Compstatin inhibits complement and cellular activation in whole blood in two models of extracorporeal circulation.
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John D Lambris | B. Nilsson | K. Ekdahl | J. Lambris | G. Elgue | R. Larsson | Jaan Hong | A. Sahu | J. Hong | J. Hong | Bo Nilsson | Rolf Larsson
[1] S. Thelin,et al. Heparin-coated equipment reduces complement activation during cardiopulmonary bypass in the pig. , 2008, Artificial organs.
[2] Dimitrios Morikis,et al. Solution structure of Compstatin, a potent complement inhibitor , 1998, Protein science : a publication of the Protein Society.
[3] B. Nilsson,et al. Inhibition of complement activation by soluble recombinant CR1 under conditions resembling those in a cardiopulmonary circuit: reduced up-regulation of CD11b and complete abrogation of binding of PMNs to the biomaterial surface. , 1997, Immunopharmacology.
[4] W. Rosse. Paroxysmal Nocturnal Hemoglobinuria as a Molecular Disease , 1997, Medicine.
[5] John D Lambris,et al. Inhibition of human complement by a C3-binding peptide isolated from a phage-displayed random peptide library. , 1996, Journal of immunology.
[6] L. Matis,et al. Blockade of C5a and C5b-9 generation inhibits leukocyte and platelet activation during extracorporeal circulation. , 1995, The Journal of clinical investigation.
[7] B. Morgan. Complement regulatory molecules: application to therapy and transplantation. , 1995, Immunology today.
[8] M. Kirschfink,et al. Activation of complement by cold agglutinins. , 1994, Infusionstherapie und Transfusionsmedizin.
[9] B. Nilsson,et al. Reconstitution of the alternative pathway of complement by plasma infusions given to a patient with an SLE-like syndrome associated with a hereditary C3 dysfunction. , 1994, Annals of the rheumatic diseases.
[10] W. T. Moore. Integration of Mass Spectrometry into Strategies for Peptide Synthesis , 1993 .
[11] B. Nilsson,et al. Evidence for iC3 generation during cardiopulmonary bypass as the result of blood‐gas interaction , 1993, Clinical and experimental immunology.
[12] W. Baumgartner,et al. Complement inhibition with soluble complement receptor type 1 in cardiopulmonary bypass. , 1993, The Annals of thoracic surgery.
[13] P. Ward,et al. Protective effects of soluble CR1 in complement- and neutrophil-mediated tissue injury. , 1992, Journal of immunology.
[14] D. Fearon. ANTI‐INFLAMMATORY AND IMMUNOSUPPRESSIVE EFFECTS OF RECOMBINANT SOLUBLE COMPLEMENT RECEPTORS , 1991, Clinical and experimental immunology.
[15] P. Sims,et al. The response of human platelets to activated components of the complement system. , 1991, Immunology today.
[16] W. Greeley,et al. Assessing the effect of cardiopulmonary bypass on the brain. , 1991, The Annals of thoracic surgery.
[17] M. Oppermann,et al. Formation of C5a during cardiopulmonary bypass: inhibition by precoating with heparin. , 1991, The Annals of thoracic surgery.
[18] J. Schifferli,et al. Purification of human complement factor D from the peritoneal fluid of patients on chronic ambulatory peritoneal dialysis. , 1991, Journal of immunological methods.
[19] R. Colman,et al. Formation of C1s-C1-inhibitor, kallikrein-C1-inhibitor, and plasmin-alpha 2-plasmin-inhibitor complexes during cardiopulmonary bypass. , 1989, Blood.
[20] D. Chenoweth,et al. Complement activation produced by biomaterials. , 1986, Artificial organs.
[21] S. Frøland,et al. Quantification of the Terminal Complement Complex in Human Plasma by an Enzyme‐Linked Immunosorbent Assay Based on Monoclonal Antibodies against a Neoantigen of the Complex , 1985, Scandinavian journal of immunology.
[22] A. Carpentier,et al. Deleterious effects of cardiopulmonary bypass. A prospective study of bubble versus membrane oxygenation. , 1985, The Journal of thoracic and cardiovascular surgery.
[23] Z. Fishelson,et al. C3 convertase of the alternative complement pathway. Demonstration of an active, stable C3b, Bb (Ni) complex. , 1983, The Journal of biological chemistry.
[24] H. Gresham,et al. Large scale isolation of functionally active components of the human complement system. , 1981, The Journal of biological chemistry.
[25] H. Müller-Eberhard,et al. ISOLATION OF A FRAGMENT (C3a) OF THE THIRD COMPONENT OF HUMAN COMPLEMENT CONTAINING ANAPHYLATOXIN AND CHEMOTACTIC ACTIVITY AND DESCRIPTION OF AN ANAPHYLATOXIN INACTIVATOR OF HUMAN SERUM , 1969, The Journal of experimental medicine.
[26] R. Colman,et al. Nafamostat Mesilate, a Broad Spectrum Protease Inhibitor, Modulates Platelet, Neutrophil and Contact Activation in Simulated Extracorporeal Circulation , 1996, Thrombosis and Haemostasis.
[27] R. Johnson,et al. Complement activation by biomaterials. , 1990, Progress in clinical and biological research.
[28] M. Kazatchkine,et al. Activation of the complement system at the interface between blood and artificial surfaces. , 1988, Biomaterials.
[29] T. Yoshikawa,et al. Inhibitory effects of FUT-175, a new synthetic protease inhibitor, on intravascular hemolysis by human serum in mice. , 1987, International journal of immunopharmacology.