Involvement of cystic fibrosis transmembrane conductance regulator in infection‐induced edema

[1]  M. Eisenhut.  Changes in ion transport in inflammatory disease , 2006, Journal of Inflammation.

[2]  U. Mete,et al.  Genital elephantiasis and sexually transmitted infections – revisited , 2006, International journal of STD & AIDS.

[3]  A. Strandell,et al.  The Presence of Cytokines and Growth Factors in Hydrosalpingeal Fluid , 2004, Journal of Assisted Reproduction and Genetics.

[4]  Roger E Bumgarner,et al.  Chlamydia trachomatis infection alters host cell transcription in diverse cellular pathways. , 2003, The Journal of infectious diseases.

[5]  E. Ng,et al.  New insights into the mechanisms underlying hydrosalpinx fluid formation and its adverse effect on IVF outcome. , 2002, Human reproduction update.

[6]  C. Q. Liu,et al.  Distribution and Regulation of ENaC Subunit and CFTR mRNA Expression in Murine Female Reproductive Tract , 2002, The Journal of Membrane Biology.

[7]  A. Clément,et al.  NF-κB Mediates Up-regulation of CFTR Gene Expression in Calu-3 Cells by Interleukin-1β* , 2001, The Journal of Biological Chemistry.

[8]  S. Miyamoto,et al.  Epithelial cells release proinflammatory cytokines and undergo c-myc-induced apoptosis on exposure to filarial parasitic sheath protein-Bcl2 mediates rescue by activating c-H-ras , 2000, In Vitro Cellular & Developmental Biology - Animal.

[9]  S. Hosseinzadeh,et al.  Coincubation of Human Spermatozoa with Chlamydia trachomatis In Vitro Causes Increased Tyrosine Phosphorylation of Sperm Proteins , 2000, Infection and Immunity.

[10]  O. Pivetta,et al.  Interleukin-1beta regulates CFTR expression in human intestinal T84 cells. , 2000, Biochimica et biophysica acta.

[11]  X. Yang,et al.  Are cytokines and growth factors responsible for the detrimental effects of hydrosalpingeal fluid on pregnancy rates after in vitro fertilization-embryo transfer? , 1999, Fertility and sterility.

[12]  D. Lewis,et al.  Chlamydia trachomatis: the role of cellular and humoral immune mechanisms in the development of blindness. , 1999, Current opinion in infectious diseases.

[13]  W. Stamm,et al.  Chlamydia trachomatis infections: progress and problems. , 1999, The Journal of infectious diseases.

[14]  C. Q. Liu,et al.  Activation of an adenosine 3',5'-cyclic monophosphate-dependent Cl- conductance in response to neurohormonal stimuli in mouse endometrial epithelial cells: the role of cystic fibrosis transmembrane conductance regulator. , 1999, Biology of reproduction.

[15]  T. Carpenter,et al.  Viral respiratory infection increases susceptibility of young rats to hypoxia-induced pulmonary edema. , 1998, Journal of applied physiology.

[16]  J. Engel,et al.  Infection with Chlamydia trachomatis alters the tyrosine phosphorylation and/or localization of several host cell proteins including cortactin , 1997, Infection and immunity.

[17]  D. Jefferson,et al.  Expression and localization of the cystic fibrosis transmembrane conductance regulator mRNA and its protein in rat brain. , 1995, The Journal of clinical investigation.

[18]  A. Nairn,et al.  Cystic Fibrosis Transmembrane Conductance Regulator Is Found Within Brain Ventricular Epithelium and Choroid Plexus , 1995, Journal of neurochemistry.

[19]  J. Annus,et al.  Tubal Infertility: Serologic Relationship to Past Chlamydial and Gonococcal Infection , 1995 .

[20]  S. Birkelund,et al.  Chlamydia trachomatis serovar L2 induces protein tyrosine phosphorylation during uptake by HeLa cells , 1994, Infection and immunity.

[21]  C. Brosnan,et al.  Ultrastructural studies of the blood-retina barrier after exposure to interleukin-1 beta or tumor necrosis factor-alpha. , 1994, Laboratory investigation; a journal of technical methods and pathology.

[22]  S. Falkow,et al.  Signal transduction in the mammalian cell during bacterial attachment and entry , 1993, Cell.

[23]  B. Koller,et al.  An Animal Model for Cystic Fibrosis Made by Gene Targeting , 1992, Science.

[24]  E. Holland,et al.  Persistent superficial punctate keratitis after resolution of chlamydial follicular conjunctivitis. , 1992, Cornea.

[25]  D. Levitt,et al.  Selective infection of astrocytes by Chlamydia trachomatis in primary mixed neuron-glial cell cultures , 1986, Infection and immunity.

[26]  Mårdh Pa,et al.  Chlamydia trachomatis infection in a patient with meningoencephalitis. , 1981 .

[27]  H H USSING,et al.  Active transport of sodium as the source of electric current in the short-circuited isolated frog skin. , 1951, Acta physiologica Scandinavica.

[28]  C. D. Da Ros,et al.  Global epidemiology of sexually transmitted diseases. , 2008, Asian journal of andrology.

[29]  GLOBAL PREVALENCE AND INCIDENCE OF SELECTED CURABLE SEXUALLY TRANSMITTED DISEASES: OVERVIEW AND ESTIMATES , 2000 .

[30]  P. Quinton.  Physiological basis of cystic fibrosis: a historical perspective. , 1999, Physiological reviews.

[31]  M. Kagnoff,et al.  Secretion of proinflammatory cytokines by epithelial cells in response to Chlamydia infection suggests a central role for epithelial cells in chlamydial pathogenesis. , 1997, The Journal of clinical investigation.

[32]  P. Mårdh,et al.  Chlamydia trachomatis infection in a patient with meningoencephalitis. , 1981, The New England journal of medicine.