patients with leukemia and lymphoma. , 1980, Developmental pharmacology and therapeutics.
[49] H. Toutenburg,et al. Lehmann, E. L., Nonparametrics: Statistical Methods Based on Ranks, San Francisco. Holden‐Day, Inc., 1975. 480 S., $ 22.95 . , 1977 .