Detection of persistent high risk human papillomavirus infections with hybrid capture II and SPF10/LiPA.

[1]  P. V. van Diest,et al.  Prevalence of HPV in cytomorphologically normal cervical smears, as determined by the polymerase chain reaction, is age‐dependent , 2009, International journal of cancer.

[2]  M. Abrahamowicz,et al.  Occurrence of Cervical Infection with Multiple Human Papillomavirus Types is Associated with Age and Cytologic Abnormalities , 2003, Sexually transmitted diseases.

[3]  M. Poljak,et al.  Hybrid Capture II HPV Test detects at least 15 human papillomavirus genotypes not included in its current high-risk probe cocktail. , 2002, Journal of clinical virology : the official publication of the Pan American Society for Clinical Virology.

[4]  Attila Lorincz,et al.  Restricted cross-reactivity of hybrid capture 2 with nononcogenic human papillomavirus types. , 2002, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.

[5]  J. Kaczorowski,et al.  Optimizing the Hybrid Capture II Human Papillomavirus Test to Detect Cervical Intraepithelial Neoplasia , 2002, Obstetrics and gynecology.

[6]  B. Thomsen,et al.  Type specific persistence of high risk human papillomavirus (HPV) as indicator of high grade cervical squamous intraepithelial lesions in young women: population based prospective follow up study , 2002, BMJ : British Medical Journal.

[7]  David R. Scott,et al.  Viral load of human papillomavirus and risk of CIN3 or cervical cancer , 2002, The Lancet.

[8]  W. Quint,et al.  Human papillomavirus and the long-term risk of cervical neoplasia. , 2002, American journal of epidemiology.

[9]  P. Snijders,et al.  Human papillomavirus 16 load in normal and abnormal cervical scrapes: An indicator of CIN II/III and viral clearance , 2002, International journal of cancer.

[10]  T. Rohan,et al.  Persistent human papillomavirus infection as a predictor of cervical intraepithelial neoplasia. , 2001, JAMA.

[11]  B. Lloveras,et al.  Human papillomavirus genotypes in rural Mozambique , 2001, The Lancet.

[12]  C. Woodman,et al.  Natural history of cervical human papillomavirus infection in young women: a longitudinal cohort study , 2001, The Lancet.

[13]  J. Kaczorowski,et al.  Prevalence and predictors of human papillomavirus infection in women in Ontario, Canada. Survey of HPV in Ontario Women (SHOW) Group. , 2000, CMAJ : Canadian Medical Association journal = journal de l'Association medicale canadienne.

[14]  C. Meijer,et al.  Screening for cervical cancer: should we test for infection with high-risk HPV? , 2000, CMAJ : Canadian Medical Association journal = journal de l'Association medicale canadienne.

[15]  J. Dillner,et al.  A population-based five-year follow-up study of cervical human papillomavirus infection. , 2000, American journal of obstetrics and gynecology.

[16]  H. Adami,et al.  Viral load of human papilloma virus 16 as a determinant for development of cervical carcinoma in situ: a nested case-control study , 2000, The Lancet.

[17]  J. Peto,et al.  Human papillomavirus is a necessary cause of invasive cervical cancer worldwide , 1999, The Journal of pathology.

[18]  Wim Quint,et al.  Development and Clinical Evaluation of a Highly Sensitive PCR-Reverse Hybridization Line Probe Assay for Detection and Identification of Anogenital Human Papillomavirus , 1999, Journal of Clinical Microbiology.

[19]  W. Quint,et al.  Technical Advance Novel Short-Fragment PCR Assay for Highly Sensitive Broad-Spectrum Detection of Anogenital Human Papillomaviruses , 1998 .

[20]  C. Clavel,et al.  Hybrid capture II, a new sensitive test for human papillomavirus detection. Comparison with hybrid capture I and PCR results in cervical lesions. , 1998, Journal of clinical pathology.

[21]  R. Burk,et al.  Natural history of cervicovaginal papillomavirus infection in young women , 1998 .

[22]  C. Meijer,et al.  PCR‐based high‐risk HPV test in cervical cancer screening gives objective risk assessment of women with cytomorphologically normal cervical smears , 1996, International journal of cancer.

[23]  J. Best,et al.  Analytic sensitivities of hybrid‐capture, consensus and type‐specific polymerase chain reactions for the detection of human papillomavirus type 16 DNA , 1996, Journal of medical virology.

[24]  J. Feldman,et al.  Declining Prevalence of Cervicovaginal Human Papillomavirus Infection With Age Is Independent of Other Risk Factors , 1996, Sexually transmitted diseases.

[25]  M. Boon,et al.  Transience of cervical HPV infection in sexually active, young women with normal cervicovaginal cytology. , 1995, British Journal of Cancer.

[26]  C. Meijer,et al.  The presence of persistent high‐risk hpv genotypes in dysplastic cervical lesions is associated with progressive disease: Natural history up to 36 months , 1995, International journal of cancer.

[27]  M. Evander,et al.  Human Papillomavirus Infection Is Transient in Young Women: A PopulationBased Cohort Study , 1995 .

[28]  A. Reingold,et al.  Genital human papillomavirus infection in female university students as determined by a PCR‐based method , 1991, JAMA.